His primary areas of study are Cell biology, PKD1, Internal medicine, Endocrinology and Polycystic kidney disease. His work carried out in the field of Cell biology brings together such families of science as Polycystin 2 and Mechanosensation. His PKD1 study integrates concerns from other disciplines, such as Protein subunit, Kidney metabolism and Kidney disease.
His research in Kidney metabolism intersects with topics in Molecular biology, Embryonic stem cell and Epithelial Physiology. His Internal medicine study incorporates themes from Gastroenterology and Pathology. His studies deal with areas such as Cell cycle, Gene and Cell growth as well as Polycystic kidney disease.
Cell biology, Polycystic kidney disease, Internal medicine, Molecular biology and PKD1 are his primary areas of study. As part of one scientific family, Jing Zhou deals mainly with the area of Cell biology, narrowing it down to issues related to the Polycystin 2, and often Biochemistry. His Polycystic kidney disease study combines topics in areas such as Cancer research and Signal transduction.
He works mostly in the field of Internal medicine, limiting it down to topics relating to Endocrinology and, in certain cases, NOD mice and Nod, as a part of the same area of interest. Jing Zhou interconnects Alport syndrome, Genetics, Type IV collagen, Peptide sequence and Sequence analysis in the investigation of issues within Molecular biology. His PKD1 research integrates issues from Embryonic stem cell, Mutant, Kidney metabolism and Kidney disease.
His scientific interests lie mostly in Cancer research, CD8, Cell biology, Chimeric antigen receptor and T cell. The concepts of his CD8 study are interwoven with issues in Cytotoxic T cell, Breast cancer and Cytokine. His biological study spans a wide range of topics, including OLMSTED SYNDROME, PKD1, Autosomal dominant polycystic kidney disease and Gene.
The PKD1 study combines topics in areas such as PI3K/AKT/mTOR pathway, Knockout mouse and Intraflagellar transport. His Polycystin 2 study, which is part of a larger body of work in Autosomal dominant polycystic kidney disease, is frequently linked to Vesicular transport protein, bridging the gap between disciplines. His Cilium study combines topics from a wide range of disciplines, such as Cardiac output, Kidney, Polycystic kidney disease, Fenoldopam and Extracellular matrix.
His primary areas of investigation include Cancer research, Chimeric antigen receptor, Immunology, Immunotherapy and Immune system. His studies in Cancer research integrate themes in fields like T cell, IL-2 receptor and Homing. His work carried out in the field of Chimeric antigen receptor brings together such families of science as Lymphocyte and Antigen.
His Immunology research incorporates themes from Sialadenitis and Nod. The study incorporates disciplines such as Cytokine and Interleukin 15 in addition to Cytokine release syndrome. His Cytokine research incorporates elements of Adoptive cell transfer and CD8.
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Polycystins 1 and 2 mediate mechanosensation in the primary cilium of kidney cells
Surya M. Nauli;Francis J. Alenghat;Ying Luo;Eric Williams.
Nature Genetics (2003)
Identification of mutations in the COL4A5 collagen gene in Alport syndrome.
David F. Barker;Sirkka Liisa Hostikka;Jing Zhou;Louise T. Chow.
Science (1990)
Polycystic kidney disease: the complete structure of the PKD1 gene and its protein
Gluecksmann-Kuis;O. Tayber;E.A. Woolf;L. Bougueleret.
(1995)
Endothelial Cilia Are Fluid Shear Sensors That Regulate Calcium Signaling and Nitric Oxide Production Through Polycystin-1
Surya M. Nauli;Yoshifumi Kawanabe;John J. Kaminski;William J. Pearce.
Circulation (2008)
Perinatal lethality with kidney and pancreas defects in mice with a targetted Pkd1 mutation.
Weining Lu;Bernard Peissel;Hermik Babakhanlou;Anna Pavlova.
Nature Genetics (1997)
Modelling kidney disease with CRISPR-mutant kidney organoids derived from human pluripotent epiblast spheroids.
Benjamin S. Freedman;Craig R. Brooks;Albert Q. Lam;Albert Q. Lam;Hongxia Fu.
Nature Communications (2015)
Deoxycytidine deaminase-resistant stereoisomer is the active form of (+/-)-2',3'-dideoxy-3'-thiacytidine in the inhibition of hepatitis B virus replication.
C.N. Chang;S.L. Doong;J.H. Zhou;J.W. Beach.
Journal of Biological Chemistry (1992)
Genetic and Functional Studies Implicate HIF1α as a 14q Kidney Cancer Suppressor Gene
Chuan Shen;Rameen Beroukhim;Steven E. Schumacher;Jing Zhou.
Cancer Discovery (2011)
Deletion of the paired alpha 5(IV) and alpha 6(IV) collagen genes in inherited smooth muscle tumors
J Zhou;T Mochizuki;H Smeets;C Antignac.
Science (1993)
Biochemical pharmacology of (+)- and (-)-2',3'-dideoxy-3'- thiacytidine as anti-hepatitis B virus agents
C N Chang;V Skalski;J H Zhou;Y C Cheng.
Journal of Biological Chemistry (1992)
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