What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Cell membrane
The scientist’s investigation covers issues in Molecular biology, Chloride channel, Biophysics, Cell biology and Patch clamp.
His Molecular biology study integrates concerns from other disciplines, such as Cardiac muscle, Endoplasmic reticulum, RNA polymerase II, Transcription and Gene isoform.
His studies in Endoplasmic reticulum integrate themes in fields like Microsome and Gene expression.
His research on Chloride channel concerns the broader Biochemistry.
His work investigates the relationship between Cell biology and topics such as Membrane protein that intersect with problems in Apical membrane, PDZ domain and Membrane transport.
His Patch clamp research includes elements of Cell culture, Tyrosine kinase, G protein, Phosphorylation and GTP'.
His most cited work include:
- Properties of volume-regulated anion channels in mammalian cells (315 citations)
- Abnormal intracellular ca(2+)homeostasis and disease. (207 citations)
- CBS domains: structure, function, and pathology in human proteins (193 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are Cell biology, Molecular biology, Biochemistry, Biophysics and Chloride channel.
His Cell biology research integrates issues from Xenopus, Ion channel and Transfection.
Jan Eggermont has included themes like Polyadenylation, Messenger RNA, Gene expression, Peptide sequence and Gene isoform in his Molecular biology study.
His work on Endoplasmic reticulum, ATPase, Thapsigargin and Pi as part of general Biochemistry research is often related to Phosphatidic acid, thus linking different fields of science.
His Biophysics study combines topics in areas such as Patch clamp, Electrophysiology and Voltage-gated ion channel.
His Chloride channel research is multidisciplinary, incorporating perspectives in Cell culture, HeLa, Extracellular, Cell membrane and Membrane protein.
He most often published in these fields:
- Cell biology (39.34%)
- Molecular biology (27.87%)
- Biochemistry (25.41%)
What were the highlights of his more recent work (between 2014-2021)?
- Cell biology (39.34%)
- ATPase (13.11%)
- Biochemistry (25.41%)
In recent papers he was focusing on the following fields of study:
Cell biology, ATPase, Biochemistry, Secretory pathway and ORAI1 are his primary areas of study.
As part of his studies on Cell biology, Jan Eggermont often connects relevant areas like Polyamine.
The various areas that he examines in his ATPase study include Pharmacology, Drug and Gene isoform.
The concepts of his Gene isoform study are interwoven with issues in Transport protein, Endoplasmic reticulum, Transmembrane domain and Function.
Jan Eggermont undertakes interdisciplinary study in the fields of Biochemistry and Phosphatidic acid through his research.
His ORAI1 research focuses on subjects like Golgi apparatus, which are linked to STIM1, Transcription factor, Nucleus and Cytosol.
Between 2014 and 2021, his most popular works were:
- A lipid switch unlocks Parkinson’s disease-associated ATP13A2 (53 citations)
- ATP13A2 deficiency disrupts lysosomal polyamine export (38 citations)
- Mutated ATP10B increases Parkinson's disease risk by compromising lysosomal glucosylceramide export. (22 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Cell membrane
Jan Eggermont focuses on Cell biology, ATPase, Phosphatidic acid, Biochemistry and Substantia nigra.
In the field of Cell biology, his study on Transport protein, Function and Endoplasmic reticulum overlaps with subjects such as Alpha-synuclein.
His ATPase research includes themes of Thapsigargin, Structure–activity relationship, Sphingomyelin and Cyclopiazonic acid.
His Phosphatidic acid research includes elements of Secretory pathway, Lysosome, Flippase, Phosphorylation and Phosphatidylinositol.
His Biochemistry research focuses on Mitochondrion, Cellular model and Autophosphorylation.
Substantia nigra is connected with Loss function, Cancer research, Mutation, Compound heterozygosity and Dementia with Lewy bodies in his research.
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