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Hiroshi Ueda

Hiroshi Ueda

D-Index & Metrics

Biology and Biochemistry

D-Index
69
Citations
15824
World Ranking
7447
National Ranking
503

Overview

Hiroshi Ueda is affiliated with Kyoto University in Japan and has a significant publication record primarily focused on biomedical and molecular research. Their work spans multiple interrelated fields, particularly medicine and biochemistry, genetics, and molecular biology.

The main areas of scientific inquiry for Hiroshi Ueda include:

  • Pain Mechanisms and Treatments
  • Fibromyalgia and Chronic Fatigue Syndrome Research
  • Sphingolipid Metabolism and Signaling
  • Neuropeptides and Animal Physiology
  • S100 Proteins and Annexins
  • Pharmacological Receptor Mechanisms and Effects
  • Pharmacological Effects of Natural Compounds

Their research is situated within broad and specialized subfields such as molecular biology, physiology, psychiatry and mental health, cellular and molecular neuroscience, and pharmacology. This multidisciplinary approach is reflected in the variety of publication venues where their work appears.

Frequently, Hiroshi Ueda publishes in scientific journals and platforms including:

  • Cells
  • arXiv (Cornell University)
  • Proceedings for Annual Meeting of The Japanese Pharmacological Society
  • IUPHAR/BPS Guide to Pharmacology CITE
  • Journal of Pharmacological Sciences

Among prominent recent papers authored or co-authored by Hiroshi Ueda are:

  • "Pathogenic mechanisms of lipid mediator lysophosphatidic acid in chronic pain" (2020, Progress in Lipid Research)
  • "Review of Kyotorphin Research: A Mysterious Opioid Analgesic Dipeptide and Its Molecular, Physiological, and Pharmacological Characteristics" (2021, Frontiers in Medical Technology)

Hiroshi Ueda has collaborated frequently with several researchers, demonstrating active partnerships within their scientific community. Notable frequent co-authors include:

  • Hiroyuki Neyama
  • Naoki Dozono
  • Brigitte L. Kieffer
  • Shuji Kaneko
  • Sebok K. Halder

This network of collaborators aligns with the interdisciplinary nature of their research topics and fields, reflecting a combined focus on pain, neurobiology, and pharmacology.

Best Publications

  • Initiation of neuropathic pain requires lysophosphatidic acid receptor signaling

    Makoto Inoue;Harunor Rashid;Ryousuke Fujita;James J A Contos

  • TRPM1 is a component of the retinal ON bipolar cell transduction channel in the mGluR6 cascade.

    Chieko Koike;Takehisa Obara;Yoshitsugu Uriu;Tomohiro Numata

  • A novel analgesic dipeptide from bovine brain is a possible Met-enkephalin releaser.

    Hiroshi Takagi;Hirohito Shiomi;Hiroshi Ueda;Hiro Amano

  • Molecular mechanisms of neuropathic pain-phenotypic switch and initiation mechanisms.

    Hiroshi Ueda

  • Morphine-like analgesia by a new dipeptide, L-tyrosyl-L-arginine (Kyotorphin) and its analogue.

    Hiroshi Takagi;Hirohito Shiomi;Hiroshi Ueda;Hiro Amano

  • Epigenetic Gene Silencing Underlies C-Fiber Dysfunctions in Neuropathic Pain

    Hitoshi Uchida;Lin Ma;Hiroshi Ueda

  • Partial loss of tolerance liability to morphine analgesia in mice lacking the nociceptin receptor gene

    Hiroshi Ueda;Taku Yamaguchi;Shogo Tokuyama;Makoto Inoue

  • Increased Expression of Vanilloid Receptor 1 on Myelinated Primary Afferent Neurons Contributes to the Antihyperalgesic Effect of Capsaicin Cream in Diabetic Neuropathic Pain in Mice

    M. D. Harunor Rashid;Makoto Inoue;Shiho Bakoshi;Hiroshi Ueda

  • Novel Expression of Vanilloid Receptor 1 on Capsaicin-Insensitive Fibers Accounts for the Analgesic Effect of Capsaicin Cream in Neuropathic Pain

    Harunor Rashid;Makoto Inoue;Saori Kondo;Toshiko Kawashima

  • Peripheral mechanisms of neuropathic pain – involvement of lysophosphatidic acid receptor-mediated demyelination

    Hiroshi Ueda

  • Low doses of naloxone produce analgesia in the mouse brain by blocking presynaptic autoinhibition of enkephalin release.

    Hiroshi Ueda;Nobuyuki Fukushima;Tadaaki Kitao;Ming Ge

  • Reconstitution of rat brain mu opioid receptors with purified guanine nucleotide-binding regulatory proteins, Gi and Go.

    Hiroshi Ueda;Hitoshi Harada;Masakatsu Nozaki;Toshiaki Katada

  • Enhanced spinal nociceptin receptor expression develops morphine tolerance and dependence.

    Hiroshi Ueda;Makoto Inoue;Hiroshi Takeshima;Yoshikazu Iwasawa

  • Autotaxin, a synthetic enzyme of lysophosphatidic acid (LPA), mediates the induction of nerve-injured neuropathic pain.

    Makoto Inoue;Lin Ma;Junken Aoki;Jerold Chun

  • Inhibition of Paclitaxel-Induced A-Fiber Hypersensitization by Gabapentin

    Misaki Matsumoto;Makoto Inoue;Andreas Hald;Weijiao Xie

  • Differential involvement of μ-opioid receptor subtypes in endomorphin-1- and -2-induced antinociception

    Shinobu Sakurada;James E Zadina;Abba J Kastin;Sou Katsuyama

  • Nociceptin/orphanin FQ-induced nociceptive responses through substance P release from peripheral nerve endings in mice

    Makoto Inoue;Motomasa Kobayashi;Shunji Kozaki;Andreas Zimmer

  • Comparison of the analgesic effects of various opioid peptides by a newly devised intracisternal injection technique in conscious mice.

    Hiroshi Ueda;Hiro Amano;Hirohito Shiomi;Hiroshi Takagi

  • Mechanisms underlying morphine analgesic tolerance and dependence.

    Hiroshi Ueda;Mutsumi Ueda

  • Lysophosphatidylcholine induces neuropathic pain through an action of autotaxin to generate lysophosphatidic acid

    Makoto Inoue;W. Xie;Y. Matsushita;J. Chun

Frequent Co-Authors

Yoshimi Misu
Yoshimi Misu Yokohama City University
Hiroshi Takagi
Hiroshi Takagi Okinawa Institute of Science and Technology
Yoshio Goshima
Yoshio Goshima Yokohama City University
Hiroshi Takagi
Hiroshi Takagi Nara Institute of Science and Technology
Jerold Chun
Jerold Chun Sanford Burnham Prebys Medical Discovery Institute
Hidemi Misawa
Hidemi Misawa Keio University
Hiroshi Takeshima
Hiroshi Takeshima Kyoto University
Junken Aoki
Junken Aoki Tohoku University
Yoshihiro Nakata
Yoshihiro Nakata Hiroshima University
Toshiaki Katada
Toshiaki Katada Musashino University

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