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Biology and Biochemistry

D-Index
57
Citations
12907
World Ranking
13739
National Ranking
5819

Overview

Gary S. Bird is affiliated with the National Institutes of Health in the United States. Their research spans multiple areas within biochemistry, genetics, molecular biology, and medicine, with a particular focus on molecular biology and cardiology. The subfields of study explored include molecular biology, cardiology and cardiovascular medicine, sensory systems, cellular and molecular neuroscience, and surgery.

The scientist's main research topics encompass signaling pathways in disease, receptor mechanisms and signaling, ion channel regulation and function, cardiac electrophysiology and arrhythmias, ion channels and receptors, neurobiology and insect physiology research, and phosphodiesterase function and regulation.

Gary S. Bird has contributed to several peer-reviewed publications, including:

  • The N terminus of Orai1 couples to the AKAP79 signaling complex to drive NFAT1 activation by local Ca 2+ entry, 2021, Proceedings of the National Academy of Sciences
  • A Reappraisal of the Effects of L-type Ca2+ Channel Blockers on Store-Operated Ca2+ Entry and Heart Failure, 2023, Function
  • Scrutinizing science to save lives: uncovering flaws in the data linking L-type calcium channels blockers to CRAC channels and heart failure, 2024, bioRxiv (Cold Spring Harbor Laboratory)
  • 488 Examining the Relationship Between Infarct Size and Circulating Extracellular Matrix Biomarkers Over Acute and Chronic Phases of Myocardial Infarction, 2020, Heart Lung and Circulation
  • L-type calcium channel blockers at therapeutic concentrations are not linked to CRAC channels and heart failure, 2025, Clinical and Experimental Hypertension

Frequent co-authors in Gary S. Bird's research include Yu-Ping Lin, Charles J. Tucker, Anant B. Parekh, Sandosh Padmanabhan, and Geoffrey A. Mueller.

The scientist's work has appeared across various publication venues such as Proceedings of the National Academy of Sciences, Function, bioRxiv (Cold Spring Harbor Laboratory), Heart Lung and Circulation, and Clinical and Experimental Hypertension.

Best Publications

  • Mechanisms of capacitative calcium entry

    James W. Putney;Lisa M. Broad;Franz-Josef Braun;Jean-Philippe Lievremont

  • The Inositol Phosphate-Calcium Signaling System in Nonexcitable Cells

    James W. Putney;Gary St. J. Bird

  • Large Store-operated Calcium Selective Currents Due to Co-expression of Orai1 or Orai2 with the Intracellular Calcium Sensor, Stim1

    Jason C. Mercer;Wayne I. DeHaven;Jeremy T. Smyth;Barbara Wedel

  • The signal for capacitative calcium entry

    James W. Putney;Gary St. J. Bird

  • SPATIAL AND TEMPORAL ASPECTS OF CELLULAR CALCIUM SIGNALING

    Andrew P. Thomas;Gary St. J. Bird;György Hajnóczky;Lawrence D. Robb-Gaspers

  • Defective mast cell effector functions in mice lacking the CRACM1 pore subunit of store-operated calcium release-activated calcium channels.

    Monika Vig;Wayne I DeHaven;Gary S Bird;James M Billingsley

  • Comparison of Human TRPC3 Channels in Receptor-activated and Store-operated Modes DIFFERENTIAL SENSITIVITY TO CHANNEL BLOCKERS SUGGESTS FUNDAMENTAL DIFFERENCES IN CHANNEL COMPOSITION

    Mohamed Trebak;Gary St. J. Bird;Richard R. McKay;James W. Putney

  • TRPC channels function independently of STIM1 and Orai1

    Wayne I. DeHaven;Bertina F. Jones;John G. Petranka;Jeremy T. Smyth

  • Role of the phospholipase C-inositol 1,4,5-trisphosphate pathway in calcium release-activated calcium current and capacitative calcium entry.

    Lisa M. Broad;Franz-Josef Braun;Jean-Philippe Lievremont;Gary St. J. Bird

  • Complex Actions of 2-Aminoethyldiphenyl Borate on Store-operated Calcium Entry

    Wayne I. DeHaven;Jeremy T. Smyth;Rebecca R. Boyles;Gary S. Bird

  • The TRPC3/6/7 subfamily of cation channels.

    Mohamed Trebak;Guillermo Vazquez;Gary St.J. Bird;James W. Putney

  • Methods for studying store-operated calcium entry.

    Gary S. Bird;Wayne I. DeHaven;Jeremy T. Smyth;James W. Putney

  • Ca2+-store-dependent and -independent reversal of Stim1 localization and function.

    Jeremy T. Smyth;Wayne I. DeHaven;Gary S. Bird;James W. Putney

  • Activation of Ca2+ entry into acinar cells by a non-phosphorylatable inositol trisphosphate.

    G. S. J. Bird;M. F. Rossier;A. R. Hughes;S. B. Shears

  • Inhibition of thapsigargin-induced calcium entry by microinjected guanine nucleotide analogues. Evidence for the involvement of a small G-protein in capacitative calcium entry.

    G. S. J. Bird;J. W. Putney

  • The mechanism for synergism between phospholipase C- and adenylylcyclase-linked hormones in liver. Cyclic AMP-dependent kinase augments inositol trisphosphate-mediated Ca2+ mobilization without increasing the cellular levels of inositol polyphosphates.

    G M Burgess;G S Bird;J F Obie;J W Putney

  • Signaling mechanism for receptor-activated canonical transient receptor potential 3 (TRPC3) channels.

    Mohamed Trebak;Gary St. J. Bird;Richard R. McKay;Lutz Birnbaumer

  • Signaling pathways underlying muscarinic receptor-induced [Ca2+]i oscillations in HEK293 cells.

    Dali Luo;Lisa M. Broad;Gary St. J. Bird;James W. Putney

  • Human Trp3 forms both inositol trisphosphate receptor-dependent and receptor-independent store-operated cation channels in DT40 avian B lymphocytes

    Guillermo Vazquez;Jean-Philippe Lievremont;Gary St. J. Bird;James W. Putney

  • Amantadine protects dopamine neurons by a dual action: Reducing activation of microglia and inducing expression of GNDF in astroglia

    Bernardino Ossola;Nadia Schendzielorz;Shih-Heng Chen;Gary S. Bird

Frequent Co-Authors

James W. Putney
James W. Putney National Institutes of Health
Mohamed Trebak
Mohamed Trebak Pennsylvania State University
Stephen B. Shears
Stephen B. Shears National Institutes of Health
Pekka T. Männistö
Pekka T. Männistö University of Helsinki
Jau-Shyong Hong
Jau-Shyong Hong National Institutes of Health
Gregory R. Monteith
Gregory R. Monteith University of Queensland
Yasuo Mori
Yasuo Mori Kyoto University
John A. Cidlowski
John A. Cidlowski National Institutes of Health
Edward M. Eddy
Edward M. Eddy National Institutes of Health
Stefan Feske
Stefan Feske New York University

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