His work in Cell biology is not limited to one particular discipline; it also encompasses Mechanotransduction. His Mechanotransduction study often links to related topics such as Cell biology. Genetics and Computational biology are two areas of study in which Friedemann Kiefer engages in interdisciplinary work. With his scientific publications, his incorporates both Computational biology and Genetics. He regularly links together related areas like Spleen in his Immunology studies. Friedemann Kiefer integrates many fields in his works, including Spleen and Bone marrow. In his study, Friedemann Kiefer carries out multidisciplinary Bone marrow and Granulocyte research. His Immunology research extends to the thematically linked field of Granulocyte. Cell is frequently linked to CD44 in his study.
In the field of Lymphatic vessel and Lymphangiogenesis Friedemann Kiefer studies Metastasis. Friedemann Kiefer combines Lymphangiogenesis and Lymphatic system in his studies. In his research, Friedemann Kiefer performs multidisciplinary study on Lymphatic system and Lymphatic vessel. He integrates Cell biology with Molecular biology in his research. While working in this field, Friedemann Kiefer studies both Molecular biology and Cell biology. In his works, Friedemann Kiefer undertakes multidisciplinary study on Immunology and T cell. Friedemann Kiefer combines T cell and Immune system in his studies. He merges Immune system with Immunology in his study. In his work, Friedemann Kiefer performs multidisciplinary research in Genetics and Cell.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The Syk Protein Tyrosine Kinase Is Essential for Fcγ Receptor Signaling in Macrophages and Neutrophils
Friedemann Kiefer;John Brumell;Nadia Al-Alawi;Sylvain Latour.
Molecular and Cellular Biology (1998)
CD44 Regulates Hematopoietic Progenitor Distribution, Granuloma Formation, and Tumorigenicity
Rudolf Schmits;Jorge Filmus;Nicole Gerwin;Giorgio Senaldi.
MLK-3 ACTIVATES THE SAPK/JNK AND P38/RK PATHWAYS VIA SEK1 AND MKK3/6
L. A. Tibbles;Y. L. Ing;F. Kiefer;J. Chan.
The EMBO Journal (1996)
The junctional adhesion molecule (JAM) family members JAM-2 and JAM-3 associate with the cell polarity protein PAR-3: a possible role for JAMs in endothelial cell polarity
Klaus Ebnet;Michel Aurrand-Lions;Annegret Kuhn;Friedemann Kiefer.
Journal of Cell Science (2003)
A novel multistep mechanism for initial lymphangiogenesis in mouse embryos based on ultramicroscopy.
René Hägerling;Cathrin Pollmann;Martin Andreas;Christian Schmidt.
The EMBO Journal (2013)
Mechanotransduction, PROX1, and FOXC2 Cooperate to Control Connexin37 and Calcineurin during Lymphatic-Valve Formation
Amélie Sabine;Amélie Sabine;Yan Agalarov;Yan Agalarov;Hélène Maby-El Hajjami;Hélène Maby-El Hajjami;Muriel Jaquet;Muriel Jaquet.
Developmental Cell (2012)
HPK1, a hematopoietic protein kinase activating the SAPK/JNK pathway.
F Kiefer;L A Tibbles;M Anafi;A Janssen.
The EMBO Journal (1996)
Stabilizing the VE-cadherin–catenin complex blocks leukocyte extravasation and vascular permeability
Dörte Schulte;Verena Küppers;Nina Dartsch;Andre Broermann.
The EMBO Journal (2011)
Molecular cloning of LSIRF, a lymphoid-specific member of the interferon regulatory factor family that binds the interferon-stimulated response element (ISRE)
Toshifumi Matsuyama;Alex Grossman;Hans willi Mittrücker;David P. Siderovski.
Nucleic Acids Research (1995)
The role of fatty acid β-oxidation in lymphangiogenesis
Brian W. Wong;Xingwu Wang;Annalisa Zecchin;Bernard Thienpont.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: