What is he best known for?
The fields of study he is best known for:
Frank Thévenod mainly investigates Cell biology, Apoptosis, Biochemistry, Mitochondrion and Kidney.
His Cell biology research includes elements of Uniporter and Calmodulin.
His Apoptosis-inducing factor and Cytochrome c study, which is part of a larger body of work in Apoptosis, is frequently linked to Population, bridging the gap between disciplines.
The concepts of his Mitochondrion study are interwoven with issues in DMT1, Reactive oxygen species, Cytosol, Mitochondrial permeability transition pore and Metallothionein.
His Kidney study combines topics from a wide range of disciplines, such as Molecular biology, Receptor and Cubilin.
As part of the same scientific family, Frank Thévenod usually focuses on Molecular biology, concentrating on Programmed cell death and intersecting with Reabsorption.
His most cited work include:
- Cadmium and cellular signaling cascades: to be or not to be? (263 citations)
- Nephrotoxicity and the proximal tubule. Insights from cadmium. (220 citations)
- Priming of insulin granules for exocytosis by granular Cl(-) uptake and acidification (160 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Cell biology, Biochemistry, Kidney, Molecular biology and Apoptosis.
His Cell biology research is multidisciplinary, incorporating perspectives in Toxicity, Metallothionein, Programmed cell death and DMT1.
In Kidney, Frank Thévenod works on issues like Cadmium, which are connected to Pharmacology.
In his research, Mitochondrial permeability transition pore is intimately related to Mitochondrion, which falls under the overarching field of Apoptosis.
His Endocrinology research is multidisciplinary, relying on both Cotransporter, Wnt signaling pathway and In vivo.
In his study, which falls under the umbrella issue of In vivo, P-glycoprotein is strongly linked to Endothelial stem cell.
He most often published in these fields:
- Cell biology (40.20%)
- Biochemistry (38.24%)
- Kidney (36.27%)
What were the highlights of his more recent work (between 2011-2021)?
- Cell biology (40.20%)
- Cadmium (10.78%)
- Molecular biology (19.61%)
In recent papers he was focusing on the following fields of study:
His main research concerns Cell biology, Cadmium, Molecular biology, Toxicity and Programmed cell death.
His Cell biology study incorporates themes from Zymogen granule, Endocytosis, Lysosome and Metallothionein.
His Cadmium research is multidisciplinary, incorporating elements of Genetics, Mitochondrion, Pharmacology and Nephrotoxicity.
His research in Molecular biology intersects with topics in Cyclosporin a, Internalization, Intestinal mucosa and Cytolysis.
His work deals with themes such as Kidney and Caco-2, which intersect with Toxicity.
His study focuses on the intersection of Programmed cell death and fields such as Unfolded protein response with connections in the field of Autophagy.
Between 2011 and 2021, his most popular works were:
- Cadmium and cellular signaling cascades: interactions between cell death and survival pathways. (140 citations)
- Toxicology of cadmium and its damage to mammalian organs. (104 citations)
- Lipocalin-2 (24p3/Neutrophil Gelatinase-associated Lipocalin (NGAL)) Receptor Is Expressed in Distal Nephron and Mediates Protein Endocytosis (57 citations)
In his most recent research, the most cited papers focused on:
His scientific interests lie mostly in Cell biology, Programmed cell death, Unfolded protein response, Apoptosis and Wnt signaling pathway.
His Cell biology study combines topics from a wide range of disciplines, such as Receptor complex, Endocytosis, Lipocalin and Renal physiology.
His studies deal with areas such as Cell signaling, PI3K/AKT/mTOR pathway, Signal transduction and Cellular adaptation as well as Unfolded protein response.
His Wnt signaling pathway research is multidisciplinary, incorporating elements of Cancer cell, P-glycoprotein, Transcription factor and Cancer research.
To a larger extent, Frank Thévenod studies Biochemistry with the aim of understanding Organic cation transport proteins.
His research investigates the connection with Biochemistry and areas like Reabsorption which intersect with concerns in Transferrin.
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