Enrico V. Avvedimento focuses on Cell biology, Molecular biology, Protein kinase A, Internal medicine and Endocrinology. Enrico V. Avvedimento has researched Cell biology in several fields, including M-Phase Promoting Factor and In vivo. His Molecular biology research incorporates elements of Chromatin, DNA repair, DNA demethylation, Response element and Histone methylation.
His research in Protein kinase A tackles topics such as Signal transduction which are related to areas like Alternative splicing and Kinase. His study in the fields of Simvastatin and Muscle hypertrophy under the domain of Internal medicine overlaps with other disciplines such as Left ventricular hypertrophy, Ventricular pressure and Ascending aorta. His Endocrinology research includes themes of Fibroblast, Systemic scleroderma and Autoimmunity.
Molecular biology, Cell biology, Protein kinase A, Immunology and Internal medicine are his primary areas of study. His Molecular biology research is multidisciplinary, relying on both Histone methylation, DNA methylation, Chromatin, DNA repair and Transcription. His work on Biochemistry expands to the thematically related Cell biology.
His Protein kinase A research is multidisciplinary, incorporating perspectives in Cytosol, Protein subunit and CREB1. In his research on the topic of Immunology, Inflammation is strongly related with Fibrosis. His study explores the link between Internal medicine and topics such as Endocrinology that cross with problems in Receptor.
His primary areas of investigation include DNA damage, Molecular biology, Immunology, DNA methylation and Cancer research. Enrico V. Avvedimento combines subjects such as Oxidative stress, Wnt signaling pathway, WIF1, Growth factor and Fibrosis with his study of DNA damage. His Molecular biology research integrates issues from Chromatin, Histone methylation and Epigenetics of physical exercise.
His Chromatin research is multidisciplinary, incorporating elements of Enhancer, Chromatin immunoprecipitation and Cell biology. His studies deal with areas such as Biomarker, Myofibroblast and Telomere as well as Immunology. His studies in Cancer research integrate themes in fields like Carcinogenesis, Platelet-derived growth factor receptor, Retinoic acid receptor, Retinoic acid receptor gamma and Estrogen receptor.
His primary scientific interests are in Molecular biology, Histone methylation, Platelet-derived growth factor receptor, NADPH oxidase and DNA methylation. His study deals with a combination of Molecular biology and Oligodendrocyte differentiation. His Histone methylation study integrates concerns from other disciplines, such as Chromatin and Chromatin remodeling.
His Platelet-derived growth factor receptor study necessitates a more in-depth grasp of Growth factor. His DNA methylation research includes elements of Methylation and DNA repair. His study looks at the intersection of DNA damage and topics like Gene silencing with Fibrosis and Cancer research.
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Stimulatory autoantibodies to the PDGF receptor in systemic sclerosis.
Silvia Svegliati Baroni;Mariarosaria Santillo;Federica Bevilacqua;Michele Luchetti.
The New England Journal of Medicine (2006)
DNA Oxidation as Triggered by H3K9me2 Demethylation Drives Estrogen-Induced Gene Expression
Bruno Perillo;Maria Neve Ombra;Alessandra Bertoni;Concetta Cuozzo.
The biological functions of A-kinase anchor proteins
Antonio Feliciello;Max E Gottesman;Enrico V Avvedimento.
Journal of Molecular Biology (2001)
Expression of thyrotropin-receptor mRNA in healthy and Graves' disease retro-orbital tissue
A. Feliciello;I. Ciullo;G.F. Fenzi;G. Bonavolontà.
The Lancet (1993)
Inhibition of cellular ras prevents smooth muscle cell proliferation after vascular injury in vivo
Ciro Indolfi;Enrico V. Avvedimento;Antonio Rapacciuolo;Emilio Di Lorenzo.
Nature Medicine (1995)
Stimulatory autoantibodies to PDGF receptor in patients with extensive chronic graft-versus-host disease
Silvia Svegliati;Attilio Olivieri;Nadia Campelli;Michele Luchetti.
Reconstitution of an ATM-Dependent Checkpoint that Inhibits Chromosomal DNA Replication following DNA Damage
Vincenzo Costanzo;Kirsten Robertson;Carol Y. Ying;Edward Kim.
Molecular Cell (2000)
Activation of cAMP-PKA signaling in vivo inhibits smooth muscle cell proliferation induced by vascular injury.
Ciro Indolfi;Enrico Vittorio Avvedimento;Emilio Di Lorenzo;Giovanni Esposito.
Nature Medicine (1997)
DNA damage, homology-directed repair, and DNA methylation.
Concetta Cuozzo;Antonio Porcellini;Tiziana Angrisano;Annalisa Morano.
PLOS Genetics (2005)
cAMP-dependent protein kinase induces cAMP-response element-binding protein phosphorylation via an intracellular calcium release/ERK-dependent pathway in striatal neurons.
Patrizia Zanassi;Mayra Paolillo;Antonio Feliciello;Enrico V. Avvedimento.
Journal of Biological Chemistry (2001)
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