His scientific interests lie mostly in Molecular biology, Gene, Genetics, Internal medicine and Endocrinology. His research integrates issues of Primary Hyperoxaluria Type I, X chromosome, Skewed X-inactivation, XIST and Signal peptide in his study of Molecular biology. His Gene research is multidisciplinary, incorporating elements of Spermatogenesis and Testicular tissue.
In his research, Intron and Ameloblast is intimately related to Amelogenesis imperfecta, which falls under the overarching field of Genetics. His research in the fields of Transplantation, Oxalobacter and Oxalate transport overlaps with other disciplines such as SLC26A6. His study on Primary hyperoxaluria and Kidney is often connected to Gestation and Luteinizing hormone as part of broader study in Endocrinology.
His primary areas of study are Internal medicine, Endocrinology, Molecular biology, Genetics and Gene. His work investigates the relationship between Internal medicine and topics such as Gastroenterology that intersect with problems in Diabetes mellitus and Immunology. His research on Endocrinology often connects related areas such as Receptor.
His research on Molecular biology also deals with topics like
His main research concerns Internal medicine, Biochemistry, Primary hyperoxaluria, Endocrinology and Gene. His Internal medicine study combines topics from a wide range of disciplines, such as Gastroenterology, Diabetes mellitus and Oncology. His Primary hyperoxaluria research is multidisciplinary, incorporating perspectives in Immunohistochemistry and Inborn error of metabolism.
His studies link Enzyme replacement therapy with Endocrinology. His Gene study often links to related topics such as Cancer research. His work carried out in the field of Mutation brings together such families of science as Phenotype and Molecular biology.
Eduardo Salido mainly investigates Biochemistry, Internal medicine, Glyoxylate cycle, Primary hyperoxaluria and Genetics. Eduardo Salido has included themes like Cyclooxygenase and Endocrinology in his Internal medicine study. The study incorporates disciplines such as Thromboxane and Fatty liver in addition to Endocrinology.
Eduardo Salido combines subjects such as Calcium oxalate, Kidney and Primary Hyperoxaluria Type I with his study of Glyoxylate cycle. His Primary Hyperoxaluria Type I research integrates issues from Molecular biology, Glycolate oxidase, Allele and CRISPR. His research investigates the link between Primary hyperoxaluria and topics such as Inborn error of metabolism that cross with problems in Peroxisome, Excretion, Clinical trial and Bioinformatics.
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Circulating urokinase receptor as a cause of focal segmental glomerulosclerosis
Changli Wei;Shafic El Hindi;Jing Li;Alessia Fornoni.
Nature Medicine (2011)
The human enamel protein gene amelogenin is expressed from both the X and the Y chromosomes.
E. C. Salido;P. H. Yen;K. Koprivnikar;Loh-Chung Yu.
American Journal of Human Genetics (1992)
PrimPol, an Archaic Primase/Polymerase Operating in Human Cells
Sara García-Gómez;Aurelio Reyes;María I. Martínez-Jiménez;E. Sandra Chocrón.
Molecular Cell (2013)
Primary hyperoxaluria Type 1: indications for screening and guidance for diagnosis and treatment
Pierre Cochat;Sally-Anne Hulton;Cécile Acquaviva;Christopher J. Danpure.
Nephrology Dialysis Transplantation (2012)
Identification and Structural and Functional Characterization of Human Enamelysin (MMP-20)†,‡
Elena Llano;Alberto M. Pendás;Vera Knäuper;Timo Sorsa.
Calcium Metabolism and Skeletal Problems after Transplantation
Armando Torres;Victor Lorenzo;Eduardo Salido.
Journal of The American Society of Nephrology (2002)
Epidermal Growth Factor and the Kidney
D A Fisher;E C Salido;L Barajas.
Annual Review of Physiology (1989)
The Human Autosomal Gene DAZLA: Testis Specificity and a Candidate for Male Infertility
Pauline H. Yen;Ning Ning Chai;Eduardo C. Salido.
Human Molecular Genetics (1996)
A missense mutation in Tbce causes progressive motor neuronopathy in mice.
Natalia Martin;Jean Jaubert;Pierre Gounon;Eduardo Salido.
Nature Genetics (2002)
A Founder Mutation in Artemis, an SNM1-Like Protein, Causes SCID in Athabascan-Speaking Native Americans
Lanying Li;Despina Moshous;Yungui Zhou;Junhua Wang.
Journal of Immunology (2002)
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