Her main research concerns Genetics, Allele, Attention deficit hyperactivity disorder, Transmission disequilibrium test and Genome-wide association study. Her study looks at the relationship between Allele and fields such as Locus, as well as how they intersect with chemical problems. Cathy L. Barr works mostly in the field of Transmission disequilibrium test, limiting it down to topics relating to Linkage disequilibrium and, in certain cases, Serotonin and Receptor.
Her research in Genome-wide association study intersects with topics in Genetic architecture, Bipolar disorder, Tourette syndrome and Heritability. In Bipolar disorder, Cathy L. Barr works on issues like Genetic association, which are connected to Brain-derived neurotrophic factor. Her Heritability research is multidisciplinary, relying on both Clinical psychology, Major depressive disorder, Genetic heterogeneity, Risk factor and Comorbidity.
Cathy L. Barr mainly investigates Genetics, Haplotype, Gene, Attention deficit hyperactivity disorder and Allele. In her study, Mood, Bipolar disorder, Internal medicine and Age of onset is inextricably linked to Mood disorders, which falls within the broad field of Haplotype. In her work, Comorbidity is strongly intertwined with Clinical psychology, which is a subfield of Bipolar disorder.
Her Attention deficit hyperactivity disorder research incorporates themes from Dopaminergic, Dopamine, Impulsivity, Genetic linkage and Genetic determinism. Her research investigates the link between Allele and topics such as Locus that cross with problems in Dyslexia. Within one scientific family, Cathy L. Barr focuses on topics pertaining to Heritability under Genome-wide association study, and may sometimes address concerns connected to Genetic heterogeneity and Risk factor.
Genetics, Genome-wide association study, Gene, Tourette syndrome and Copy-number variation are her primary areas of study. Her biological study spans a wide range of topics, including Bipolar disorder, Psychiatry, Genetic association, Autism spectrum disorder and Clinical psychology. Her studies in Bipolar disorder integrate themes in fields like Heritability, Major depressive disorder, Genetic heterogeneity, Risk factor and Comorbidity.
Her work focuses on many connections between Gene and other disciplines, such as Computational biology, that overlap with her field of interest in Chromatin. The various areas that Cathy L. Barr examines in her Tourette syndrome study include SLITRK1, Candidate gene, Genetic architecture, Neuroscience and DNA sequencing. The Copy-number variation study combines topics in areas such as Mutation, Child and adolescent psychiatry, Tourette's syndrome and Bioinformatics.
The scientist’s investigation covers issues in Genome-wide association study, Bipolar disorder, Comorbidity, Risk factor and Genetic heterogeneity. Her Genome-wide association study study integrates concerns from other disciplines, such as Tourette syndrome and Genetic architecture. Her Genetic architecture research is multidisciplinary, incorporating elements of Schizophrenia, Psychiatry, Pleiotropy, Autism spectrum disorder and Genotype.
Genotype is a subfield of Genetics that Cathy L. Barr explores. Her study in the fields of Chromosome conformation capture, CTCF and ChIA-PET under the domain of Genetics overlaps with other disciplines such as Scaffold/matrix attachment region and Cohesin complex. Her research integrates issues of Major depressive disorder, Genetic association, Clinical psychology and Heritability in her study of Comorbidity.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Analysis of shared heritability in common disorders of the brain
Verneri Anttila;Verneri Anttila;Brendan Bulik-Sullivan;Brendan Bulik-Sullivan;Hilary K. Finucane;Raymond K. Walters;Raymond K. Walters.
Science (2018)
A hypervariable segment in the human dopamine receptor D4 (DRD4) gene
Jay B. Lichter;Cathy L. Barr;James L. Kennedy;Hubert H.M. Van Tol.
Human Molecular Genetics (1993)
A Compendium of Chromatin Contact Maps Reveals Spatially Active Regions in the Human Genome
Anthony D. Schmitt;Ming Hu;Inkyung Jung;Zheng Xu.
Cell Reports (2016)
Dopamine genes and ADHD.
J. M. Swanson;J. M. Swanson;Pamela Flodman;James Kennedy;M. Anne Spence.
Neuroscience & Biobehavioral Reviews (2000)
Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders
Phil H. Lee;Verneri Anttila;Hyejung Won;Yen-Chen A. Feng.
Cell (2019)
Base-Resolution Analyses of Sequence and Parent-of-Origin Dependent DNA Methylation in the Mouse Genome
Wei Xie;Cathy L. Barr;Audrey Kim;Feng Yue.
Cell (2012)
Restraint and Cancellation: Multiple Inhibition Deficits in Attention Deficit Hyperactivity Disorder
Russell Schachar;Gordon D. Logan;Philippe Robaey;Shirley Chen.
Journal of Abnormal Child Psychology (2007)
Partitioning the heritability of tourette syndrome and obsessive compulsive disorder reveals differences in genetic architecture
Lea K. Davis;Dongmei Yu;Clare L. Keenan;Eric R. Gamazon.
PLOS Genetics (2013)
Identification of DNA variants in the SNAP-25 gene and linkage study of these polymorphisms and attention-deficit hyperactivity disorder.
C L Barr;Y Feng;K Wigg;S Bloom.
Molecular Psychiatry (2000)
Joint analysis of the DRD5 marker concludes association with attention-deficit/hyperactivity disorder confined to the predominantly inattentive and combined subtypes.
Naomi Lowe;Aiveen Kirley;Ziarih Hawi;Pak Sham.
American Journal of Human Genetics (2004)
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