What is she best known for?
The fields of study she is best known for:
Andréa C. LeBlanc mainly investigates Programmed cell death, Cell biology, Caspase, Molecular biology and Apoptosis.
Her Programmed cell death study combines topics from a wide range of disciplines, such as Cell and Neurodegeneration.
Her Cell biology study combines topics in areas such as P3 peptide and Amyloid beta.
Andréa C. LeBlanc studies Caspase, namely Caspase 6.
The concepts of her Molecular biology study are interwoven with issues in Bcl-2-associated X protein and Cytosol.
Her Apoptosis research focuses on Intracellular and how it connects with Human brain and Amyloid precursor protein.
Her most cited work include:
- Involvement of Caspases in Proteolytic Cleavage of Alzheimer’s Amyloid-β Precursor Protein and Amyloidogenic Aβ Peptide Formation (699 citations)
- Fatal familial insomnia and familial Creutzfeldt-Jakob disease: disease phenotype determined by a DNA polymorphism (582 citations)
- Fatal Familial Insomnia, a Prion Disease with a Mutation at Codon 178 of the Prion Protein Gene (488 citations)
What are the main themes of her work throughout her whole career to date?
Andréa C. LeBlanc mainly focuses on Cell biology, Caspase 6, Molecular biology, Caspase and Programmed cell death.
Her Cell biology research is multidisciplinary, relying on both Apoptosis, Cell, Amyloid beta and Prion protein.
The various areas that she examines in her Caspase 6 study include In vitro, Cysteine protease, Alzheimer's disease, Senile plaques and Caspase 1.
Her research in Molecular biology intersects with topics in Gene expression, Neurodegeneration, Endoplasmic reticulum, Bcl-2-associated X protein and Regulation of gene expression.
She has researched Caspase in several fields, including Enzyme activator and Small molecule.
Her studies deal with areas such as Mutation, Neuroprotection and Cytosol as well as Programmed cell death.
She most often published in these fields:
- Cell biology (46.87%)
- Caspase 6 (49.37%)
- Molecular biology (43.12%)
What were the highlights of her more recent work (between 2013-2021)?
- Caspase 6 (49.37%)
- Caspase (38.75%)
- Alzheimer's disease (33.12%)
In recent papers she was focusing on the following fields of study:
Caspase 6, Caspase, Alzheimer's disease, Neurodegeneration and Cognitive decline are her primary areas of study.
Her studies in Caspase 6 integrate themes in fields like Caspase 1, Cysteine protease and Cell biology.
She integrates many fields in her works, including Cell biology and Synaptic plasticity.
Her Caspase study introduces a deeper knowledge of Biochemistry.
Her Alzheimer's disease research includes elements of Cancer research, In vitro and Episodic memory.
Her Neurodegeneration research focuses on subjects like Neuroscience, which are linked to Immunolabeling.
Between 2013 and 2021, her most popular works were:
- Neuronal NLRP1 inflammasome activation of Caspase-1 coordinately regulates inflammatory interleukin-1-beta production and axonal degeneration-associated Caspase-6 activation (132 citations)
- Neuronal NLRP1 inflammasome activation of Caspase-1 coordinately regulates inflammatory interleukin-1-beta production and axonal degeneration-associated Caspase-6 activation (132 citations)
- Caspase-1 inhibition alleviates cognitive impairment and neuropathology in an Alzheimer's disease mouse model. (79 citations)
In her most recent research, the most cited papers focused on:
Andréa C. LeBlanc focuses on Neuroscience, Caspase, Biochemistry, Memory impairment and Amyloid beta.
Her Neuroscience research is multidisciplinary, incorporating elements of Alzheimer's disease, Neural development and Neurodegeneration.
She works mostly in the field of Neurodegeneration, limiting it down to topics relating to Hippocampus and, in certain cases, Caspase 6, as a part of the same area of interest.
Her Caspase 6 research integrates issues from Inflammasome and AIM2.
Her studies in Caspase integrate themes in fields like Neurite, Wallerian degeneration, Axon, Cysteine and Cytoskeleton.
Andréa C. LeBlanc has researched Amyloid beta in several fields, including Cell biology, Neuroinflammation, Caspase 1 and NLRP1.
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