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Neuroscience

D-Index
68
Citations
14617
World Ranking
2776
National Ranking
272

Overview

William F. Blakemore is affiliated with the University of Cambridge in the United Kingdom. Their professional role centers on academic research within this institution.

There are no specific recent papers available to detail their publication record. Similarly, information about frequent co-authors or common publication venues has not been provided.

Data regarding fields and subfields of study, as well as main topics of research work, are not available for this individual. Likewise, there are no records of book publications or awards won at this time.

Given the information available, William F. Blakemore's profile reflects an academic association without further publicly documented details on research output or contributions.

Best Publications

  • Response of the oligodendrocyte progenitor cell population (defined by NG2 labelling) to demyelination of the adult spinal cord.

    Hans S. Keirstead;Joel M. Levine;William F. Blakemore

  • Repair of demyelinated lesions by transplantation of purified O-2A progenitor cells

    A. K. Groves;S. C. Barnett;R. J. M. Franklin;A. J. Crang

  • Remyelination protects axons from demyelination-associated axon degeneration.

    K. A. Irvine;W. F. Blakemore

  • Central remyelination restores secure conduction

    Smith Kj;Blakemore Wf;McDonald Wi

  • Remyelination occurs as extensively but more slowly in old rats compared to young rats following gliotoxin-induced CNS demyelination.

    Simon A. Shields;Jennifer M. Gilson;William F. Blakemore;Robin J.M. Franklin

  • Identification of post-mitotic oligodendrocytes incapable of remyelination within the demyelinated adult spinal cord.

    Hans S. Keirstead;William F. Blakemore

  • Remyelination in Experimental Models of Toxin-Induced Demyelination

    W. F. Blakemore;R. J. M. Franklin

  • Lines of Murine Oligodendroglial Precursor Cells Immortalized by an Activated neu Tyrosine Kinase Show Distinct Degrees of Interaction with Axons In Vitro and In Vivo

    M. Jung;E. Krämer;M. Grzenkowski;K. Tang

  • Inflammation stimulates remyelination in areas of chronic demyelination

    A. K. Foote;W. F. Blakemore

  • Locomotor deficits induced by experimental spinal cord demyelination are abolished by spontaneous remyelination.

    N D Jeffery;W F Blakemore

  • Remyelination of CNS axons by Schwann cells transplanted from the sciatic nerve

    Blakemore Wf

  • Local recruitment of remyelinating cells in the repair of demyelination in the central nervous system.

    Robin J.M. Franklin;Jennifer M. Gilson;William F. Blakemore

  • Remyelination of mouse spinal cord axons demyelinated by local injection of lysolecithin.

    N D Jeffery;W F Blakemore

  • Observations on oligodendrocyte degeneration, the resolution of status spongiosus and remyelination in cuprizone intoxication in mice

    Unknown

  • The origin of remyelinating cells in the central nervous system

    William F Blakemore;Hans S Keirstead

  • THE RESTORATION OF CONDUCTION BY CENTRAL REMYELINATION

    K. J. Smith;W. F. Blakemore;W. I. Mcdonald

  • Requirements for Schwann cell migration within CNS environments: a viewpoint.

    R.J.M. Franklin;W.F. Blakemore

  • The role of oligodendrocytes and oligodendrocyte progenitors in CNS remyelination.

    Hans S. Keirstead;William F. Blakemore

  • Polysialylated Neural Cell Adhesion Molecule-Positive CNS Precursors Generate Both Oligodendrocytes and Schwann Cells to Remyelinate the CNS after Transplantation

    H. S. Keirstead;T. Ben-Hur;B. Rogister;B. Rogister;M. T. O’Leary

  • Transplanted CG4 Cells (an Oligodendrocyte Progenitor Cell Line) Survive, Migrate, and Contribute to Repair of Areas of Demyelination in X-Irradiated and Damaged Spinal Cord but Not in Normal Spinal Cord

    Robin J.M. Franklin;Susan A. Bayley;William F. Blakemore

  • Efficient recolonisation of progenitor-depleted areas of the CNS by adult oligodendrocyte progenitor cells.

    Divya M. Chari;William F. Blakemore

  • Transplanted type-1 astrocytes facilitate repair of demyelinating lesions by host oligodendrocytes in adult rat spinal cord

    R. J. M. Franklin;A. J. Crang;W. F. Blakemore

  • Locomotor deficits induced by experimental spinal cord demyelination are abolished by spontaneous

    N. D. Jeffery;W. F. Blakemore;Mrc Cambridge

Frequent Co-Authors

Susan C. Barnett
Susan C. Barnett University of Glasgow
Melitta Schachner
Melitta Schachner Rutgers, The State University of New Jersey
Anne Dell
Anne Dell Imperial College London
Chao Zhao
Chao Zhao University of Cambridge
Joel M. Levine
Joel M. Levine Stony Brook University
Olivier Raineteau
Olivier Raineteau Claude Bernard University Lyon 1
James W. Fawcett
James W. Fawcett University of Cambridge
Anne Elizabeth Rosser
Anne Elizabeth Rosser Cardiff University
Helmut Kettenmann
Helmut Kettenmann Shenzhen Institutes of Advanced Technology

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Best Scientists Citing William F. Blakemore