What is he best known for?
The fields of study he is best known for:
Walter J. Atwood mainly focuses on Virology, Virus, JC virus, Progressive multifocal leukoencephalopathy and BK virus.
His Virology study focuses on Capsid in particular.
His studies deal with areas such as Tumor necrosis factor alpha, Neuroglia, microRNA, Endocytosis and Gene silencing as well as Virus.
His JC virus study integrates concerns from other disciplines, such as Virus latency, Monoclonal antibody therapy, Glycolipid, Biochemistry and Entry into host.
The concepts of his Progressive multifocal leukoencephalopathy study are interwoven with issues in Demyelinating disease, Stromal cell, Immune system and Oligodendrocyte.
The various areas that Walter J. Atwood examines in his BK virus study include RNA, Gene expression, Immunology and Leflunomide.
His most cited work include:
- The human polyomavirus, JCV, uses serotonin receptors to infect cells. (389 citations)
- Molecular biology, epidemiology, and pathogenesis of progressive multifocal leukoencephalopathy, the JC virus-induced demyelinating disease of the human brain. (266 citations)
- JC virus infection of hematopoietic progenitor cells, primary B lymphocytes, and tonsillar stromal cells: implications for viral latency. (256 citations)
What are the main themes of his work throughout his whole career to date?
His main research concerns Virology, JC virus, Virus, Progressive multifocal leukoencephalopathy and BK virus.
His study in Virology is interdisciplinary in nature, drawing from both Cell culture, Sialic acid, Polyomavirus Infections, Immunology and Receptor.
His work on JC Virus Infection as part of general JC virus study is frequently linked to Polyomaviridae, therefore connecting diverse disciplines of science.
The study incorporates disciplines such as Neuroglia and Cell biology in addition to Virus.
His work deals with themes such as Demyelinating disease, Immunosuppression, Immune system, Tissue tropism and Choroid plexus, which intersect with Progressive multifocal leukoencephalopathy.
Walter J. Atwood has included themes like Vero cell and Leflunomide in his BK virus study.
He most often published in these fields:
- Virology (82.30%)
- JC virus (46.90%)
- Virus (46.90%)
What were the highlights of his more recent work (between 2012-2020)?
- Virology (82.30%)
- Progressive multifocal leukoencephalopathy (38.94%)
- Virus (46.90%)
In recent papers he was focusing on the following fields of study:
The scientist’s investigation covers issues in Virology, Progressive multifocal leukoencephalopathy, Virus, JC virus and Demyelinating disease.
His biological study spans a wide range of topics, including Sialic acid and Gene.
His Progressive multifocal leukoencephalopathy research includes themes of Receptor, Disease and Immune system.
His Virus study incorporates themes from HEK 293 cells, Amino acid, BK Virus Infection and Cell type.
His JC virus study integrates concerns from other disciplines, such as Cell culture, BK virus, Polyomavirus Infections, Innate immune system and Choroid plexus.
The Demyelinating disease study combines topics in areas such as Cell, Immunosuppression and Recombinant DNA.
Between 2012 and 2020, his most popular works were:
- Genome-wide siRNA screen identifies the retromer as a cellular entry factor for human papillomavirus (133 citations)
- 5-HT2 Receptors Facilitate JC Polyomavirus Entry (53 citations)
- A Retrograde Trafficking Inhibitor of Ricin and Shiga-Like Toxins Inhibits Infection of Cells by Human and Monkey Polyomaviruses (53 citations)
In his most recent research, the most cited papers focused on:
His primary scientific interests are in Virology, Progressive multifocal leukoencephalopathy, Sialic acid, Demyelinating disease and Receptor.
His Virology research is multidisciplinary, incorporating elements of Golgi apparatus, RNA interference and Late endosome.
His Progressive multifocal leukoencephalopathy research is under the purview of Virus.
His Virus study is concerned with Immunology in general.
His Sialic acid study combines topics in areas such as Viral protein and Recombinant DNA.
The concepts of his Demyelinating disease study are interwoven with issues in Immunosuppression and Immune system.
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