His scientific interests lie mostly in Cell biology, Extracellular, Connexin, Secretion and Gap junction. His study on Cell biology is mostly dedicated to connecting different topics, such as Calcium. The concepts of his Extracellular study are interwoven with issues in Biophysics and Membrane transport.
His Connexin study combines topics from a wide range of disciplines, such as Endocrinology, Internal medicine and Osteoblast. His Secretion research is multidisciplinary, relying on both Intracellular and Fura-2. His work carried out in the field of Gap junction brings together such families of science as Molecular biology and Transfection.
His primary areas of investigation include Cell biology, Extracellular, Biochemistry, Gap junction and Internal medicine. His work deals with themes such as Calcium and Transfection, which intersect with Cell biology. As a part of the same scientific study, Thomas H. Steinberg usually deals with the Extracellular, concentrating on Cytotoxicity and frequently concerns with Apoptosis and Lysis.
In general Biochemistry study, his work on Intracellular, Membrane transport and Cytoplasm often relates to the realm of Organic anion transport, thereby connecting several areas of interest. His Gap junction study combines topics from a wide range of disciplines, such as Molecular biology, Membrane, Immunostaining and Cell–cell interaction. In Internal medicine, he works on issues like Endocrinology, which are connected to Osteoblast.
The scientist’s investigation covers issues in Cell biology, Purinergic receptor, Internal medicine, Immunology and Second messenger system. His study in Calcium signaling and Gap junction falls under the purview of Cell biology. The study incorporates disciplines such as Cell signaling and Osteoblast in addition to Calcium signaling.
His Internal medicine research is multidisciplinary, incorporating elements of Endocrinology, Surgery and Malaria. Thomas H. Steinberg has included themes like Extracellular, Osteoclast, Multinucleate and Macrophage fusion in his Immunology study. His Second messenger system research integrates issues from Protein kinase A and Phosphorylation.
His primary scientific interests are in Cell biology, Gap junction, Purinergic receptor, Osteoblast and Cell signaling. Thomas H. Steinberg works in the field of Cell biology, focusing on Second messenger system in particular. His Gap junction research is multidisciplinary, incorporating perspectives in Cardiomyopathy, Heart failure, Heart transplantation, Immunostaining and Pathology.
Purinergic receptor is a primary field of his research addressed under Internal medicine. His Osteoblast research includes elements of Autocrine signalling and Calcium signaling.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
ATP4- permeabilizes the plasma membrane of mouse macrophages to fluorescent dyes.
T H Steinberg;A S Newman;J A Swanson;S C Silverstein.
Journal of Biological Chemistry (1987)
Inhibition of Fura-2 sequestration and secretion with organic anion transport blockers.
F Di Virgilio;T.H Steinberg;S.C Silverstein.
Cell Calcium (1990)
Connexin43 Deficiency Causes Delayed Ossification, Craniofacial Abnormalities, and Osteoblast Dysfunction
Fernando Lecanda;Pamela M. Warlow;Sharmin Sheikh;Federico Furlan.
Journal of Cell Biology (2000)
Hypertension and prolonged vasoconstrictor signaling in RGS2-deficient mice
Scott P. Heximer;Russell H. Knutsen;Xiaoguang Sun;Kevin M. Kaltenbronn.
Journal of Clinical Investigation (2003)
Cell to Cell Communication in Response to Mechanical Stress via Bilateral Release of Atp and Utp in Polarized Epithelia
László Homolya;Thomas H. Steinberg;Richard C. Boucher.
Journal of Cell Biology (2000)
Gap junctional communication modulates gene expression in osteoblastic cells
Fernando Lecanda;Dwight A. Towler;Konstantinos Ziambaras;Su Li Cheng.
Molecular Biology of the Cell (1998)
ATP- and gap junction-dependent intercellular calcium signaling in osteoblastic cells
Niklas R. Jørgensen;Steven T. Geist;Roberto Civitelli;Thomas H. Steinberg.
Journal of Cell Biology (1997)
Connexin43 and connexin45 form gap junctions with different molecular permeabilities in osteoblastic cells.
T.H. Steinberg;R. Civitelli;S.T. Geist;A.J. Robertson.
The EMBO Journal (1994)
Connexin43 mediates direct intercellular communication in human osteoblastic cell networks.
R Civitelli;E C Beyer;P M Warlow;A J Robertson.
Journal of Clinical Investigation (1993)
Extracellular ATP4- promotes cation fluxes in the J774 mouse macrophage cell line.
T.H. Steinberg;S.C. Silverstein.
Journal of Biological Chemistry (1987)
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