What is he best known for?
The fields of study he is best known for:
The scientist’s investigation covers issues in Cell biology, Receptor, Extracellular, Purinergic receptor and Signal transduction.
His study in the field of Intracellular is also linked to topics like Neuroglia.
His work focuses on many connections between Receptor and other disciplines, such as Proinflammatory cytokine, that overlap with his field of interest in Enzyme-linked receptor, CD86, Transcription factor and Molecular biology.
His research integrates issues of Cytotoxic T cell and Programmed cell death in his study of Extracellular.
The study incorporates disciplines such as Dependence receptor and Fas ligand, Death-inducing signaling complex in addition to Signal transduction.
His studies deal with areas such as Inflammation, Apyrase and Purinergic signalling as well as P2Y receptor.
His most cited work include:
- Activation of microglial cells by beta-amyloid protein and interferon-gamma (1214 citations)
- Apoptosis signaling by death receptors (892 citations)
- The P2X7 receptor: a key player in IL-1 processing and release. (853 citations)
What are the main themes of his work throughout his whole career to date?
Davide Ferrari spends much of his time researching Cell biology, Receptor, Extracellular, Purinergic receptor and Intracellular.
Particularly relevant to Signal transduction is his body of work in Cell biology.
His Receptor study integrates concerns from other disciplines, such as Inflammation, Secretion, Adenosine and Immune system.
The various areas that Davide Ferrari examines in his Extracellular study include Calcium, Transmembrane protein, Stimulation, Cell adhesion molecule and Proinflammatory cytokine.
His work deals with themes such as Cytotoxic T cell, Molecular biology, Cell signaling and Immunology, which intersect with Purinergic receptor.
His work is dedicated to discovering how Intracellular, Depolarization are connected with Macrophage and other disciplines.
He most often published in these fields:
- Cell biology (69.49%)
- Receptor (45.76%)
- Extracellular (39.83%)
What were the highlights of his more recent work (between 2014-2021)?
- Purinergic signalling (18.64%)
- Adenosine receptor (11.86%)
- Cell biology (69.49%)
In recent papers he was focusing on the following fields of study:
Davide Ferrari focuses on Purinergic signalling, Adenosine receptor, Cell biology, Purinergic receptor and Inflammation.
His work in Adenosine receptor covers topics such as Adenosine which are related to areas like Neuroinflammation, Cell surface receptor and Neuroscience.
His Cell biology research focuses on Extracellular in particular.
His Purinergic receptor research is multidisciplinary, relying on both Cell signaling, Signal transduction, Cancer cell, Immunology and Atheroma.
His Inflammation research is multidisciplinary, incorporating perspectives in Receptor, In vitro, ATP Receptors and CD38.
His Receptor research includes elements of T cell and Peripheral blood mononuclear cell.
Between 2014 and 2021, his most popular works were:
- Purinergic signaling in scarring (43 citations)
- Purinergic Signaling During Immune Cell Trafficking (39 citations)
- Roles and Modalities of Ectonucleotidases in Remodeling the Multiple Myeloma Niche. (32 citations)
In his most recent research, the most cited papers focused on:
His scientific interests lie mostly in Cell biology, Purinergic signalling, Purinergic receptor, Cell signaling and Receptor.
Davide Ferrari has researched Cell biology in several fields, including Autocrine signalling, Microvesicles, Immunology, Adenosine receptor and Atheroma.
Davide Ferrari has included themes like Signal transduction, Chemokine receptor and Paracrine signalling in his Autocrine signalling study.
Davide Ferrari combines subjects such as Extracellular and Adenosine with his study of Atheroma.
His Purinergic signalling research is multidisciplinary, incorporating elements of Uridine triphosphate, P2Y receptor, Purinergic Agents and Fibrosis.
His research on Purinergic receptor concerns the broader Endocrinology.
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