Takeo Yoshikawa mainly focuses on Genetics, Single-nucleotide polymorphism, Schizophrenia, Bipolar disorder and Psychosis. Haplotype, Genetic association, Allele frequency, Linkage disequilibrium and Gene are subfields of Genetics in which his conducts study. Takeo Yoshikawa has researched Single-nucleotide polymorphism in several fields, including Proband, Allele and Candidate gene.
His biological study deals with issues like Microsatellite, which deal with fields such as Craniofacial, Chromosome and SNP. Takeo Yoshikawa interconnects Genotyping, Genetic linkage and Genome Scan in the investigation of issues within Bipolar disorder. The concepts of his Psychosis study are interwoven with issues in Endocrinology, Mutation, Pentosidine, Alternative splicing and Internal medicine.
His primary areas of study are Genetics, Schizophrenia, Single-nucleotide polymorphism, Internal medicine and Endocrinology. Many of his studies on Genetics involve topics that are commonly interrelated, such as Bipolar disorder. Takeo Yoshikawa combines subjects such as Quantitative trait locus, Psychosis, Prefrontal cortex and Autism spectrum disorder with his study of Schizophrenia.
The Single-nucleotide polymorphism study combines topics in areas such as Allele frequency and Candidate gene. His work carried out in the field of Internal medicine brings together such families of science as Corpus callosum and Oncology. The various areas that Takeo Yoshikawa examines in his Endocrinology study include Cholecystokinin, Receptor, Methamphetamine and Polyunsaturated fatty acid.
His primary scientific interests are in Genetics, Internal medicine, Schizophrenia, Endocrinology and Gene. His work on Genetics deals in particular with Candidate gene, Missense mutation, Allele, FABP7 and Penetrance. His studies deal with areas such as Genome-wide association study, Genetic variation and Copy-number variation as well as Candidate gene.
His Internal medicine study combines topics in areas such as Docosahexaenoic acid, IDH2, IDH1, Pathology and IDH3G. His Schizophrenia research is multidisciplinary, incorporating perspectives in Young adult, Psychosis, Case-control study, Genetic association and Autism spectrum disorder. His Endocrinology research is multidisciplinary, incorporating elements of Offspring, Corpus callosum and Polyunsaturated fatty acid.
Takeo Yoshikawa mainly investigates Genetics, Neuroscience, Gene, Autism spectrum disorder and Autism. Genome-wide association study and Genetic association are among the areas of Genetics where Takeo Yoshikawa concentrates his study. His research in Genome-wide association study intersects with topics in Polymorphism and Candidate gene.
His study in Candidate gene is interdisciplinary in nature, drawing from both Neurodevelopmental disorder, SNP array, Single-nucleotide polymorphism, Comparative genomic hybridization and Genetic variation. His research investigates the connection between Genetic association and topics such as In silico that intersect with problems in Schizophrenia. His biological study spans a wide range of topics, including Synaptic plasticity and Induced pluripotent stem cell.
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Genome-wide association study identifies common variants at four loci as genetic risk factors for Parkinson's disease
Wataru Satake;Yuko Nakabayashi;Yuko Nakabayashi;Ikuko Mizuta;Ikuko Mizuta;Yushi Hirota;Yushi Hirota.
Nature Genetics (2009)
Genome scan meta-analysis of schizophrenia and bipolar disorder, part III: Bipolar disorder.
Ricardo Segurado;Sevilla D. Detera-Wadleigh;Douglas F. Levinson;Cathryn M. Lewis.
American Journal of Human Genetics (2003)
A high-density genome scan detects evidence for a bipolar-disorder susceptibility locus on 13q32 and other potential loci on 1q32 and 18p11.2
Sevilla D. Detera-Wadleigh;Judith A. Badner;Judith A. Badner;Wade H. Berrettini;Takeo Yoshikawa.
Proceedings of the National Academy of Sciences of the United States of America (1999)
Increased L1 Retrotransposition in the Neuronal Genome in Schizophrenia
Miki Bundo;Manabu Toyoshima;Yohei Okada;Wado Akamatsu.
DNA Methylation Status of SOX10 Correlates with Its Downregulation and Oligodendrocyte Dysfunction in Schizophrenia
Kazuya Iwamoto;Miki Bundo;Kazuo Yamada;Hitomi Takao.
The Journal of Neuroscience (2005)
Autistic-like phenotypes in Cadps2-knockout mice and aberrant CADPS2 splicing in autistic patients.
Tetsushi Sadakata;Miwa Washida;Yoshimi Iwayama;Satoshi Shoji.
Journal of Clinical Investigation (2007)
Two genetic variants of CD38 in subjects with autism spectrum disorder and controls.
Toshio Munesue;Shigeru Yokoyama;Kazuhiko Nakamura;Ayyappan Anitha.
Neuroscience Research (2010)
Low serum levels of brain-derived neurotrophic factor and epidermal growth factor in patients with chronic schizophrenia.
Yumiko Ikeda;Noriaki Yahata;Itsuo Ito;Masatoshi Nagano.
Schizophrenia Research (2008)
Genome-wide profiling of promoter methylation in human
Izuho Hatada;Masayuki Fukasawa;Mika Kimura;Sumiyo Morita.
Fabp7 Maps to a Quantitative Trait Locus for a Schizophrenia Endophenotype
Akiko Watanabe;Tomoko Toyota;Yuji Owada;Takeshi Hayashi.
PLOS Biology (2007)
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