D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Genetics D-index 49 Citations 6,148 132 World Ranking 3243 National Ranking 146

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Genetics
  • Enzyme

His primary areas of investigation include Genetics, Single-nucleotide polymorphism, Haplotype, Psychosis and Bipolar disorder. His study focuses on the intersection of Genetics and fields such as Schizophrenia with connections in the field of Relapse prevention. His study in Single-nucleotide polymorphism is interdisciplinary in nature, drawing from both Internal medicine, Proband, Allele frequency and Candidate gene.

His Psychosis research is multidisciplinary, relying on both Transmission disequilibrium test, Mutation and Alternative splicing. His Bipolar disorder research is multidisciplinary, incorporating perspectives in Human mitochondrial genetics and Genotyping. His research integrates issues of Molecular biology and Promoter in his study of Allele.

His most cited work include:

  • DNA Methylation Status of SOX10 Correlates with Its Downregulation and Oligodendrocyte Dysfunction in Schizophrenia (204 citations)
  • Fabp7 Maps to a Quantitative Trait Locus for a Schizophrenia Endophenotype (154 citations)
  • Two genetic variants of CD38 in subjects with autism spectrum disorder and controls. (146 citations)

What are the main themes of his work throughout his whole career to date?

The scientist’s investigation covers issues in Genetics, Single-nucleotide polymorphism, Haplotype, Schizophrenia and Genetic association. His Genetics research includes elements of Bipolar disorder and Psychosis. The Bipolar disorder study combines topics in areas such as Mood disorders and DISC1.

Within one scientific family, Kazuo Yamada focuses on topics pertaining to Candidate gene under Single-nucleotide polymorphism, and may sometimes address concerns connected to Polymorphism. As a part of the same scientific family, Kazuo Yamada mostly works in the field of Haplotype, focusing on Promoter and, on occasion, Molecular biology and NMDA receptor. He studied Schizophrenia and Internal medicine that intersect with Psychiatry.

He most often published in these fields:

  • Genetics (67.86%)
  • Single-nucleotide polymorphism (37.86%)
  • Haplotype (25.00%)

What were the highlights of his more recent work (between 2011-2017)?

  • Genetics (67.86%)
  • Single-nucleotide polymorphism (37.86%)
  • Genetic association (23.57%)

In recent papers he was focusing on the following fields of study:

Kazuo Yamada mainly investigates Genetics, Single-nucleotide polymorphism, Genetic association, Schizophrenia and Genome-wide association study. His Genetics study frequently intersects with other fields, such as Autism. His studies deal with areas such as Young adult, Bipolar disorder and Theory of mind as well as Single-nucleotide polymorphism.

He has included themes like Alternative medicine, Guideline, Mood disorders and Electroconvulsive therapy in his Bipolar disorder study. Kazuo Yamada combines subjects such as Psychosis, Social cognition, Zinc finger protein 804A, Human genetics and Haplotype with his study of Genetic association. His studies in Schizophrenia integrate themes in fields like SNP, Internal medicine, Dorsolateral prefrontal cortex and Endocrinology.

Between 2011 and 2017, his most popular works were:

  • Brain region-specific altered expression and association of mitochondria-related genes in autism (81 citations)
  • Genome-wide Association Study of Autism Spectrum Disorder in the East Asian Populations. (46 citations)
  • Protocadherin α(PCDHA ) as a novel susceptibility gene for autism (41 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Genetics

His scientific interests lie mostly in Genetics, Genome-wide association study, Single-nucleotide polymorphism, Autism and Psychiatry. His research combines Bipolar disorder and Genetics. His biological study spans a wide range of topics, including SNP, Schizophrenia, Dorsolateral prefrontal cortex, Japanese population and KCNJ3.

His work deals with themes such as Neurodevelopmental disorder, Genetic variation, Autism spectrum disorder and Candidate gene, which intersect with Single-nucleotide polymorphism. Kazuo Yamada has researched Autism in several fields, including Linkage disequilibrium, Gene, Human genetics, Genetic association and Mitochondrion. His Schizophrenia and Electroconvulsive therapy study, which is part of a larger body of work in Psychiatry, is frequently linked to Association, bridging the gap between disciplines.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

DNA Methylation Status of SOX10 Correlates with Its Downregulation and Oligodendrocyte Dysfunction in Schizophrenia

Kazuya Iwamoto;Miki Bundo;Kazuo Yamada;Hitomi Takao.
The Journal of Neuroscience (2005)

259 Citations

Two genetic variants of CD38 in subjects with autism spectrum disorder and controls.

Toshio Munesue;Shigeru Yokoyama;Kazuhiko Nakamura;Ayyappan Anitha.
Neuroscience Research (2010)

203 Citations

Fabp7 Maps to a Quantitative Trait Locus for a Schizophrenia Endophenotype

Akiko Watanabe;Tomoko Toyota;Yuji Owada;Takeshi Hayashi.
PLOS Biology (2007)

190 Citations

Identification of multiple serine racemase (SRR) mRNA isoforms and genetic analyses of SRR and DAO in schizophrenia and D-serine levels.

Kazuo Yamada;Tetsuo Ohnishi;Kenji Hashimoto;Hisako Ohba.
Biological Psychiatry (2005)

170 Citations

Genome-wide profiling of promoter methylation in human

Izuho Hatada;Masayuki Fukasawa;Mika Kimura;Sumiyo Morita.
Oncogene (2006)

166 Citations

Genetic analysis of the calcineurin pathway identifies members of the EGR gene family, specifically EGR3, as potential susceptibility candidates in schizophrenia.

Kazuo Yamada;David J. Gerber;Yoshimi Iwayama;Tetsuo Ohnishi.
Proceedings of the National Academy of Sciences of the United States of America (2007)

163 Citations

Association between polymorphisms in the promoter region of the sialyltransferase 8B (SIAT8B) gene and schizophrenia.

Makoto Arai;Kazuo Yamada;Tomoko Toyota;Nanako Obata.
Biological Psychiatry (2006)

155 Citations

No association between the Val66Met polymorphism of the brain-derived neurotrophic factor gene and bipolar disorder in a Japanese population: a multicenter study.

Hiroshi Kunugi;Yoshimi Iijima;Masahiko Tatsumi;Mariko Yoshida.
Biological Psychiatry (2004)

146 Citations

A microsatellite repeat in the promoter of the N-methyl-D-aspartate receptor 2A subunit (GRIN2A) gene suppresses transcriptional activity and correlates with chronic outcome in schizophrenia.

Masanari Itokawa;Kazuo Yamada;Kiyoshi Yoshitsugu;Tomoko Toyota.
Pharmacogenetics (2003)

135 Citations

Genomewide high-density SNP linkage analysis of 236 Japanese families supports the existence of schizophrenia susceptibility loci on chromosomes 1p, 14q, and 20p.

Tadao Arinami;Tsuyuka Ohtsuki;Hiroki Ishiguro;Hiroshi Ujike.
American Journal of Human Genetics (2005)

135 Citations

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