His primary scientific interests are in Biochemistry, Glutathione, Oxidative stress, Astrocyte and Metabolism. His work in Glutathione reductase, Reactive oxygen species, Incubation, Intracellular and GPX4 is related to Biochemistry. His research in Glutathione reductase intersects with topics in GPX3 and GPX1.
His Glutathione study combines topics from a wide range of disciplines, such as Extracellular, Cell biology, Molecular biology and Antioxidant. The Oxidative stress study combines topics in areas such as Homeostasis, DMT1, Glutamate receptor, Neurodegeneration and Neuroprotection. His Astrocyte study integrates concerns from other disciplines, such as Ferric, Signal transduction, Neuroglia and Gap junction.
The scientist’s investigation covers issues in Biochemistry, Glutathione, Metabolism, Oxidative stress and Astrocyte. His biological study spans a wide range of topics, including Biophysics and Neuroglia. Ralf Dringen combines subjects such as Molecular biology, Catalase and Cell biology with his study of Glutathione.
His Metabolism research incorporates elements of Amino acid, Ferritin, Heme oxygenase and Multidrug Resistance Protein 1. The concepts of his Oxidative stress study are interwoven with issues in Glutamine synthetase and Neurodegeneration. Ralf Dringen works mostly in the field of Astrocyte, limiting it down to concerns involving Ferric and, occasionally, Ferrous and Transferrin.
His primary areas of study are Biochemistry, Biophysics, Metabolism, Glutathione and Viability assay. His Biochemistry research incorporates themes from Toxicity and Copper. His Biophysics study incorporates themes from Nanoparticle, Iron oxide nanoparticles, Protein Corona, Zeta potential and Intracellular.
His work is dedicated to discovering how Metabolism, Primary are connected with Enzyme and Metabolite and other disciplines. Specifically, his work in Glutathione is concerned with the study of Glutathione disulfide. His Viability assay study combines topics in areas such as Live cell imaging and Incubation.
His scientific interests lie mostly in Biochemistry, Biophysics, Oxidative stress, Toxicity and Viability assay. His studies in Glutathione, Endocytosis and Metabolism are all subfields of Biochemistry research. His research in Glutathione focuses on subjects like Glycolysis, which are connected to Stimulation, Astrocyte, Extracellular, Arsenite and Dehydrogenase.
His studies in Biophysics integrate themes in fields like Iron oxide nanoparticles, Nanotechnology, Biocompatibility, Protein Corona and Zeta potential. His Oxidative stress research incorporates themes from Liberation and Reactive oxygen species. His research in Toxicity intersects with topics in Cell biology and Copper.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Metabolism and functions of glutathione in brain.
Ralf Dringen.
Progress in Neurobiology (2000)
Glutathione metabolism in brain
Ralf Dringen;Jan M Gutterer;Johannes Hirrlinger.
FEBS Journal (2000)
Synthesis of the antioxidant glutathione in neurons: supply by astrocytes of CysGly as precursor for neuronal glutathione.
Ralf Dringen;Brigitte Pfeiffer;Bernd Hamprecht.
The Journal of Neuroscience (1999)
Glutathione pathways in the brain
Ralf Dringen;Johannes Hirrlinger.
Biological Chemistry (2003)
Glycogen in astrocytes: possible function as lactate supply for neighboring cells
Ralf Dringen;Rolf Gebhardt;Bernd Hamprecht.
Brain Research (1993)
Astrocytes: glutamate producers for neurons.
Leif Hertz;Ralf Dringen;Arne Schousboe;Stephen R. Robinson.
Journal of Neuroscience Research (1999)
Peroxide detoxification by brain cells.
Ralf Dringen;Ralf Dringen;Petra G. Pawlowski;Johannes Hirrlinger.
Journal of Neuroscience Research (2005)
Colorimetric ferrozine-based assay for the quantitation of iron in cultured cells.
Jan Riemer;Jan Riemer;Hans Hermann Hoepken;Hans Hermann Hoepken;Hania Barbara Czerwinska;Stephen R Robinson.
Analytical Biochemistry (2004)
Fumarates improve psoriasis and multiple sclerosis by inducing type II dendritic cells
Kamran Ghoreschi;Jürgen Brück;Christina Kellerer;Caishu Deng.
Journal of Experimental Medicine (2011)
bcl-2 transgene expression can protect neurons against developmental and induced cell death.
Peter G. Farlie;Ralf Dringen;Sandra M. Rees;George Kannourakis.
Proceedings of the National Academy of Sciences of the United States of America (1995)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
University of Tübingen
University of Bremen
TU Dresden
RWTH Aachen University
Washington University in St. Louis
New York Medical College
Universidade Nova de Lisboa
Deakin University
Universidade Nova de Lisboa
University of Groningen
Quaid-i-Azam University
Smith-Kettlewell Eye Research Institute
Max Planck Society
Kyoto University
Massey University
Google (United States)
Genentech
Innsbruck Medical University
University of California, Los Angeles
The University of Texas at Austin
The Open University
University of North Carolina at Chapel Hill
Boston University
Mohammed Bin Rashid University of Medicine and Health Sciences
Johns Hopkins University
National Institute for Astrophysics