D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 73 Citations 21,551 160 World Ranking 1244 National Ranking 638

Research.com Recognitions

Awards & Achievements

2012 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Immune system

His primary scientific interests are in Microglia, Immunology, Spinal cord injury, Neuroinflammation and Spinal cord. His work deals with themes such as Proinflammatory cytokine, Gliosis, Neuroscience and Monocyte, which intersect with Microglia. His Immunology research includes elements of Myelin, Endocrinology, Functional recovery and Internal medicine.

His Spinal cord injury study combines topics from a wide range of disciplines, such as Wound healing, Infiltration, Pathology and Central nervous system. His Neuroinflammation study necessitates a more in-depth grasp of Inflammation. His Spinal cord research integrates issues from Central nervous system disease, Neuroprotection and Anatomy.

His most cited work include:

  • Identification of two distinct macrophage subsets with divergent effects causing either neurotoxicity or regeneration in the injured mouse spinal cord. (1416 citations)
  • Basso Mouse Scale for locomotion detects differences in recovery after spinal cord injury in five common mouse strains. (872 citations)
  • Cellular inflammatory response after spinal cord injury in Sprague-Dawley and Lewis rats. (782 citations)

What are the main themes of his work throughout his whole career to date?

Phillip G. Popovich spends much of his time researching Spinal cord injury, Immunology, Microglia, Neuroscience and Spinal cord. His Spinal cord injury research incorporates elements of Lesion, Pathology, Anesthesia, Neuroprotection and Immune system. His studies in Immunology integrate themes in fields like Progenitor cell, Myelin, Central nervous system, Gliosis and Cell biology.

His Microglia study combines topics in areas such as Neuroinflammation, Endocrinology and Monocyte. His study in the fields of Axon and Dorsal root ganglion under the domain of Neuroscience overlaps with other disciplines such as Context and Neurotoxicity. His work is dedicated to discovering how Spinal cord, Anatomy are connected with Central nervous system disease and other disciplines.

He most often published in these fields:

  • Spinal cord injury (50.87%)
  • Immunology (35.84%)
  • Microglia (33.53%)

What were the highlights of his more recent work (between 2017-2021)?

  • Spinal cord injury (50.87%)
  • Spinal cord (30.06%)
  • Inflammation (21.97%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Spinal cord injury, Spinal cord, Inflammation, Microglia and Neuroscience. His Spinal cord injury research is multidisciplinary, incorporating perspectives in Lesion, Pathology, Immune system and Neuroprotection. His Immune system study results in a more complete grasp of Immunology.

His Spinal cord study incorporates themes from Paralysis, Axon and Regeneration. Neuroinflammation is the focus of his Inflammation research. His Microglia study integrates concerns from other disciplines, such as Phagocytosis, Receptor, Signal transduction, Monocyte and Neurogenesis.

Between 2017 and 2021, his most popular works were:

  • MicroRNAs: Roles in Regulating Neuroinflammation: (93 citations)
  • Cell-Type-Specific Interleukin 1 Receptor 1 Signaling in the Brain Regulates Distinct Neuroimmune Activities. (47 citations)
  • Traumatic brain injury-induced neuronal damage in the somatosensory cortex causes formation of rod-shaped microglia that promote astrogliosis and persistent neuroinflammation. (37 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Immune system

Phillip G. Popovich mainly investigates Spinal cord injury, Immune system, Inflammation, Neuroinflammation and Microglia. His Spinal cord injury study is concerned with Spinal cord in general. His biological study spans a wide range of topics, including microRNA and Nervous system.

His Inflammation research incorporates themes from Somatosensory system, Axotomy, Physical medicine and rehabilitation, Forelimb and Polytrauma. He interconnects Cerebral cortex, Rehabilitation, Electrophysiology and Astrogliosis in the investigation of issues within Neuroinflammation. His Microglia research is multidisciplinary, incorporating elements of Fibronectin, Cord, Axon, Neuroscience and Crush injury.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Identification of two distinct macrophage subsets with divergent effects causing either neurotoxicity or regeneration in the injured mouse spinal cord.

Kristina A. Kigerl;John C. Gensel;Daniel P. Ankeny;Jessica K. Alexander.
The Journal of Neuroscience (2009)

2091 Citations

Basso Mouse Scale for locomotion detects differences in recovery after spinal cord injury in five common mouse strains.

D. Michele Basso;Lesley C. Fisher;Aileen J. Anderson;Lyn B. Jakeman.
Journal of Neurotrauma (2006)

1289 Citations

Cellular inflammatory response after spinal cord injury in Sprague-Dawley and Lewis rats.

Phillip G. Popovich;Ping Wei;Bradford T. Stokes.
The Journal of Comparative Neurology (1997)

1090 Citations

Inflammation and its role in neuroprotection, axonal regeneration and functional recovery after spinal cord injury

Dustin J. Donnelly;Phillip G. Popovich.
Experimental Neurology (2008)

1033 Citations

Wallerian degeneration: gaining perspective on inflammatory events after peripheral nerve injury

Andrew D Gaudet;Andrew D Gaudet;Phillip G Popovich;Matt S Ramer.
Journal of Neuroinflammation (2011)

803 Citations

Depletion of hematogenous macrophages promotes partial hindlimb recovery and neuroanatomical repair after experimental spinal cord injury.

P. G. Popovich;Zhen Guan;Ping Wei;I. Huitinga.
Experimental Neurology (1999)

796 Citations

Novel Markers to Delineate Murine M1 and M2 Macrophages

Kyle A. Jablonski;Stephanie A. Amici;Lindsay M. Webb;Juan de Dios Ruiz-Rosado.
PLOS ONE (2015)

726 Citations

Microglia induce motor neuron death via the classical NF-κB pathway in amyotrophic lateral sclerosis

Ashley E. Frakes;Laura Ferraiuolo;Amanda M. Haidet-Phillips;Leah Schmelzer.
Neuron (2014)

548 Citations

Cytokine mRNA Profiles in Contused Spinal Cord and Axotomized Facial Nucleus Suggest a Beneficial Role for Inflammation and Gliosis

Wolfgang J. Streit;Susan L. Semple-Rowland;Sean D. Hurley;Rehae C. Miller.
Experimental Neurology (1998)

424 Citations

Rats and mice exhibit distinct inflammatory reactions after spinal cord injury

Julie M. Sroga;T. Bucky Jones;Kristina A. Kigerl;Violeta M. McGaughy.
The Journal of Comparative Neurology (2003)

401 Citations

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