2012 - Fellow of the American Association for the Advancement of Science (AAAS)
His primary scientific interests are in Microglia, Immunology, Spinal cord injury, Neuroinflammation and Spinal cord. His work deals with themes such as Proinflammatory cytokine, Gliosis, Neuroscience and Monocyte, which intersect with Microglia. His Immunology research includes elements of Myelin, Endocrinology, Functional recovery and Internal medicine.
His Spinal cord injury study combines topics from a wide range of disciplines, such as Wound healing, Infiltration, Pathology and Central nervous system. His Neuroinflammation study necessitates a more in-depth grasp of Inflammation. His Spinal cord research integrates issues from Central nervous system disease, Neuroprotection and Anatomy.
Phillip G. Popovich spends much of his time researching Spinal cord injury, Immunology, Microglia, Neuroscience and Spinal cord. His Spinal cord injury research incorporates elements of Lesion, Pathology, Anesthesia, Neuroprotection and Immune system. His studies in Immunology integrate themes in fields like Progenitor cell, Myelin, Central nervous system, Gliosis and Cell biology.
His Microglia study combines topics in areas such as Neuroinflammation, Endocrinology and Monocyte. His study in the fields of Axon and Dorsal root ganglion under the domain of Neuroscience overlaps with other disciplines such as Context and Neurotoxicity. His work is dedicated to discovering how Spinal cord, Anatomy are connected with Central nervous system disease and other disciplines.
His primary areas of study are Spinal cord injury, Spinal cord, Inflammation, Microglia and Neuroscience. His Spinal cord injury research is multidisciplinary, incorporating perspectives in Lesion, Pathology, Immune system and Neuroprotection. His Immune system study results in a more complete grasp of Immunology.
His Spinal cord study incorporates themes from Paralysis, Axon and Regeneration. Neuroinflammation is the focus of his Inflammation research. His Microglia study integrates concerns from other disciplines, such as Phagocytosis, Receptor, Signal transduction, Monocyte and Neurogenesis.
Phillip G. Popovich mainly investigates Spinal cord injury, Immune system, Inflammation, Neuroinflammation and Microglia. His Spinal cord injury study is concerned with Spinal cord in general. His biological study spans a wide range of topics, including microRNA and Nervous system.
His Inflammation research incorporates themes from Somatosensory system, Axotomy, Physical medicine and rehabilitation, Forelimb and Polytrauma. He interconnects Cerebral cortex, Rehabilitation, Electrophysiology and Astrogliosis in the investigation of issues within Neuroinflammation. His Microglia research is multidisciplinary, incorporating elements of Fibronectin, Cord, Axon, Neuroscience and Crush injury.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Identification of two distinct macrophage subsets with divergent effects causing either neurotoxicity or regeneration in the injured mouse spinal cord.
Kristina A. Kigerl;John C. Gensel;Daniel P. Ankeny;Jessica K. Alexander.
The Journal of Neuroscience (2009)
Basso Mouse Scale for locomotion detects differences in recovery after spinal cord injury in five common mouse strains.
D. Michele Basso;Lesley C. Fisher;Aileen J. Anderson;Lyn B. Jakeman.
Journal of Neurotrauma (2006)
Cellular inflammatory response after spinal cord injury in Sprague-Dawley and Lewis rats.
Phillip G. Popovich;Ping Wei;Bradford T. Stokes.
The Journal of Comparative Neurology (1997)
Inflammation and its role in neuroprotection, axonal regeneration and functional recovery after spinal cord injury
Dustin J. Donnelly;Phillip G. Popovich.
Experimental Neurology (2008)
Wallerian degeneration: gaining perspective on inflammatory events after peripheral nerve injury
Andrew D Gaudet;Andrew D Gaudet;Phillip G Popovich;Matt S Ramer.
Journal of Neuroinflammation (2011)
Depletion of hematogenous macrophages promotes partial hindlimb recovery and neuroanatomical repair after experimental spinal cord injury.
P. G. Popovich;Zhen Guan;Ping Wei;I. Huitinga.
Experimental Neurology (1999)
Novel Markers to Delineate Murine M1 and M2 Macrophages
Kyle A. Jablonski;Stephanie A. Amici;Lindsay M. Webb;Juan de Dios Ruiz-Rosado.
PLOS ONE (2015)
Microglia induce motor neuron death via the classical NF-κB pathway in amyotrophic lateral sclerosis
Ashley E. Frakes;Laura Ferraiuolo;Amanda M. Haidet-Phillips;Leah Schmelzer.
Neuron (2014)
Cytokine mRNA Profiles in Contused Spinal Cord and Axotomized Facial Nucleus Suggest a Beneficial Role for Inflammation and Gliosis
Wolfgang J. Streit;Susan L. Semple-Rowland;Sean D. Hurley;Rehae C. Miller.
Experimental Neurology (1998)
Rats and mice exhibit distinct inflammatory reactions after spinal cord injury
Julie M. Sroga;T. Bucky Jones;Kristina A. Kigerl;Violeta M. McGaughy.
The Journal of Comparative Neurology (2003)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
The Ohio State University
The Ohio State University
The Ohio State University
West Virginia University
University of Arizona
University of California, San Francisco
The Ohio State University
University of Miami
Vrije Universiteit Amsterdam
The Ohio State University
Duke University
Northwestern University
Purdue University West Lafayette
Oak Ridge National Laboratory
Max Planck Society
Tulane University
Université Savoie Mont Blanc
Princeton University
Virginia Tech
University of California, Davis
National Institutes of Health
University of Bergen
University of Buenos Aires
Northwestern University
Johns Hopkins University
University of South Carolina