D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 95 Citations 26,566 239 World Ranking 545 National Ranking 313

Research.com Recognitions

Awards & Achievements

1986 - Fellow of John Simon Guggenheim Memorial Foundation

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Neuroscience
  • Neuron

His main research concerns Neuroscience, Hippocampal formation, Hippocampus, Inhibitory postsynaptic potential and Excitatory postsynaptic potential. His work in Pyramidal cell, Electrophysiology, Axon, Stimulation and Dentate gyrus are all subfields of Neuroscience research. His Hippocampal formation research is multidisciplinary, relying on both Long-term potentiation, Bursting, Depolarization and Intracellular.

His research in Hippocampus intersects with topics in Kainic acid, Cerebral cortex and Cerebellum. His Inhibitory postsynaptic potential research is multidisciplinary, incorporating elements of Osmotic concentration and Postsynaptic potential. His Excitatory postsynaptic potential study integrates concerns from other disciplines, such as Dendritic spine and Synapse.

His most cited work include:

  • Estradiol Increases the Sensitivity of Hippocampal CA1 Pyramidal Cells to NMDA Receptor-Mediated Synaptic Input: Correlation with Dendritic Spine Density (609 citations)
  • Heteromultimeric K+ channels in terminal and juxtaparanodal regions of neurons (541 citations)
  • Neurotrophin expression in rat hippocampal slices: a stimulus paradigm inducing LTP in CA1 evokes increases in BDNF and NT-3 mRNAs. (521 citations)

What are the main themes of his work throughout his whole career to date?

Philip A. Schwartzkroin spends much of his time researching Neuroscience, Hippocampal formation, Hippocampus, Epilepsy and Inhibitory postsynaptic potential. His Neuroscience study frequently links to other fields, such as Postsynaptic potential. The study incorporates disciplines such as Biophysics, Depolarization and Intracellular in addition to Hippocampal formation.

Philip A. Schwartzkroin has included themes like Kainic acid, Cerebral cortex, Central nervous system and Cell biology in his Hippocampus study. His Epilepsy study combines topics in areas such as Tuberous sclerosis and Pathology. His Inhibitory postsynaptic potential research incorporates elements of Bicuculline, GABAA receptor, Reversal potential and Neurotransmission.

He most often published in these fields:

  • Neuroscience (73.19%)
  • Hippocampal formation (39.15%)
  • Hippocampus (25.11%)

What were the highlights of his more recent work (between 2003-2017)?

  • Epilepsy (24.68%)
  • Neuroscience (73.19%)
  • Epileptogenesis (8.94%)

In recent papers he was focusing on the following fields of study:

His scientific interests lie mostly in Epilepsy, Neuroscience, Epileptogenesis, Hippocampal formation and Pathology. His Epilepsy study combines topics in areas such as Internal medicine, Tuberous sclerosis and Encyclopedia. His Neuroscience study frequently intersects with other fields, such as Knockout mouse.

The various areas that he examines in his Epileptogenesis study include GABAergic and Traumatic brain injury. His study in Hippocampal formation is interdisciplinary in nature, drawing from both Kainic acid, DNA, DNA damage, Apoptosis and Bicuculline. His Pathology study which covers Neocortex that intersects with Hippocampus.

Between 2003 and 2017, his most popular works were:

  • Models of Seizures and Epilepsy (412 citations)
  • Physiological and morphological characterization of dentate granule cells in the p35 knock-out mouse hippocampus: evidence for an epileptic circuit. (75 citations)
  • Deletion of the Kv2.1 delayed rectifier potassium channel leads to neuronal and behavioral hyperexcitability. (61 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Neuron
  • Neuroscience

His primary scientific interests are in Epilepsy, Neuroscience, Hippocampal formation, Bicuculline and Pathology. His Epilepsy study incorporates themes from Internal medicine, Tuberous sclerosis and Endocrinology. The concepts of his Neuroscience study are interwoven with issues in World Wide Web and Encyclopedia.

His Hippocampal formation research includes themes of Neuronal migration disorder, Flurothyl, Antidromic and Excitatory postsynaptic potential. His research investigates the connection between Bicuculline and topics such as Neocortex that intersect with problems in Hippocampus. His study explores the link between Pathology and topics such as Knockout mouse that cross with problems in Immunocytochemistry, Status epilepticus, Silver stain, Molecular biology and Rat model.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Estradiol increases the sensitivity of hippocampal CA1 pyramidal cells to NMDA receptor-mediated synaptic input: correlation with dendritic spine density.

Catherine S. Woolley;Nancy G. Weiland;Bruce S. McEwen;Philip A. Schwartzkroin.
The Journal of Neuroscience (1997)

824 Citations

Elimination of zinc from synaptic vesicles in the intact mouse brain by disruption of the ZnT3 gene

Toby B. Cole;H. Jürgen Wenzel;Kathy E. Kafer;Philip A. Schwartzkroin.
Proceedings of the National Academy of Sciences of the United States of America (1999)

739 Citations

Characteristics of CA1 neurons recorded intracellularly in the hippocampal in vitro slice preparation.

Philip A. Schwartzkroin.
Brain Research (1975)

730 Citations

Neurotrophin expression in rat hippocampal slices: a stimulus paradigm inducing LTP in CA1 evokes increases in BDNF and NT-3 mRNAs.

Susan L. Patterson;Larry M. Grover;Philip A. Schwartzkroin;Philip A. Schwartzkroin;Mark Bothwell.
Neuron (1992)

703 Citations

Heteromultimeric K+ channels in terminal and juxtaparanodal regions of neurons

Hao Wang;Dennis D. Kunkel;Troy M. Martin;Philip A. Schwartzkroin.
Nature (1993)

691 Citations

Deletion of the KV1.1 Potassium Channel Causes Epilepsy in Mice

Sharon L. Smart;Valeri Lopantsev;C.L. Zhang;Carol A. Robbins.
Neuron (1998)

616 Citations

Bax Involvement in p53-Mediated Neuronal Cell Death

Hong Xiang;Yoshito Kinoshita;C. Michael Knudson;Stanley J. Korsmeyer.
The Journal of Neuroscience (1998)

493 Citations

Electrophysiology of Hippocampal Neurons

Philip A. Schwartzkroin;Alan L. Mueller.
(1987)

486 Citations

Long-lasting facilitation of a synaptic potential following tetanization in the in vitro hippocampal slice.

Philip A. Schwartzkroin;Knut Wester.
Brain Research (1975)

481 Citations

Ultrastructural localization of zinc transporter-3 (ZnT-3) to synaptic vesicle membranes within mossy fiber boutons in the hippocampus of mouse and monkey

H. Jürgen Wenzel;Toby B. Cole;Donald E. Born;Philip A. Schwartzkroin.
Proceedings of the National Academy of Sciences of the United States of America (1997)

466 Citations

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