1986 - Fellow of John Simon Guggenheim Memorial Foundation
His main research concerns Neuroscience, Hippocampal formation, Hippocampus, Inhibitory postsynaptic potential and Excitatory postsynaptic potential. His work in Pyramidal cell, Electrophysiology, Axon, Stimulation and Dentate gyrus are all subfields of Neuroscience research. His Hippocampal formation research is multidisciplinary, relying on both Long-term potentiation, Bursting, Depolarization and Intracellular.
His research in Hippocampus intersects with topics in Kainic acid, Cerebral cortex and Cerebellum. His Inhibitory postsynaptic potential research is multidisciplinary, incorporating elements of Osmotic concentration and Postsynaptic potential. His Excitatory postsynaptic potential study integrates concerns from other disciplines, such as Dendritic spine and Synapse.
Philip A. Schwartzkroin spends much of his time researching Neuroscience, Hippocampal formation, Hippocampus, Epilepsy and Inhibitory postsynaptic potential. His Neuroscience study frequently links to other fields, such as Postsynaptic potential. The study incorporates disciplines such as Biophysics, Depolarization and Intracellular in addition to Hippocampal formation.
Philip A. Schwartzkroin has included themes like Kainic acid, Cerebral cortex, Central nervous system and Cell biology in his Hippocampus study. His Epilepsy study combines topics in areas such as Tuberous sclerosis and Pathology. His Inhibitory postsynaptic potential research incorporates elements of Bicuculline, GABAA receptor, Reversal potential and Neurotransmission.
His scientific interests lie mostly in Epilepsy, Neuroscience, Epileptogenesis, Hippocampal formation and Pathology. His Epilepsy study combines topics in areas such as Internal medicine, Tuberous sclerosis and Encyclopedia. His Neuroscience study frequently intersects with other fields, such as Knockout mouse.
The various areas that he examines in his Epileptogenesis study include GABAergic and Traumatic brain injury. His study in Hippocampal formation is interdisciplinary in nature, drawing from both Kainic acid, DNA, DNA damage, Apoptosis and Bicuculline. His Pathology study which covers Neocortex that intersects with Hippocampus.
His primary scientific interests are in Epilepsy, Neuroscience, Hippocampal formation, Bicuculline and Pathology. His Epilepsy study incorporates themes from Internal medicine, Tuberous sclerosis and Endocrinology. The concepts of his Neuroscience study are interwoven with issues in World Wide Web and Encyclopedia.
His Hippocampal formation research includes themes of Neuronal migration disorder, Flurothyl, Antidromic and Excitatory postsynaptic potential. His research investigates the connection between Bicuculline and topics such as Neocortex that intersect with problems in Hippocampus. His study explores the link between Pathology and topics such as Knockout mouse that cross with problems in Immunocytochemistry, Status epilepticus, Silver stain, Molecular biology and Rat model.
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Estradiol increases the sensitivity of hippocampal CA1 pyramidal cells to NMDA receptor-mediated synaptic input: correlation with dendritic spine density.
Catherine S. Woolley;Nancy G. Weiland;Bruce S. McEwen;Philip A. Schwartzkroin.
The Journal of Neuroscience (1997)
Elimination of zinc from synaptic vesicles in the intact mouse brain by disruption of the ZnT3 gene
Toby B. Cole;H. Jürgen Wenzel;Kathy E. Kafer;Philip A. Schwartzkroin.
Proceedings of the National Academy of Sciences of the United States of America (1999)
Characteristics of CA1 neurons recorded intracellularly in the hippocampal in vitro slice preparation.
Philip A. Schwartzkroin.
Brain Research (1975)
Neurotrophin expression in rat hippocampal slices: a stimulus paradigm inducing LTP in CA1 evokes increases in BDNF and NT-3 mRNAs.
Susan L. Patterson;Larry M. Grover;Philip A. Schwartzkroin;Philip A. Schwartzkroin;Mark Bothwell.
Heteromultimeric K+ channels in terminal and juxtaparanodal regions of neurons
Hao Wang;Dennis D. Kunkel;Troy M. Martin;Philip A. Schwartzkroin.
Deletion of the KV1.1 Potassium Channel Causes Epilepsy in Mice
Sharon L. Smart;Valeri Lopantsev;C.L. Zhang;Carol A. Robbins.
Bax Involvement in p53-Mediated Neuronal Cell Death
Hong Xiang;Yoshito Kinoshita;C. Michael Knudson;Stanley J. Korsmeyer.
The Journal of Neuroscience (1998)
Electrophysiology of Hippocampal Neurons
Philip A. Schwartzkroin;Alan L. Mueller.
Long-lasting facilitation of a synaptic potential following tetanization in the in vitro hippocampal slice.
Philip A. Schwartzkroin;Knut Wester.
Brain Research (1975)
Ultrastructural localization of zinc transporter-3 (ZnT-3) to synaptic vesicle membranes within mossy fiber boutons in the hippocampus of mouse and monkey
H. Jürgen Wenzel;Toby B. Cole;Donald E. Born;Philip A. Schwartzkroin.
Proceedings of the National Academy of Sciences of the United States of America (1997)
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