His primary areas of study are Cell biology, Tetraspanin, Cell adhesion, Molecular biology and Intercellular Adhesion Molecule-1. Peter N. Monk has researched Cell adhesion in several fields, including Endothelial stem cell, Cell migration, Cell adhesion molecule and Transmembrane protein. The study incorporates disciplines such as Amino acid and Binding site in addition to Molecular biology.
Peter N. Monk works mostly in the field of Amino acid, limiting it down to concerns involving COS cells and, occasionally, CD81. His study in Binding site is interdisciplinary in nature, drawing from both Protein G and G protein. Peter N. Monk has researched Intercellular Adhesion Molecule-1 in several fields, including Lipid raft, Integrin, Actin cytoskeleton and Cell membrane.
His scientific interests lie mostly in Cell biology, Receptor, C5a receptor, Tetraspanin and Biochemistry. His Cell biology research incorporates themes from Endothelial stem cell and Cell activation. The concepts of his Receptor study are interwoven with issues in Anaphylatoxin, Complement system and Transfection.
His C5a receptor study integrates concerns from other disciplines, such as 5-HT5A receptor, Complement C5a, Molecular biology, G protein and Binding site. His Tetraspanin study incorporates themes from Cell adhesion, CD81, Cell fusion and Transmembrane protein. His study explores the link between Cell adhesion and topics such as Intercellular Adhesion Molecule-1 that cross with problems in Cell migration.
His primary areas of investigation include Cell biology, Tetraspanin, Microbiology, Receptor and Immunology. The Cell biology study combines topics in areas such as Cytotoxic T cell, Innate immune system and Cell adhesion. In his works, Peter N. Monk performs multidisciplinary study on Tetraspanin and CRISPR.
His research in Receptor intersects with topics in Cell, C5a receptor, Anaphylatoxin, Complement system and Flow cytometry. His C5a receptor study combines topics from a wide range of disciplines, such as Arrestin, Complement C5a and Peptide. In CD81, Peter N. Monk works on issues like Conserved sequence, which are connected to Molecular biology.
Peter N. Monk spends much of his time researching Cell biology, Tetraspanin, Receptor, Cell and Glycosaminoglycan. Peter N. Monk has included themes like Integrin, beta 6, Integrin, Cell adhesion and Cytotoxic T cell in his Cell biology study. His research integrates issues of CD81, Conserved sequence, Molecular biology, Cell fusion and Giant cell in his study of Tetraspanin.
His Receptor research is multidisciplinary, relying on both C5a receptor and Complement system. His C5a receptor research focuses on subjects like Arrestin, which are linked to Immunology. His Cell research is multidisciplinary, incorporating perspectives in IC50, Flow cytometry and Heparin.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Characterization of Hepatitis C Virus E2 Glycoprotein Interaction with a Putative Cellular Receptor, CD81
Mike Flint;Catherine Maidens;Larry D. Loomis-Price;Christine Shotton.
Journal of Virology (1999)
CD81 is required for hepatitis C virus glycoprotein-mediated viral infection.
Jie Zhang;Glenn Randall;Adrian Higginbottom;Peter Monk.
Journal of Virology (2004)
Function, structure and therapeutic potential of complement C5a receptors
Monk Pn;Scola Am;Madala P;Fairlie Dp.
British Journal of Pharmacology (2007)
T helper 1 immunity requires complement-driven NLRP3 inflammasome activity in CD4+ T cells
Giuseppina Arbore;Erin E. West;Rosanne Spolski;Avril A. B. Robertson.
Cxc chemokine receptor expression on human endothelial cells.
Craig Murdoch;Peter N Monk;Adam Finn.
Identification of Amino Acid Residues in CD81 Critical for Interaction with Hepatitis C Virus Envelope Glycoprotein E2
Adrian Higginbottom;Elizabeth R. Quinn;Chiung-Chi Kuo;Mike Flint.
Journal of Virology (2000)
The Orphan Receptor C5L2 Has High Affinity Binding Sites for Complement Fragments C5a and C5a des-Arg74
Stuart A. Cain;Peter N. Monk.
Journal of Biological Chemistry (2002)
Endothelial adhesion receptors are recruited to adherent leukocytes by inclusion in preformed tetraspanin nanoplatforms
Olga Barreiro;Moreno Zamai;María Yáñez-Mó;Emilio Tejera.
Journal of Cell Biology (2008)
International Union of Basic and Clinical Pharmacology. LXXXVII. Complement Peptide C5a, C4a, and C3a Receptors
Andreas Klos;Elisabeth Wende;Kathryn J. Wareham;Peter N. Monk.
Pharmacological Reviews (2013)
Microglia as potential contributors to motor neuron injury in amyotrophic lateral sclerosis.
Siranush A. Sargsyan;Peter N. Monk;Pamela J. Shaw.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: