Her main research concerns Neuroscience, Cholinergic, Synaptic plasticity, Striatum and Long-term potentiation. Her Neuroscience research is multidisciplinary, incorporating perspectives in Metabotropic glutamate receptor 1, Metabotropic glutamate receptor and Metabotropic receptor. She combines subjects such as Glutamatergic, Dopamine receptor D2, Excitatory postsynaptic potential, Interneuron and Dystonia with her study of Cholinergic.
Her study on Glutamatergic also encompasses disciplines like
Her primary areas of study are Neuroscience, Cholinergic, Synaptic plasticity, Dystonia and Medium spiny neuron. Her work carried out in the field of Neuroscience brings together such families of science as Long-term potentiation, Glutamatergic and Metabotropic glutamate receptor. Her studies in Cholinergic integrate themes in fields like Agonist, Dopamine receptor D2, Acetylcholine, Depolarization and Interneuron.
The various areas that Paola Bonsi examines in her Synaptic plasticity study include Long-Term Synaptic Depression, Long-term depression and Neurotransmission. Her work deals with themes such as Muscarinic acetylcholine receptor, Mutation, Period, Motor learning and Movement disorders, which intersect with Dystonia. In her research on the topic of Medium spiny neuron, Biophysics is strongly related with Biochemistry.
Neuroscience, Dystonia, Synaptic plasticity, Striatum and Period are her primary areas of study. Her research integrates issues of Globus pallidus and Heterogeneous group in her study of Neuroscience. Her Dystonia research incorporates themes from Receptor, Dopamine receptor D2, Downregulation and upregulation, AMPA receptor and Movement disorders.
Her biological study spans a wide range of topics, including Anesthesia and Cholinergic. Her Synaptic plasticity research is multidisciplinary, incorporating elements of Postsynaptic potential, Neuroligin, Programmed cell death and Long-term depression. The Period study combines topics in areas such as Structural plasticity, Mutation, Long-term potentiation, Neuroplasticity and Mouse Striatum.
Paola Bonsi focuses on Neuroscience, Dystonia, Synaptic plasticity, Disease and Multiple body regions. Her Neuroscience research is multidisciplinary, relying on both Signal transduction and Long-Term Synaptic Depression. Her Signal transduction research is multidisciplinary, relying on both Receptor, Dopamine receptor D2, Dopaminergic and Downregulation and upregulation.
Paola Bonsi interconnects Striatum, Metaplasticity, Neurotransmission, Neuroligin and Glutamatergic in the investigation of issues within Long-Term Synaptic Depression. Her Disease research incorporates elements of Twisting movements, Essential tremor and Involuntary muscle contractions. Her research in Neuroplasticity intersects with topics in AMPA receptor, Long-term potentiation, Mutation and Period.
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Impaired dopamine release and synaptic plasticity in the striatum of PINK1-deficient mice
Tohru Kitada;Antonio Pisani;Douglas R. Porter;Hiroo Yamaguchi.
Proceedings of the National Academy of Sciences of the United States of America (2007)
Metabotropic glutamate receptor 5 mediates the potentiation of N-methyl-D-aspartate responses in medium spiny striatal neurons
A Pisani;P Gubellini;P Bonsi;F Conquet.
Neuroscience (2001)
Toxicity of selected plant volatiles in microbial and mammalian short-term assays
A. Stammati;P. Bonsi;F. Zucco;R. Moezelaar.
Food and Chemical Toxicology (1999)
Impairment of bidirectional synaptic plasticity in the striatum of a mouse model of DYT1 dystonia: role of endogenous acetylcholine
Giuseppina Martella;Annalisa Tassone;Giuseppe Sciamanna;Paola Platania.
Brain (2009)
Activation of D2-like dopamine receptors reduces synaptic inputs to striatal cholinergic interneurons.
Antonio Pisani;Paola Bonsi;Diego Centonze;Paolo Calabresi.
The Journal of Neuroscience (2000)
Inhibition of mitochondrial complex II induces a long-term potentiation of NMDA-mediated synaptic excitation in the striatum requiring endogenous dopamine.
Paolo Calabresi;Paolo Gubellini;Barbara Picconi;Diego Centonze.
The Journal of Neuroscience (2001)
Endogenous Serotonin Excites Striatal Cholinergic Interneurons via the Activation of 5-HT 2C, 5-HT6, and 5-HT7 Serotonin Receptors: Implications for Extrapyramidal Side Effects of Serotonin Reuptake Inhibitors
Paola Bonsi;Dario Cuomo;Jun Ding;Giuseppe Sciamanna.
Neuropsychopharmacology (2007)
Intracellular calcium increase in epileptiform activity: modulation by levetiracetam and lamotrigine.
Antonio Pisani;Paola Bonsi;Giuseppina Martella;Cristiano De Persis.
Epilepsia (2004)
Altered responses to dopaminergic D2 receptor activation and N-type calcium currents in striatal cholinergic interneurons in a mouse model of DYT1 dystonia
A. Pisani;G. Martella;A. Tscherter;P. Bonsi.
Neurobiology of Disease (2006)
Striatal cholinergic dysfunction as a unifying theme in the pathophysiology of dystonia
K.L. Eskow Jaunarajs;P. Bonsi;M.F. Chesselet;D.G. Standaert.
Progress in Neurobiology (2015)
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