His primary scientific interests are in Neuroscience, Synaptic plasticity, Long-term potentiation, Long-term depression and Basal ganglia. Neuroscience is often connected to Parkinson's disease in his work. His Synaptic plasticity study also includes fields such as
Alessandro Tozzi studies Synaptic fatigue which is a part of Long-term potentiation. His study in Long-term depression is interdisciplinary in nature, drawing from both Metaplasticity and Neurotransmitter. The various areas that Alessandro Tozzi examines in his Basal ganglia study include Neuroplasticity and Premovement neuronal activity.
The scientist’s investigation covers issues in Neuroscience, Long-term potentiation, Synaptic plasticity, Neurotransmission and Internal medicine. His Neuroscience research incorporates elements of Glutamate receptor and Parkinson's disease. As part of one scientific family, Alessandro Tozzi deals mainly with the area of Long-term potentiation, narrowing it down to issues related to the Experimental autoimmune encephalomyelitis, and often Neuroinflammation, Microglia and Neurodegeneration.
His work is dedicated to discovering how Synaptic plasticity, Long-term depression are connected with Metaplasticity, Neuron and Neurotransmitter and other disciplines. His Neurotransmission research includes elements of Postsynaptic potential and Excitatory postsynaptic potential. His work in the fields of Dopamine, such as Dopaminergic and Medium spiny neuron, overlaps with other areas such as Membrane hyperpolarization.
Alessandro Tozzi spends much of his time researching Neurotransmission, Neuroscience, Long-term potentiation, Synaptic plasticity and Parkinson's disease. His work in Neurotransmission addresses issues such as NMDA receptor, which are connected to fields such as Kinase activity, Glutamatergic and Endocrinology. His research in Neuroscience intersects with topics in Autism, Knockout mouse and Intracellular.
His Long-term potentiation research is multidisciplinary, relying on both Glutamate receptor, Central nervous system, Biological neural network, Hippocampus and Pharmacology. The study incorporates disciplines such as Hippocampal formation, Protein kinase B and Electrophysiology in addition to Synaptic plasticity. His research integrates issues of Dopaminergic, Dopamine and Neuroprotection in his study of Parkinson's disease.
His scientific interests lie mostly in Neurotransmission, Cell biology, Dopamine, Parkinson's disease and Quinpirole. His Neurotransmission research integrates issues from NMDA receptor, Electrophysiology and Toxicity. His Cell biology study frequently draws connections between adjacent fields such as Postsynaptic potential.
His work deals with themes such as Glutamate receptor, Neuroprotection, Alpha-synuclein and LRRK2, which intersect with Dopamine. The various areas that Alessandro Tozzi examines in his Parkinson's disease study include Long-term potentiation, Medium spiny neuron and Neuroscience. His work deals with themes such as AMPA receptor and Kinase activity, which intersect with Quinpirole.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Dopamine-mediated regulation of corticostriatal synaptic plasticity
Paolo Calabresi;Barbara Picconi;Alessandro Tozzi;Massimiliano Di Filippo.
Trends in Neurosciences (2007)
Direct and indirect pathways of basal ganglia: a critical reappraisal
Paolo Calabresi;Barbara Picconi;Alessandro Tozzi;Veronica Ghiglieri.
Nature Neuroscience (2014)
Effects of central and peripheral inflammation on hippocampal synaptic plasticity
Massimiliano Di Filippo;Davide Chiasserini;Fabrizio Gardoni;Barbara Viviani.
Neurobiology of Disease (2013)
Plasticity and repair in the post-ischemic brain
Massimiliano Di Filippo;Alessandro Tozzi;Cinzia Costa;Vincenzo Belcastro.
Neuropharmacology (2008)
Mitochondria and the link between neuroinflammation and neurodegeneration
Massimiliano Di Filippo;Davide Chiasserini;Alessandro Tozzi;Barbara Picconi.
Journal of Alzheimer's Disease (2010)
Decreased NR2B subunit synaptic levels cause impaired long-term potentiation but not long-term depression
F Gardoni;D Mauceri;M Malinverno;F Polli.
The Journal of Neuroscience (2009)
The distinct role of medium spiny neurons and cholinergic interneurons in the D₂/A₂A receptor interaction in the striatum: implications for Parkinson's disease.
Alessandro Tozzi;Antonio de Iure;Massimiliano Di Filippo;Michela Tantucci.
The Journal of Neuroscience (2011)
Involvement of transient receptor potential-like channels in responses to mGluR-I activation in midbrain dopamine neurons.
Alessandro Tozzi;C. Peter Bengtson;Patrizia Longone;Corrado Carignani.
European Journal of Neuroscience (2003)
Short-term and long-term plasticity at corticostriatal synapses: Implications for learning and memory
Massimiliano Di Filippo;Barbara Picconi;Michela Tantucci;Veronica Ghiglieri.
Behavioural Brain Research (2009)
Inhibition of phosphodiesterases rescues striatal long-term depression and reduces levodopa-induced dyskinesia.
Barbara Picconi;Vincenza Bagetta;Veronica Ghiglieri;Vincent Paillè.
Brain (2011)
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