D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 48 Citations 6,000 157 World Ranking 3643 National Ranking 173

Overview

What is he best known for?

The fields of study he is best known for:

  • Enzyme
  • Internal medicine
  • Neurotransmitter

Robert Nisticò spends much of his time researching Neuroscience, Long-term potentiation, Hippocampus, Synaptic plasticity and Hippocampal formation. His Neuroscience study incorporates themes from Translational research and Disease. The Long-term potentiation study combines topics in areas such as NMDA receptor, Glutamate receptor, Glutamatergic, Neurotransmission and Excitatory postsynaptic potential.

In Neurotransmission, Robert Nisticò works on issues like Kainate receptor, which are connected to Neural facilitation, Mossy fiber, Biophysics and Antagonist. Robert Nisticò does research in Synaptic plasticity, focusing on LTP induction specifically. His Hippocampal formation research incorporates elements of AMPA receptor and Knockout mouse.

His most cited work include:

  • A Role for Ca2+ Stores in Kainate Receptor-Dependent Synaptic Facilitation and LTP at Mossy Fiber Synapses in the Hippocampus (162 citations)
  • Inflammation subverts hippocampal synaptic plasticity in experimental multiple sclerosis. (97 citations)
  • Characterisation of UBP296: a novel, potent and selective kainate receptor antagonist. (84 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Neuroscience, Long-term potentiation, Synaptic plasticity, Hippocampus and Hippocampal formation. His Neuroscience research is multidisciplinary, incorporating elements of NMDA receptor, Glutamate receptor, Neurotransmission, Disease and Long-term depression. His Long-term potentiation research includes elements of Dentate gyrus, Neuroplasticity, Dendritic spine and Agonist.

The concepts of his Synaptic plasticity study are interwoven with issues in Synapse, Multiple sclerosis, Dopamine receptor D2 and Long-Term Synaptic Depression. His study looks at the intersection of Hippocampus and topics like Nerve growth factor with Cholinergic. His research in Hippocampal formation intersects with topics in LTP induction, Electrophysiology and Pharmacology, Neuroprotection.

He most often published in these fields:

  • Neuroscience (56.52%)
  • Long-term potentiation (31.06%)
  • Synaptic plasticity (27.95%)

What were the highlights of his more recent work (between 2018-2021)?

  • Neuroscience (56.52%)
  • Disease (11.18%)
  • Synaptic plasticity (27.95%)

In recent papers he was focusing on the following fields of study:

The scientist’s investigation covers issues in Neuroscience, Disease, Synaptic plasticity, Long-term potentiation and Hippocampus. His Neuroscience study combines topics from a wide range of disciplines, such as Precision medicine, Pathological, Glutamate receptor, Neurotransmission and TREM2. In the field of Disease, his study on Apolipoprotein E overlaps with subjects such as Deregulation.

His work carried out in the field of Long-term potentiation brings together such families of science as Experimental autoimmune encephalomyelitis, Inhibitory postsynaptic potential, GABAergic and Neuroplasticity. Robert Nisticò has included themes like Cholinergic, Cholinergic neuron, Nerve growth factor, Transcranial magnetic stimulation and Hippocampal formation in his Hippocampus study. He usually deals with Cholinergic and limits it to topics linked to Neurogenesis and Endocrinology and Internal medicine.

Between 2018 and 2021, his most popular works were:

  • Plasma amyloid β 40/42 ratio predicts cerebral amyloidosis in cognitively normal individuals at risk for Alzheimer's disease. (50 citations)
  • A Path Toward Precision Medicine for Neuroinflammatory Mechanisms in Alzheimer's Disease. (37 citations)
  • The β-Secretase BACE1 in Alzheimer's Disease. (22 citations)

In his most recent research, the most cited papers focused on:

  • Enzyme
  • Internal medicine
  • Neurotransmitter

Robert Nisticò mainly focuses on Neuroscience, Disease, Biomarker, Synaptic plasticity and Precision medicine. As part of his studies on Neuroscience, Robert Nisticò often connects relevant subjects like Long-term potentiation. His Long-term potentiation study combines topics in areas such as Systems pharmacology and Drug.

His research investigates the connection with Disease and areas like Cohort which intersect with concerns in Receiver operating characteristic, Clinical study design, Amyloidosis and Positron emission tomography. His work deals with themes such as Neurotransmission, Hippocampal formation, Alzheimer's disease, Long-term depression and Drug discovery, which intersect with Synaptic plasticity. His study in Precision medicine is interdisciplinary in nature, drawing from both Proinflammatory cytokine, TREM2, Neuroinflammation and Systems biology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

A Role for Ca2+ Stores in Kainate Receptor-Dependent Synaptic Facilitation and LTP at Mossy Fiber Synapses in the Hippocampus

Sari E Lauri;Zuner A Bortolotto;Robert Nistico;David Bleakman.
Neuron (2003)

231 Citations

Stress dynamically regulates behavior and glutamatergic gene expression in hippocampus by opening a window of epigenetic plasticity

Carla Nasca;Danielle Zelli;Benedetta Bigio;Sonia Piccinin.
Proceedings of the National Academy of Sciences of the United States of America (2015)

154 Citations

Inflammation subverts hippocampal synaptic plasticity in experimental multiple sclerosis.

Robert Nisticò;Dalila Mango;Georgia Mandolesi;Sonia Piccinin.
PLOS ONE (2013)

137 Citations

Characterisation of UBP296: a novel, potent and selective kainate receptor antagonist.

Julia C.A. More;Robert Nistico;Nigel P. Dolman;Vernon R.J. Clarke.
Neuropharmacology (2004)

135 Citations

Perspective on future role of biological markers in clinical therapy trials of Alzheimer's disease: A long-range point of view beyond 2020

Harald Hampel;Simone Lista;Stefan J. Teipel;Francesco Garaci.
Biochemical Pharmacology (2014)

122 Citations

L-DOPA: a scapegoat for accelerated neurodegeneration in Parkinson's disease?

Janusz Lipski;Robert Nistico;Nicola Berretta;Ezia Guatteo.
Progress in Neurobiology (2011)

121 Citations

Paraquat- and rotenone-induced models of Parkinson's disease

R. Nisticò;B. Mehdawy;S. Piccirilli;N. Mercuri.
International Journal of Immunopathology and Pharmacology (2011)

110 Citations

The role of oxidative stress in paraquat-induced neurotoxicity in rats: protection by non peptidyl superoxide dismutase mimetic

Vincenzo Mollace;Michelangelo Iannone;Carolina Muscoli;Emesto Palma.
Neuroscience Letters (2003)

107 Citations

Profile of gantenerumab and its potential in the treatment of Alzheimer's disease

Dijana Novakovic;Marco Feligioni;Sergio Scaccianoce;Alessandra Caruso.
Drug Design Development and Therapy (2013)

105 Citations

Targeting Synaptic Dysfunction in Alzheimer’s Disease Therapy

Robert Nisticò;Marco Pignatelli;Sonia Piccinin;Nicola B Mercuri.
Molecular Neurobiology (2012)

95 Citations

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