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Neuroscience

D-Index
38
Citations
5401
World Ranking
8589
National Ranking
68

Overview

Michael Tal is affiliated with the Hebrew University of Jerusalem in Israel. Their primary research contributions fall within the broad field of Medicine, with a focus on subfields including Pulmonary and Respiratory Medicine, Emergency Medical Services, Immunology, Surgery, and Cellular and Molecular Neuroscience.

Their research work frequently addresses topics such as Central Venous Catheters and Hemodialysis, Vascular Procedures and Complications, Phagocytosis and Immune Regulation, Nerve Injury and Regeneration, Neuroinflammation and Neurodegeneration Mechanisms, Venous Thromboembolism Diagnosis and Management, and Pain Mechanisms and Treatments.

Michael Tal has published a series of papers, notable among which are:

  • "Deletion of CD47 from Schwann cells and macrophages hastens myelin disruption/dismantling and scavenging in Schwann cells and augments myelin debris phagocytosis in macrophages," 2023, Journal of Neuroinflammation
  • "Reduced nontarget embolization and increased targeted delivery with a reflux-control microcatheter in a swine model," 2021, Diagnostic and Interventional Imaging
  • "Catheter-related blood stream infection in hemodialysis patients with symmetric tunneled non-side-hole hemodialysis catheters," 2021, The Journal of Vascular Access
  • "Clot accumulation at the tip of hemodialysis catheters in a large animal model," 2020, The Journal of Vascular Access
  • "Staphylococcus aureus accumulation at the tip of hemodialysis catheters with or without tip side holes in catheter related bloodstream infection in a large animal," 2021, The Journal of Vascular Access

Their research has appeared predominantly in the following venues:

  • The Journal of Vascular Access
  • Journal of Neuroinflammation
  • Diagnostic and Interventional Imaging
  • ASAIO Journal
  • BMJ Sexual & Reproductive Health

Frequent collaborators include Alexander S. Yevzlin, Miri Gitik, Gerard Elberg, Fanny Reichert, and Shlomo Rotshenker. These co-authors have contributed multiple works alongside Tal, indicating ongoing collaborative research efforts.

Best Publications

  • Extra-territorial pain in rats with a peripheral mononeuropathy: mechano-hyperalgesia and mechano-allodynia in the territory of an uninjured nerve.

    Michael Tal;Gary J. Bennett

  • Tyrosine Phosphorylation of the NR2B Subunit of the NMDA Receptor in the Spinal Cord during the Development and Maintenance of Inflammatory Hyperalgesia

    Wei Guo;Shiping Zou;Yun Guan;Tetsuya Ikeda

  • Genetic impairment of interleukin-1 signaling attenuates neuropathic pain, autotomy, and spontaneous ectopic neuronal activity, following nerve injury in mice.

    Gilly Wolf;Eran Gabay;Michael Tal;Raz Yirmiya

  • Sporadic Amplification of the HER2/neu Protooncogene in Adenocarcinomas of Various Tissues

    M Tal;M Wetzler;Z Josefberg;A Deutch

  • A novel antioxidant alleviates heat hyperalgesia in rats with an experimental painful peripheral neuropathy.

    Michael Tal

  • Susceptibility to chronic pain following nerve injury is genetically affected by CACNG2

    Jonathan Nissenbaum;Marshall Devor;Ze'ev Seltzer;Mathias Gebauer

  • Abnormal discharge originates at the site of nerve injury in experimental constriction neuropathy (CCI) in the rat

    Michael Tal;Eli Eliav

  • Dextrorphan relieves neuropathic heat-evoked hyperalgesia in the rat

    Michael Tal;Gary J. Bennett

  • The effects of axotomy on neurons and satellite glial cells in mouse trigeminal ganglion

    Pavel S Cherkas;Tian-Ying Huang;Thomas Pannicke;Michael Tal

  • Inflammation with no axonal damage of the rat saphenous nerve trunk induces ectopic discharge and mechanosensitivity in myelinated axons.

    Eli Eliav;Rafael Benoliel;Michael Tal

  • The pancreatic β-cell glucose sensor

    Shimon Efrat;Michael Tal;Harvey F. Lodish

  • A novel putative neuropeptide receptor expressed in neural tissue, including sensory epithelia.

    Michael Tal;David A. Ammar;Marcela Karpuj;Valery Krizhanovsky

  • Human HER2 (neu) promoter: evidence for multiple mechanisms for transcriptional initiation.

    M Tal;C R King;M H Kraus;A Ullrich

  • Streptococcus pneumoniae Cell Wall-Localized Trigger Factor Elicits a Protective Immune Response and Contributes to Bacterial Adhesion to the Host.

    Aviad Cohen;Shani Troib;Shahar Dotan;Hastyar Najmuldeen;Hastyar Najmuldeen

  • P2 receptors in satellite glial cells in trigeminal ganglia of mice

    M Weick;P.S Cherkas;W Härtig;T Pannicke

  • Ectopic discharge in injured nerves: comparison of trigeminal and somatic afferent

    Michael Tal;Marshall Devor

  • The effects of brain stem transections on the neuronal networks responsible for rhythmical jaw muscle activity in the guinea pig

    SH Chandler;M Tal

  • Neuropathic pain sensations are differentially sensitive to dextrorphan.

    Tal M;Bennett Gj

  • The role of IL-6 and IL-1beta in painful perineural inflammatory neuritis.

    Eli Eliav;Rafael Benoliel;Uri Herzberg;Mythili Kalladka

  • Application of a pro-inflammatory agent to the orbital portion of the rat infraorbital nerve induces changes indicative of ongoing trigeminal pain

    Rafael Benoliel;Asaf Wilensky;Michael Tal;Eli Eliav

  • The organization and activity patterns of the anterior and posterior heads of the guinea pig digastric muscle

    A. Lev-Tov;M. Tal

Frequent Co-Authors

Marshall Devor
Marshall Devor Hebrew University of Jerusalem
Raz Yirmiya
Raz Yirmiya Hebrew University of Jerusalem
Gary J. Bennett
Gary J. Bennett McGill University
Menachem Hanani
Menachem Hanani Hebrew University of Jerusalem
Donald A. Morrison
Donald A. Morrison University of Illinois at Chicago
Yun Guan
Yun Guan Johns Hopkins University School of Medicine
Thomas Pannicke
Thomas Pannicke Leipzig University
Ze'ev Seltzer
Ze'ev Seltzer University of Toronto
Timothy J. Mitchell
Timothy J. Mitchell University of Birmingham
Yosef Shiloh
Yosef Shiloh Tel Aviv University

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Combining neuroscience with these related fields, through flexible and affordable online study, can significantly expand your career options and enhance your impact in both clinical and research settings.

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