What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Internal medicine
His primary areas of study are Lipofuscin, Cell biology, Retina, Retinal and Retinal pigment epithelium.
His Lipofuscin study deals with the bigger picture of Biochemistry.
Michael E. Boulton has researched Cell biology in several fields, including Oxidative stress, Retinal ganglion and Genetics.
His studies deal with areas such as Diabetic retinopathy, Retinopathy, Vascular endothelial growth factor, Pathology and mitochondrial fusion as well as Retina.
Michael E. Boulton has researched Retinal in several fields, including Endocrinology, Oxidation reduction, Antioxidant, Protein oxidation and Internal medicine.
His research in Retinal pigment epithelium intersects with topics in Melanin, Biophysics, Mitochondrial DNA and Macular degeneration.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- The Role of Oxidative Stress in the Pathogenesis of Age-Related Macular Degeneration (1564 citations)
- Oxidative damage and age-related macular degeneration. (660 citations)
What are the main themes of his work throughout his whole career to date?
Cell biology, Retinal, Retinal pigment epithelium, Retina and Internal medicine are his primary areas of study.
His Cell biology study combines topics from a wide range of disciplines, such as Autophagy, Oxidative stress, Angiogenesis and Cell growth.
His Retinal study combines topics in areas such as Endothelial stem cell, In vitro, Cell culture, Neovascularization and Pathology.
Michael E. Boulton interconnects Retinal degeneration, Lipofuscin, Molecular biology and Macular degeneration in the investigation of issues within Retinal pigment epithelium.
The study incorporates disciplines such as Ophthalmology and Anatomy in addition to Retina.
His Internal medicine research is multidisciplinary, incorporating elements of Diabetes mellitus and Endocrinology.
He most often published in these fields:
- Cell biology (35.38%)
- Retinal (26.48%)
- Retinal pigment epithelium (20.34%)
What were the highlights of his more recent work (between 2012-2021)?
- Internal medicine (18.22%)
- Endocrinology (17.80%)
- Cell biology (35.38%)
In recent papers he was focusing on the following fields of study:
His primary scientific interests are in Internal medicine, Endocrinology, Cell biology, Diabetic retinopathy and Retinal pigment epithelium.
His Internal medicine research is multidisciplinary, relying on both Diabetes mellitus and Downregulation and upregulation.
His work carried out in the field of Endocrinology brings together such families of science as CD34, Retina and Retinal.
The various areas that Michael E. Boulton examines in his Cell biology study include Autophagy, Lipofuscin and microRNA.
As a part of the same scientific family, he mostly works in the field of Autophagy, focusing on Oxidative stress and, on occasion, Reactive oxygen species.
His biological study spans a wide range of topics, including Retinal degeneration, Anatomy and Pathology.
Between 2012 and 2021, his most popular works were:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Autophagy and heterophagy dysregulation leads to retinal pigment epithelium dysfunction and development of age-related macular degeneration (204 citations)
- Dysregulated autophagy in the RPE is associated with increased susceptibility to oxidative stress and AMD. (198 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Internal medicine
Michael E. Boulton mostly deals with Endocrinology, Internal medicine, Cell biology, Retinal pigment epithelium and Autophagy.
His Endocrinology research incorporates themes from Retinal and Bone marrow.
Michael E. Boulton does research in Cell biology, focusing on Lipid raft specifically.
His work deals with themes such as Lipofuscin, Pathology, Signal transduction and Secretion, which intersect with Retinal pigment epithelium.
His Autophagy research integrates issues from Oxidative stress, Computational biology and Programmed cell death.
His Autophagosome research is multidisciplinary, incorporating perspectives in MAP1LC3B, Oxidative phosphorylation, Physiology and Sequestosome 1.
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