Masaki Fukata focuses on Cell biology, IQGAP1, RAC1, GTPase and AMPA receptor. His work deals with themes such as Biochemistry and Cell polarity, which intersect with Cell biology. His IQGAP1 research is multidisciplinary, incorporating elements of Cell morphology and GTP-binding protein regulators.
His research integrates issues of Cadherin and Cell adhesion in his study of RAC1. His GTPase study combines topics from a wide range of disciplines, such as Guanosine, Molecular biology and G protein. His AMPA receptor study incorporates themes from Synaptic plasticity and Epilepsy.
The scientist’s investigation covers issues in Cell biology, Neuroscience, Biochemistry, IQGAP1 and RAC1. The concepts of his Cell biology study are interwoven with issues in Cell adhesion and Lipid modification. His Neuroscience research incorporates themes from Synaptic plasticity, AMPA receptor, Postsynaptic potential and Neurotransmission.
As a member of one scientific family, Masaki Fukata mostly works in the field of IQGAP1, focusing on Membrane ruffling and, on occasion, Filamentous actin. His RAC1 study combines topics in areas such as CDC42 and GTPase. His GTPase study integrates concerns from other disciplines, such as Guanosine and Molecular biology.
His main research concerns Cell biology, Neuroscience, Monoclonal, Autoantibody and Immunology. Masaki Fukata has researched Cell biology in several fields, including Lipid microdomain and Cell–cell interaction. All of his Neuroscience and Macaque, Premovement neuronal activity, Motor cortex, Stimulation and Primary motor cortex investigations are sub-components of the entire Neuroscience study.
His work deals with themes such as Epitope, Isotype and Affinity maturation, which intersect with Monoclonal. Masaki Fukata works mostly in the field of Isotype, limiting it down to topics relating to Limbic encephalitis and, in certain cases, Prednisolone, Status epilepticus, Pathology and Necrosis. Masaki Fukata has included themes like Hippocampal formation, Hippocampus and Epilepsy in his Prednisolone study.
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Rac1 and Cdc42 Capture Microtubules through IQGAP1 and CLIP-170
Masaki Fukata;Takashi Watanabe;Jun Noritake;Masato Nakagawa.
Role of IQGAP1, a Target of the Small GTPases Cdc42 and Rac1, in Regulation of E-Cadherin- Mediated Cell-Cell Adhesion
Shinya Kuroda;Masaki Fukata;Masato Nakagawa;Katsuhiko Fujii.
Roles of Rho-family GTPases in cell polarisation and directional migration.
Masaki Fukata;Masato Nakagawa;Kozo Kaibuchi.
Current Opinion in Cell Biology (2003)
Interaction with IQGAP1 Links APC to Rac1, Cdc42, and Actin Filaments during Cell Polarization and Migration
Takashi Watanabe;Shujie Wang;Jun Noritake;Kazumasa Sato.
Developmental Cell (2004)
Rho-family GTPases in cadherin-mediated cell — cell adhesion
Masaki Fukata;Kozo Kaibuchi.
Nature Reviews Molecular Cell Biology (2001)
Identification of PSD-95 palmitoylating enzymes.
Masaki Fukata;Yuko Fukata;Hillel Adesnik;Roger A. Nicoll.
Protein palmitoylation in neuronal development and synaptic plasticity
Yuko Fukata;Yuko Fukata;Masaki Fukata.
Nature Reviews Neuroscience (2010)
Identification of IQGAP as a Putative Target for the Small GTPases, Cdc42 and Rac1
Shinya Kuroda;Masaki Fukata;Kenta Kobayashi;Masato Nakafuku.
Journal of Biological Chemistry (1996)
Epilepsy-Related Ligand/Receptor Complex LGI1 and ADAM22 Regulate Synaptic Transmission
Yuko Fukata;Hillel Adesnik;Tsuyoshi Iwanaga;David S. Bredt.
Recruitment and activation of Rac1 by the formation of E-cadherin-mediated cell-cell adhesion sites
Masato Nakagawa;Masaki Fukata;Masaki Yamaga;Naohiro Itoh.
Journal of Cell Science (2001)
Profile was last updated on December 6th, 2021.
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