H-Index & Metrics Top Publications

H-Index & Metrics

Discipline name H-index Citations Publications World Ranking National Ranking
Biology and Biochemistry H-index 62 Citations 13,517 109 World Ranking 4845 National Ranking 330

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Cell membrane

Masaki Fukata focuses on Cell biology, IQGAP1, RAC1, GTPase and AMPA receptor. His work deals with themes such as Biochemistry and Cell polarity, which intersect with Cell biology. His IQGAP1 research is multidisciplinary, incorporating elements of Cell morphology and GTP-binding protein regulators.

His research integrates issues of Cadherin and Cell adhesion in his study of RAC1. His GTPase study combines topics from a wide range of disciplines, such as Guanosine, Molecular biology and G protein. His AMPA receptor study incorporates themes from Synaptic plasticity and Epilepsy.

His most cited work include:

  • Rac1 and Cdc42 Capture Microtubules through IQGAP1 and CLIP-170 (516 citations)
  • Role of IQGAP1, a Target of the Small GTPases Cdc42 and Rac1, in Regulation of E-Cadherin- Mediated Cell-Cell Adhesion (441 citations)
  • Roles of Rho-family GTPases in cell polarisation and directional migration. (423 citations)

What are the main themes of his work throughout his whole career to date?

The scientist’s investigation covers issues in Cell biology, Neuroscience, Biochemistry, IQGAP1 and RAC1. The concepts of his Cell biology study are interwoven with issues in Cell adhesion and Lipid modification. His Neuroscience research incorporates themes from Synaptic plasticity, AMPA receptor, Postsynaptic potential and Neurotransmission.

As a member of one scientific family, Masaki Fukata mostly works in the field of IQGAP1, focusing on Membrane ruffling and, on occasion, Filamentous actin. His RAC1 study combines topics in areas such as CDC42 and GTPase. His GTPase study integrates concerns from other disciplines, such as Guanosine and Molecular biology.

He most often published in these fields:

  • Cell biology (85.07%)
  • Neuroscience (29.10%)
  • Biochemistry (28.36%)

What were the highlights of his more recent work (between 2018-2021)?

  • Cell biology (85.07%)
  • Neuroscience (29.10%)
  • Monoclonal (3.73%)

In recent papers he was focusing on the following fields of study:

His main research concerns Cell biology, Neuroscience, Monoclonal, Autoantibody and Immunology. Masaki Fukata has researched Cell biology in several fields, including Lipid microdomain and Cell–cell interaction. All of his Neuroscience and Macaque, Premovement neuronal activity, Motor cortex, Stimulation and Primary motor cortex investigations are sub-components of the entire Neuroscience study.

His work deals with themes such as Epitope, Isotype and Affinity maturation, which intersect with Monoclonal. Masaki Fukata works mostly in the field of Isotype, limiting it down to topics relating to Limbic encephalitis and, in certain cases, Prednisolone, Status epilepticus, Pathology and Necrosis. Masaki Fukata has included themes like Hippocampal formation, Hippocampus and Epilepsy in his Prednisolone study.

Between 2018 and 2021, his most popular works were:

  • Human Cerebrospinal Fluid Monoclonal LGI1 Autoantibodies Increase Neuronal Excitability (18 citations)
  • Human Cerebrospinal Fluid Monoclonal LGI1 Autoantibodies Increase Neuronal Excitability (18 citations)
  • ABHD10 is an S-depalmitoylase affecting redox homeostasis through peroxiredoxin-5. (17 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Top Publications

Rac1 and Cdc42 Capture Microtubules through IQGAP1 and CLIP-170

Masaki Fukata;Takashi Watanabe;Jun Noritake;Masato Nakagawa.
Cell (2002)

733 Citations

Role of IQGAP1, a Target of the Small GTPases Cdc42 and Rac1, in Regulation of E-Cadherin- Mediated Cell-Cell Adhesion

Shinya Kuroda;Masaki Fukata;Masato Nakagawa;Katsuhiko Fujii.
Science (1998)

623 Citations

Roles of Rho-family GTPases in cell polarisation and directional migration.

Masaki Fukata;Masato Nakagawa;Kozo Kaibuchi.
Current Opinion in Cell Biology (2003)

581 Citations

Interaction with IQGAP1 Links APC to Rac1, Cdc42, and Actin Filaments during Cell Polarization and Migration

Takashi Watanabe;Shujie Wang;Jun Noritake;Kazumasa Sato.
Developmental Cell (2004)

524 Citations

Rho-family GTPases in cadherin-mediated cell — cell adhesion

Masaki Fukata;Kozo Kaibuchi.
Nature Reviews Molecular Cell Biology (2001)

511 Citations

Identification of PSD-95 palmitoylating enzymes.

Masaki Fukata;Yuko Fukata;Hillel Adesnik;Roger A. Nicoll.
Neuron (2004)

501 Citations

Protein palmitoylation in neuronal development and synaptic plasticity

Yuko Fukata;Yuko Fukata;Masaki Fukata.
Nature Reviews Neuroscience (2010)

475 Citations

Identification of IQGAP as a Putative Target for the Small GTPases, Cdc42 and Rac1

Shinya Kuroda;Masaki Fukata;Kenta Kobayashi;Masato Nakafuku.
Journal of Biological Chemistry (1996)

389 Citations

Epilepsy-Related Ligand/Receptor Complex LGI1 and ADAM22 Regulate Synaptic Transmission

Yuko Fukata;Hillel Adesnik;Tsuyoshi Iwanaga;David S. Bredt.
Science (2006)

351 Citations

Recruitment and activation of Rac1 by the formation of E-cadherin-mediated cell-cell adhesion sites

Masato Nakagawa;Masaki Fukata;Masaki Yamaga;Naohiro Itoh.
Journal of Cell Science (2001)

336 Citations

Profile was last updated on December 6th, 2021.
Research.com Ranking is based on data retrieved from the Microsoft Academic Graph (MAG).
The ranking h-index is inferred from publications deemed to belong to the considered discipline.

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