Mikio Hoshino focuses on Cell biology, Neuroscience, Cytoskeleton, RAC1 and CDC42. His Cell biology research is multidisciplinary, relying on both Cerebral cortex and Cofilin. His work on Cerebellum, Neuron and Neurogenesis as part of his general Neuroscience study is frequently connected to Structural organization, thereby bridging the divide between different branches of science.
His studies in Cerebellum integrate themes in fields like Calbindin, Rhombic lip, Cerebrum and GABAergic. His studies deal with areas such as Guanosine and Guanine nucleotide exchange factor, GTPase as well as Cytoskeleton. His research integrates issues of Psychological repression, Mitogen-activated protein kinase kinase, Tubulin, Microtubule and Phosphorylation in his study of RAC1.
His primary areas of study are Cell biology, Neuroscience, Cerebellum, Photochemistry and Transcription factor. His work on Cell biology is being expanded to include thematically relevant topics such as Cerebral cortex. His work on GABAergic, Inhibitory postsynaptic potential and Neuron as part of general Neuroscience study is frequently connected to Glutamatergic, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
His biological study spans a wide range of topics, including Progenitor cell, Rhombic lip, Granule cell, Anatomy and Cell type. His work in Photochemistry addresses issues such as Luminescence, which are connected to fields such as Copper. His research investigates the connection between RAC1 and topics such as CDC42 that intersect with issues in RHOA, Lamellipodium and Cytoskeleton.
Mikio Hoshino focuses on Cell biology, Neuroscience, Transcription factor, Granule cell and Cerebellum. His Cell biology research integrates issues from Cadherin and Neural development. Mikio Hoshino has included themes like Functional analysis and Autism in his Neuroscience study.
His Transcription factor study incorporates themes from Chromatin, Cell cycle and Malignant transformation. His Granule cell study integrates concerns from other disciplines, such as Progenitor cell and Phosphorylation. In his study, Neocortex, JAG1, Notch signaling pathway and NEUROD1 is strongly linked to Cell type, which falls under the umbrella field of Cerebellum.
His primary areas of investigation include Transcription factor, Cell biology, Neuroscience, Excitatory postsynaptic potential and Inhibitory postsynaptic potential. His work carried out in the field of Transcription factor brings together such families of science as Protein kinase R, Endoplasmic reticulum and Downregulation and upregulation. His primary area of study in Cell biology is in the field of Protein kinase A.
His Hippocampus, Behavioral neuroscience and Synapse study in the realm of Neuroscience connects with subjects such as AMPA receptor. His studies deal with areas such as Autism and Electrophysiology as well as Excitatory postsynaptic potential.
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Specific induction of neuronal cells from bone marrow stromal cells and application for autologous transplantation
Mari Dezawa;Hiroshi Kanno;Mikio Hoshino;Hirotomi Cho.
Journal of Clinical Investigation (2004)
Bone marrow stromal cells generate muscle cells and repair muscle degeneration.
Mari Dezawa;Hiroto Ishikawa;Yutaka Itokazu;Tomoyuki Yoshihara.
Highly Efficient Green Organic Light-Emitting Diodes Containing Luminescent Three-Coordinate Copper(I) Complexes
Masashi Hashimoto;Satoshi Igawa;Masataka Yashima;Isao Kawata.
Journal of the American Chemical Society (2011)
Ptf1a, a bHLH transcriptional gene, defines GABAergic neuronal fates in cerebellum.
Mikio Hoshino;Shoko Nakamura;Kiyoshi Mori;Kiyoshi Mori;Takeshi Kawauchi.
PAR-6–PAR-3 mediates Cdc42-induced Rac activation through the Rac GEFs STEF/Tiam1
Takashi Nishimura;Tomoya Yamaguchi;Katsuhiro Kato;Masato Yoshizawa.
Nature Cell Biology (2005)
The in vivo roles of STEF/Tiam1, Rac1 and JNK in cortical neuronal migration
Takeshi Kawauchi;Kaori Chihama;Kaori Chihama;Yo-ichi Nabeshima;Mikio Hoshino;Mikio Hoshino.
The EMBO Journal (2003)
Rab GTPases-Dependent Endocytic Pathways Regulate Neuronal Migration and Maturation through N-Cadherin Trafficking
Takeshi Kawauchi;Takeshi Kawauchi;Takeshi Kawauchi;Katsutoshi Sekine;Mima Shikanai;Kaori Chihama.
Cdk5 phosphorylates and stabilizes p27kip1 contributing to actin organization and cortical neuronal migration.
Takeshi Kawauchi;Kaori Chihama;Yo-ichi Nabeshima;Mikio Hoshino;Mikio Hoshino.
Nature Cell Biology (2006)
Induction of DNA damage by dimethylarsine, a metabolite of inorganic arsenics, is for the major part likely due to its peroxyl radical
Kenzo Yamanaka;Mikio Hoshino;Mayumi Okamoto;Ryoji Sawamura.
Biochemical and Biophysical Research Communications (1990)
Consensus Paper: Cerebellar Development.
Ketty Leto;Marife Arancillo;Esther B. E. Becker;Annalisa Buffo.
The Cerebellum (2016)
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