Her scientific interests lie mostly in Biochemistry, Cell biology, Monocyte, Superoxide and Inflammation. Her research in Cell biology intersects with topics in Janus kinase 3 and ZAP70. Her Macrophage research extends to the thematically linked field of Monocyte.
She interconnects Ldl metabolism, Peroxidase, Nitric oxide metabolism, Nitric oxide and Monocytes macrophages in the investigation of issues within Superoxide. In her study, Glutathione is inextricably linked to Lipoprotein oxidation, which falls within the broad field of Inflammation. Her NADPH oxidase study deals with Phosphorylation intersecting with Molecular biology.
Her primary areas of study are Cell biology, Monocyte, Biochemistry, Molecular biology and Signal transduction. Her Cell biology study combines topics in areas such as Inflammation and Chemotaxis. Her Monocyte research incorporates elements of NADPH oxidase, Phospholipase, Phospholipase A2, Zymosan and Protein kinase C.
When carried out as part of a general Biochemistry research project, her work on Superoxide, Low-density lipoprotein, Superoxide dismutase and Lipoxygenase is frequently linked to work in Lipid oxidation, therefore connecting diverse disciplines of study. The various areas that she examines in her Molecular biology study include Interleukin 2, Foam cell and Phosphorylation, Tyrosine phosphorylation. Many of her research projects under Signal transduction are closely connected to Regulation of gene expression with Regulation of gene expression, tying the diverse disciplines of science together.
Martha K. Cathcart focuses on Cell biology, Monocyte, Inflammation, Signal transduction and Chemotaxis. Her Cell biology research incorporates themes from Receptor and Apoptosis. Her studies in Monocyte integrate themes in fields like NADPH oxidase, Foam cell, Biochemistry, Macrophage-1 antigen and Zymosan.
Her study in Foam cell is interdisciplinary in nature, drawing from both Molecular biology and Integrin. Her work investigates the relationship between Inflammation and topics such as Macrophage that intersect with problems in CD18 and Proinflammatory cytokine. She has researched Signal transduction in several fields, including Programmed cell death and Cytokine.
Her primary areas of investigation include Monocyte, Biochemistry, Inflammation, Cell biology and Molecular biology. Martha K. Cathcart interconnects Phospholipase A2, Foam cell and Arachidonic acid in the investigation of issues within Monocyte. Her study on Epoxide hydrolase 2, Lipoxygenase, Monocyte chemotaxis and Chemotaxis is often connected to In vivo as part of broader study in Biochemistry.
She studies Cell biology, focusing on Signal transduction in particular. Her study in Signal transduction is interdisciplinary in nature, drawing from both Tyrosine kinase 2 and Innate immune system. Her research in Molecular biology intersects with topics in Zymosan, NADPH oxidase and Superoxide.
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Monocytes and neutrophils oxidize low density lipoprotein making it cytotoxic.
Martha K. Cathcart;Diane W. Morel;Guy M. Chisolm.
Journal of Leukocyte Biology (1985)
Regulation of superoxide anion production by NADPH oxidase in monocytes/macrophages: contributions to atherosclerosis.
Martha K. Cathcart.
Arteriosclerosis, Thrombosis, and Vascular Biology (2004)
Lipoxygenase contributes to the oxidation of lipids in human atherosclerotic plaques.
V. A. Folcik;R. A. Nivar-Aristy;L. P. Krajewski;Martha K Cathcart.
Journal of Clinical Investigation (1995)
Superoxide anion participation in human monocyte-mediated oxidation of low-density lipoprotein and conversion of low-density lipoprotein to a cytotoxin.
Martha K Cathcart;A. K. McNally;D. W. Morel;G. M. Chisolm.
Journal of Immunology (1989)
CCR1+/CCR5+ Mononuclear Phagocytes Accumulate in the Central Nervous System of Patients with Multiple Sclerosis
Corinna Trebst;Torben Lykke Sørensen;Pia Kivisäkk;Martha K. Cathcart.
American Journal of Pathology (2001)
The oxidation of lipoproteins by monocytes-macrophages. Biochemical and biological mechanisms
Guy M. Chisolm;Stanley L. Hazen;Paul L. Fox;Martha K. Cathcart.
Journal of Biological Chemistry (1999)
Protein Kinase Cδ Is Required for p47phox Phosphorylation and Translocation in Activated Human Monocytes
Erik A. Bey;Bo Xu;Ashish Bhattacharjee;Claudine M. Oldfield.
Journal of Immunology (2004)
IL-4 and IL-13 employ discrete signaling pathways for target gene expression in alternatively activated monocytes/macrophages.
Ashish Bhattacharjee;Meenakshi Shukla;Meenakshi Shukla;Valentin P. Yakubenko;Valentin P. Yakubenko;Anny Mulya;Anny Mulya.
Free Radical Biology and Medicine (2013)
Activated human monocytes oxidize low-density lipoprotein by a lipoxygenase-dependent pathway.
Amy K. Mcnally;Guy M. Chisolm;Diane W. Morel;Martha K. Cathcart.
Journal of Immunology (1990)
Lipoxygenase-mediated transformation of human low density lipoprotein to an oxidized and cytotoxic complex.
M K Cathcart;A K McNally;G M Chisolm.
Journal of Lipid Research (1991)
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