Mario Ascoli spends much of his time researching Receptor, Endocrinology, Gonadotropin, Leydig cell and Cell culture. His Receptor research includes themes of Hormone, Luteinizing hormone, Transfection and Tumor cells. Mario Ascoli combines subjects such as Ovulation and Pregnancy with his study of Luteinizing hormone.
His Endocrinology research incorporates elements of Epidermal growth factor and Inositol phosphate. His work on luteinizing hormone/choriogonadotropin receptor as part of general Gonadotropin research is often related to Touch sensation, thus linking different fields of science. His study looks at the relationship between Leydig cell and topics such as Protein kinase A, which overlap with Forskolin and Second messenger system.
His primary scientific interests are in Endocrinology, Receptor, Cell biology, Leydig cell and Gonadotropin. His Endocrinology research incorporates themes from Cell culture and Epidermal growth factor. His work carried out in the field of Receptor brings together such families of science as Molecular biology, Transfection and Phosphorylation.
Mario Ascoli studied Cell biology and Mutant that intersect with G protein-coupled receptor. His work in Leydig cell addresses issues such as Protein kinase A, which are connected to fields such as Protein kinase C. His biological study spans a wide range of topics, including Complementary DNA, Messenger RNA and Leydig Cell Tumor.
Mario Ascoli focuses on Endocrinology, luteinizing hormone/choriogonadotropin receptor, Receptor, Phosphorylation and Cell biology. While the research belongs to areas of Endocrinology, Mario Ascoli spends his time largely on the problem of Gq alpha subunit, intersecting his research to questions surrounding Phospholipase C and Corpus luteum. The study incorporates disciplines such as Epidermal growth factor, Signal transduction, Inositol phosphate and Granulosa cell in addition to luteinizing hormone/choriogonadotropin receptor.
Mario Ascoli has included themes like Mutation and Mutant in his Receptor study. Mario Ascoli has researched Phosphorylation in several fields, including Molecular biology, Downregulation and upregulation and Epidermal growth factor receptor. His Cell biology study which covers Transfection that intersects with Growth factor, Fusion protein, Ovarian follicle, Transport protein and Cell.
The scientist’s investigation covers issues in Endocrinology, luteinizing hormone/choriogonadotropin receptor, Luteinizing hormone, Signal transduction and Gonadotropin. Mario Ascoli studies Endocrinology, focusing on Leydig cell in particular. His research ties Receptor and luteinizing hormone/choriogonadotropin receptor together.
His research integrates issues of Steroid biosynthesis, Kinase and Gonadotropic cell in his study of Receptor. His Luteinizing hormone research integrates issues from Ovulation, Protein kinase B, Cell culture and Follicular phase. His work in Signal transduction addresses subjects such as Inositol phosphate, which are connected to disciplines such as Follicle-stimulating hormone receptor and Multiplicity of infection.
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The Lutropin/Choriogonadotropin Receptor, A 2002 Perspective
Mario Ascoli;Francesca Fanelli;Deborah L. Segaloff.
Endocrine Reviews (2002)
Characterization of Several Clonal Lines of Cultured Ley dig Tumor Cells: Gonadotropin Receptors and Steroidogenic Responses*
The Lutropin/Choriogonadotropin Receptor… 4 Years Later*
Deborah L. Segaloff;Mario Ascoli.
Endocrine Reviews (1993)
Degradation of receptor-bound human choriogonadotropin by murine Leydig tumor cells.
M Ascoli;D Puett.
Journal of Biological Chemistry (1978)
Gonadotropin binding and stimulation of steroidogenesis in Leydig tumor cells
Mario Ascoli;David Puett.
Proceedings of the National Academy of Sciences of the United States of America (1978)
Internalization and degradation of receptor-bound human choriogonadotropin in Leydig tumor cells. Fate of the hormone subunits.
Journal of Biological Chemistry (1982)
Regulation of gonadotropin receptors and gonadotropin responses in a clonal strain of Leydig tumor cells by epidermal growth factor.
Journal of Biological Chemistry (1981)
The Lutropin/Choriogonadotropin Receptor-Induced Phosphorylation of the Extracellular Signal-Regulated Kinases in Leydig Cells Is Mediated by a Protein Kinase A-Dependent Activation of Ras
Takashi Hirakawa;Mario Ascoli.
Molecular Endocrinology (2003)
GIPC binds to the human lutropin receptor (hLHR) through an unusual PDZ domain binding motif, and it regulates the sorting of the internalized human choriogonadotropin and the density of cell surface hLHR.
Takashi Hirakawa;Colette Galet;Mikiko Kishi;Mario Ascoli.
Journal of Biological Chemistry (2003)
The inositol phosphate/diacylglycerol pathway in MA-10 Leydig tumor cells. Activation by arginine vasopressin and lack of effect of epidermal growth factor and human choriogonadotropin.
M Ascoli;O P Pignataro;D L Segaloff.
Journal of Biological Chemistry (1989)
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