His scientific interests lie mostly in Genetics, Genome-wide association study, Single-nucleotide polymorphism, Alzheimer's disease and Apolipoprotein E. His Genetics research incorporates themes from Disease and Late onset. His Genome-wide association study research integrates issues from Neurodegeneration, Linkage disequilibrium and Allele frequency.
His work in Single-nucleotide polymorphism addresses subjects such as Neuropathology, which are connected to disciplines such as Corticobasal degeneration, Tauopathy, Degenerative disease and Progressive supranuclear palsy. His Alzheimer's disease study combines topics from a wide range of disciplines, such as Biological pathway, Gene expression, Case-control study and Genomics. His study on Apolipoprotein E also encompasses disciplines like
His primary areas of study are Genetics, Disease, Computational biology, Genome-wide association study and Gene. His Genetics study integrates concerns from other disciplines, such as Alzheimer's disease and Apolipoprotein E. The study incorporates disciplines such as Whole genome sequencing and Bioinformatics in addition to Disease.
Li-San Wang combines subjects such as ENCODE, Genomics, Expression quantitative trait loci, Genetic variants and Small RNA with his study of Computational biology. His research integrates issues of SNP, Linkage disequilibrium, Genetic association and Functional genomics in his study of Genome-wide association study. He interconnects Phylogenetics, Neighbor joining and Phylogenetic tree in the investigation of issues within Genome.
His scientific interests lie mostly in Disease, Computational biology, Genetics, Genomics and Genome. The Disease study combines topics in areas such as Cognition and Whole genome sequencing. His Computational biology research is multidisciplinary, incorporating perspectives in Genome-wide association study, Functional genomics, Expression quantitative trait loci and Epigenetics, Gene.
Li-San Wang focuses mostly in the field of Genome-wide association study, narrowing it down to topics relating to Locus and, in certain cases, Linkage disequilibrium. The various areas that he examines in his Genetics study include Stroke and Pleiotropy. His Genome research includes themes of Exome sequencing, Promoter and Chromosome.
Disease, Gene, Computational biology, Genome-wide association study and Dementia are his primary areas of study. His studies deal with areas such as Genotype, Cognition, Allele and Mendelian inheritance as well as Disease. His Computational biology study incorporates themes from Histone H3, Epigenetics, Transcription, Transcriptome and Epigenome.
Genome-wide association study is a subfield of Genetics that Li-San Wang explores. His Genetics study frequently links to adjacent areas such as Effects of sleep deprivation on cognitive performance. Li-San Wang has researched SORL1 in several fields, including Immunoglobulin gene, Genome, Genotyping, Alzheimer's disease and Haplotype.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease
Jean-Charles Lambert;Jean-Charles Lambert;Jean-Charles Lambert;Carla A Ibrahim-Verbaas;Denise Harold;Adam C Naj.
Nature Genetics (2013)
Synaptic, transcriptional and chromatin genes disrupted in autism
Silvia De Rubeis;Xin-Xin He;Arthur P Goldberg;Christopher S. Poultney.
Nature (2014)
Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's disease.
Adam C. Naj;Gyungah Jun;Gary W. Beecham;Li-San Wang.
Nature Genetics (2011)
Patterns and rates of exonic de novo mutations in autism spectrum disorders
Benjamin M. Neale;Yan Kou;Li Liu;Avi Ma'Ayan.
Nature (2012)
Enhancing CD8 T-cell memory by modulating fatty acid metabolism.
Erika L. Pearce;Matthew C. Walsh;Pedro J. Cejas;Gretchen M. Harms.
Nature (2009)
Gene-wide analysis detects two new susceptibility genes for Alzheimer's disease.
Valentina Escott-Price;Céline Bellenguez;Li-San Wang;Seung-Hoan Choi.
PLOS ONE (2014)
Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease
Rebecca Sims;Sven J. Van Der Lee;Adam C. Naj;Céline Bellenguez;Céline Bellenguez.
Nature Genetics (2017)
Rare structural variants found in attention-deficit hyperactivity disorder are preferentially associated with neurodevelopmental genes
J. Elia;X. Gai;H. M. Xie;J. C. Perin.
Molecular Psychiatry (2010)
Identification of common variants influencing risk of the tauopathy progressive supranuclear palsy
Günter U. Höglinger;Nadine M. Melhem;Dennis W. Dickson;Patrick M A Sleiman.
Nature Genetics (2011)
Common variants at 7p21 are associated with frontotemporal lobar degeneration with TDP-43 inclusions
Vivianna M. Van Deerlin;Patrick M A Sleiman;Maria Martinez-Lage;Maria Martinez-Lage;Alice Chen-Plotkin.
Nature Genetics (2010)
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