2007 - Hellman Fellow
His main research concerns Microbiology, Cell biology, Biochemistry, Escherichia coli and Plasmid. His Microbiology study combines topics in areas such as Bacteriophage, MRSA bacteremia, Methicillin-resistant Staphylococcus aureus, Daptomycin and Bacteria. The concepts of his Cell biology study are interwoven with issues in FtsZ, Cell division, Cytoskeleton, Peptidoglycan and SecYEG Translocon.
The Biochemistry study combines topics in areas such as Staphylococcus epidermidis and Staphylococcus aureus. The study incorporates disciplines such as Transcription and Protein folding in addition to Escherichia coli. Joe Pogliano works mostly in the field of Plasmid, limiting it down to concerns involving Molecular biology and, occasionally, DNA.
His primary areas of study are Microbiology, Cell biology, Staphylococcus aureus, Antibiotics and Daptomycin. His Microbiology research includes elements of Vancomycin and Methicillin-resistant Staphylococcus aureus. His studies deal with areas such as Bacteriophage, DNA, Cell division, Bacillus subtilis and Cytoskeleton as well as Cell biology.
His work in Staphylococcus aureus addresses issues such as Virulence, which are connected to fields such as Function. His Antibiotics research integrates issues from Bacteria and Antimicrobial peptides. While the research belongs to areas of Daptomycin, he spends his time largely on the problem of Enterococcus faecium, intersecting his research to questions surrounding Ampicillin and Enterococcus.
His primary areas of investigation include Microbiology, Antibiotics, Staphylococcus aureus, Antibiotic resistance and Antimicrobial. Specifically, his work in Microbiology is concerned with the study of Azithromycin. His Antibiotics study integrates concerns from other disciplines, such as In vivo and Mechanism of action.
In his study, which falls under the umbrella issue of In vivo, Cell biology is strongly linked to DNA replication. His studies in Cell biology integrate themes in fields like DNA and Capsid. Within one scientific family, he focuses on topics pertaining to Virulence under Staphylococcus aureus, and may sometimes address concerns connected to Computational biology, Transcriptome, Gene expression, Transcription factor and Function.
Joe Pogliano spends much of his time researching Antibiotics, Gene, Cell biology, Microbiology and Bacteriophage. His Antibiotics research is multidisciplinary, relying on both Antimicrobial and Acinetobacter baumannii. His work carried out in the field of Antimicrobial brings together such families of science as Streptococcus pneumoniae, Neisseria gonorrhoeae and Staphylococcus aureus.
His Acinetobacter baumannii research is multidisciplinary, incorporating elements of Escherichia coli, Azithromycin, Bacillus subtilis and Mechanism of action. His work in the fields of Gene, such as Gene expression, Transcriptome and Transcription factor, overlaps with other areas such as CCPA. His research in Cell biology intersects with topics in CRISPR and Endonuclease.
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Bacteriophage adhering to mucus provide a non-host-derived immunity.
Jeremy J. Barr;Rita Auro;Mike Furlan;Katrine L. Whiteson.
Proceedings of the National Academy of Sciences of the United States of America (2013)
SecA membrane cycling at SecYEG is driven by distinct ATP binding and hydrolysis events and is regulated by SecD and SecF
Anastassios Economou;Joseph A. Pogliano;Jonathan Beckwith;Donald B. Oliver.
Regulation of Escherichia coli cell envelope proteins involved in protein folding and degradation by the Cpx two-component system
J. Pogliano;A. S. Lynch;Dominique Belin;E. C. Lin.
Genes & Development (1997)
Daptomycin mediated reorganization of membrane architecture causes mislocalization of essential cell division proteins
Joe Pogliano;Nicolas Pogliano;Jared A. Silverman.
Journal of Bacteriology (2012)
Use of Antistaphylococcal β-Lactams to Increase Daptomycin Activity in Eradicating Persistent Bacteremia Due to Methicillin-Resistant Staphylococcus aureus: Role of Enhanced Daptomycin Binding
Abhay Dhand;Arnold S. Bayer;Arnold S. Bayer;Joseph Pogliano;Soo-Jin Yang;Soo-Jin Yang.
Clinical Infectious Diseases (2011)
ParE toxin encoded by the broad‐host‐range plasmid RK2 is an inhibitor of Escherichia coli gyrase
Yong Jiang;Joe Pogliano;Donald R. Helinski;Igor Konieczny.
Molecular Microbiology (2002)
SecD and SecF facilitate protein export in Escherichia coli.
J.A. Pogliano;J. Beckwith.
The EMBO Journal (1994)
Inactivation of FtsI inhibits constriction of the FtsZ cytokinetic ring and delays the assembly of FtsZ rings at potential division sites
Joe Pogliano;Kit Pogliano;David S. Weiss;Richard Losick.
Proceedings of the National Academy of Sciences of the United States of America (1997)
A vital stain for studying membrane dynamics in bacteria: a novel mechanism controlling septation during Bacillus subtilis sporulation
Joe Pogliano;Nick Osborne;Marc D. Sharp;Angelica Abanes-De Mello.
Molecular Microbiology (1999)
Treadmilling of a prokaryotic tubulin-like protein, TubZ, required for plasmid stability in Bacillus thuringiensis
Rachel A. Larsen;Christina Cusumano;Akina Fujioka;Grace Lim-Fong.
Genes & Development (2007)
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