Genetics, Molecular biology, Antigen, Virology and Virus are his primary areas of study. His Molecular biology research is multidisciplinary, relying on both Retinoblastoma protein, Clone, Gene product, Ribosome and Adenocarcinoma. His Antigen research is multidisciplinary, incorporating elements of Carcinogenesis, SV40 large T antigen and Cell cycle.
His study looks at the intersection of SV40 large T antigen and topics like DNA-binding protein with E2F. His work in the fields of Virology, such as Capsid, overlaps with other areas such as Medical school. As part of the same scientific family, he usually focuses on Virus, concentrating on Viral evolution and intersecting with Metagenomics, Human virome, Genomics, Plant virus and Genetic diversity.
James M. Pipas mostly deals with Antigen, Virology, Molecular biology, Virus and Cell biology. His Antigen research incorporates elements of SV40 large T antigen, Mutation, Virion assembly, Mutant and Cytotoxic T cell. Many of his studies on Virology apply to Viral evolution as well.
His Molecular biology study also includes fields such as
James M. Pipas focuses on Virus, Genetics, Gene, Virology and Genome. His study looks at the relationship between Virus and topics such as Whole genome sequencing, which overlap with Open reading frame and Metagenomics. His Viral metagenomics, E2F and DNA Tumor Virus study in the realm of Gene connects with subjects such as Biological classification.
James M. Pipas combines subjects such as Carcinogenesis, Cell and Liver cancer with his study of Virology. His study in Cell is interdisciplinary in nature, drawing from both Tumor necrosis factor alpha and Antigen. His Genome study combines topics from a wide range of disciplines, such as Computational biology and DNA sequencing.
His primary areas of investigation include Genome, Cancer research, Gene, Virus and Immunology. His studies in Genome integrate themes in fields like Virology, Carcinogenesis, Computational biology, DNA sequencing and Metagenomics. James M. Pipas has included themes like Merkel cell, Merkel cell carcinoma and Merkel cell polyomavirus in his Virology study.
His Cancer research study incorporates themes from Cancer and E2F, Gene expression. His work on Viral evolution, Phylogenetic tree and Viral metagenomics as part of general Gene research is often related to Biological classification, thus linking different fields of science. His Virus research is multidisciplinary, relying on both Liver cancer, Cell culture, Human virome and IRF3.
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SV40-encoded microRNAs regulate viral gene expression and reduce susceptibility to cytotoxic T cells
Christopher S. Sullivan;Adam T. Grundhoff;Satvir Tevethia;James M. Pipas.
SV40 large T antigen targets multiple cellular pathways to elicit cellular transformation
Deepika Ahuja;M Teresa Sáenz-Robles;James M Pipas.
The retinoblastoma susceptibility gene product undergoes cell cycle-dependent dephosphorylation and binding to and release from SV40 large T.
John W. Ludlow;John Shon;James M. Pipas;David M. Livingston.
Specific repression of TATA-mediated but not initiator-mediated transcription by wild-type p53
David H. Mack;Jai Vartikar;James M. Pipas;Laimonis A. Laimins;Laimonis A. Laimins.
pp60c-src activation in human colon carcinoma.
C A Cartwright;M P Kamps;A I Meisler;J M Pipas.
Journal of Clinical Investigation (1989)
Common and unique features of T antigens encoded by the polyomavirus group.
J M Pipas.
Journal of Virology (1992)
The amino-terminal transforming region of simian virus 40 large T and small t antigens functions as a J domain.
Ashok Srinivasan;Amie J. Mcclellan;Jai Vartikar;Ian Marks.
Molecular and Cellular Biology (1997)
T Antigens of Simian Virus 40: Molecular Chaperones for Viral Replication and Tumorigenesis
Christopher S. Sullivan;James M. Pipas.
Microbiology and Molecular Biology Reviews (2002)
Ribosomal protein genes are overexpressed in colorectal cancer : isolation of a cDNA clone encoding the human S3 ribosomal protein
Kay Pogue-Geile;John R. Geiser;Min Shu;Carla Miller.
Molecular and Cellular Biology (1991)
DnaJ/hsp40 chaperone domain of SV40 large T antigen promotes efficient viral DNA replication.
K S Campbell;K P Mullane;I A Aksoy;H Stubdal.
Genes & Development (1997)
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