What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Enzyme
His main research concerns Immunology, Apoptosis, Tumor necrosis factor alpha, Cell biology and Microglia.
He has researched Immunology in several fields, including Pharmacology, Neuroprotection, Neurodegeneration and Astrocyte.
His Apoptosis research is multidisciplinary, incorporating elements of Platelet-activating factor, Neurotoxicity and Transcription factor.
His biological study spans a wide range of topics, including Interleukin, Cytokine, Cancer research and Monocyte.
His Cell biology research includes themes of Excitotoxicity, Programmed cell death and Receptor, Biochemistry.
His research in Microglia intersects with topics in Pathogenesis, Pathology, Glutamate receptor, Neurotoxin and Central nervous system.
His most cited work include:
- Mitochondrial membrane potential probes and the proton gradient: a practical usage guide. (657 citations)
- Cytokines and arachidonic metabolites produced during human immunodeficiency virus (HIV)-infected macrophage-astroglia interactions: implications for the neuropathogenesis of HIV disease. (479 citations)
- Paraquat elicited neurobehavioral syndrome caused by dopaminergic neuron loss. (350 citations)
What are the main themes of his work throughout his whole career to date?
Harris A. Gelbard mainly investigates Immunology, Microglia, Cell biology, Pharmacology and Neuroprotection.
The various areas that he examines in his Immunology study include Central nervous system and Astrocyte.
His Microglia research includes elements of Neurotoxicity, Glutamate receptor, Hippocampal formation, Neuroscience and Signal transduction.
His Cell biology study incorporates themes from Receptor, Apoptosis, Biochemistry and Neuron.
His study in Apoptosis is interdisciplinary in nature, drawing from both Platelet-activating factor and Transcription factor.
His Neuroprotection research incorporates elements of Dementia and Neurodegeneration.
He most often published in these fields:
- Immunology (29.94%)
- Microglia (22.60%)
- Cell biology (14.69%)
What were the highlights of his more recent work (between 2016-2021)?
- Pharmacology (12.99%)
- Microgliosis (5.65%)
- Autophagy (2.26%)
In recent papers he was focusing on the following fields of study:
His primary scientific interests are in Pharmacology, Microgliosis, Autophagy, Neurocognitive and Synapse.
Harris A. Gelbard has included themes like Neuroscience research and Bacterial endotoxin in his Pharmacology study.
His studies deal with areas such as Protease inhibitor, Clonidine, Macrophage and Atazanavir as well as Autophagy.
His Macrophage research focuses on Immunology and how it connects with Drug discovery.
His Synapse study also includes fields such as
- Knockout mouse which intersects with area such as Multiple sclerosis, Nervous system, HIV-associated neurocognitive disorder and Proinflammatory cytokine,
- Experimental autoimmune encephalomyelitis that connect with fields like Microglia, Excitatory postsynaptic potential, LRRK2 and Neuroprotection.
Harris A. Gelbard usually deals with Kinase and limits it to topics linked to Programmed cell death and Cell biology.
Between 2016 and 2021, his most popular works were:
- Autophagy facilitates macrophage depots of sustained-release nanoformulated antiretroviral drugs (31 citations)
- URMC-099 facilitates amyloid-β clearance in a murine model of Alzheimer's disease. (18 citations)
- Mixed-lineage kinase 3 pharmacological inhibition attenuates murine nonalcoholic steatohepatitis (18 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Enzyme
Harris A. Gelbard focuses on Pharmacology, Microglia, Cognitive decline, Neuroprotection and Perioperative.
His work on Liver injury and Intraperitoneal injection as part of general Pharmacology research is frequently linked to Saturated fat and Homeostatic model assessment, thereby connecting diverse disciplines of science.
His research investigates the connection with Microglia and areas like Synapse which intersect with concerns in Multiple sclerosis.
His work focuses on many connections between Cognitive decline and other disciplines, such as Neurocognitive, that overlap with his field of interest in Neuroinflammation and Neurology.
His work carried out in the field of Neuroprotection brings together such families of science as Neurodegeneration, Kinase, Excitatory postsynaptic potential and LRRK2.
His Microgliosis research is within the category of Immunology.
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