Hagir B. Suliman mainly investigates Mitochondrial biogenesis, Mitochondrion, Molecular biology, Oxidative phosphorylation and Heme oxygenase. Hagir B. Suliman focuses mostly in the field of Mitochondrial biogenesis, narrowing it down to matters related to Intensive care and, in some cases, Mitochondrial Size. His Mitochondrion study combines topics from a wide range of disciplines, such as Internal medicine, Endothelial NOS and Endocrinology.
His study looks at the relationship between Molecular biology and fields such as NRF1, as well as how they intersect with chemical problems. His Oxidative phosphorylation research includes themes of Oxidative stress and TFAM. His study on HMOX1 is often connected to Doxorubicin as part of broader study in Heme oxygenase.
Hagir B. Suliman spends much of his time researching Mitochondrial biogenesis, Mitochondrion, Molecular biology, Cell biology and Oxidative stress. Hagir B. Suliman focuses mostly in the field of Mitochondrial biogenesis, narrowing it down to topics relating to Heme oxygenase and, in certain cases, Hemeprotein. The concepts of his Mitochondrion study are interwoven with issues in Endocrinology, Internal medicine, SOD2 and Pathology.
His Molecular biology research is multidisciplinary, incorporating elements of NFKB1, Promoter, Lung injury and Toll-like receptor. His Cell biology research is multidisciplinary, incorporating perspectives in Inflammation, mitochondrial fusion, Cell damage and Mitophagy. Hagir B. Suliman works mostly in the field of Oxidative stress, limiting it down to concerns involving Oxidative phosphorylation and, occasionally, Glutathione and Cytochrome c oxidase.
Hagir B. Suliman mainly focuses on Mitochondrial biogenesis, Mitochondrion, Sepsis, Molecular biology and Cell biology. His Mitochondrial biogenesis study focuses on TFAM in particular. His work in TFAM covers topics such as NRF1 which are related to areas like Tumor necrosis factor alpha, Andrology and Heme oxygenase.
His Mitochondrion study introduces a deeper knowledge of Biochemistry. Hagir B. Suliman combines subjects such as Dialysis, Lung injury, Lung and Immunohistochemistry with his study of Sepsis. His Cell biology study combines topics from a wide range of disciplines, such as mitochondrial fusion, Oxidative stress and Mitophagy.
His primary scientific interests are in Mitochondrial biogenesis, Mitochondrion, Cell biology, Mitophagy and Molecular biology. Mitochondrial biogenesis is a subfield of Biochemistry that Hagir B. Suliman studies. His Biochemistry research includes elements of Reperfusion injury and NFE2L2.
His research in Cell biology intersects with topics in mitochondrial fusion and Oxidative stress. His study looks at the relationship between Molecular biology and topics such as TFAM, which overlap with NRF1, FIS1 and Mitochondrial fission. Hagir B. Suliman works mostly in the field of Autophagy, limiting it down to topics relating to Programmed cell death and, in certain cases, HMOX1, Cardiac fibrosis, Autophagosome, Lung injury and Bronchoalveolar lavage.
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Heme Oxygenase-1 Regulates Cardiac Mitochondrial Biogenesis via Nrf2-Mediated Transcriptional Control of Nuclear Respiratory Factor-1
Claude A. Piantadosi;Martha Sue Carraway;Abdelwahid Babiker;Hagir B. Suliman.
Circulation Research (2008)
Survival in critical illness is associated with early activation of mitochondrial biogenesis.
Jane E. Carré;Jean Christophe Orban;Lorenza Re;Karen Felsmann.
American Journal of Respiratory and Critical Care Medicine (2010)
EXTRACELLULAR SUPEROXIDE DISMUTASE IN THE AIRWAYS OF TRANSGENIC MICE REDUCES INFLAMMATION AND ATTENUATES LUNG TOXICITY FOLLOWING HYPEROXIA
Rodney J. Folz;Amir M. Abushamaa;Hagir B. Suliman.
Journal of Clinical Investigation (1999)
Extracellular superoxide dismutase.
Eva Nozik-Grayck;Hagir B. Suliman;Claude A. Piantadosi.
The International Journal of Biochemistry & Cell Biology (2005)
Lipopolysaccharide induces oxidative cardiac mitochondrial damage and biogenesis
Hagir B. Suliman;Karen E. Welty-Wolf;Martha Sue Carraway;Lynn Tatro.
Cardiovascular Research (2004)
The CO/HO system reverses inhibition of mitochondrial biogenesis and prevents murine doxorubicin cardiomyopathy
Hagir B. Suliman;Martha Sue Carraway;Abdelwahid S. Ali;Chrystal M. Reynolds.
Journal of Clinical Investigation (2007)
Mitochondrial transcription factor-A induction by redox activation of nuclear respiratory factor-1
Claude A. Piantadosi;Hagir B. Suliman.
Journal of Biological Chemistry (2006)
Heme Oxygenase-1 Couples Activation of Mitochondrial Biogenesis to Anti-inflammatory Cytokine Expression *
Claude A. Piantadosi;Crystal M. Withers;Raquel R. Bartz;Nancy Chou MacGarvey.
Journal of Biological Chemistry (2011)
Mitochondrial Quality Control as a Therapeutic Target
Hagir B. Suliman;Claude A. Piantadosi.
Pharmacological Reviews (2016)
Lipopolysaccharide stimulates mitochondrial biogenesis via activation of nuclear respiratory factor-1.
Hagir B. Suliman;Martha S. Carraway;Karen E. Welty-Wolf;A.Richard Whorton.
Journal of Biological Chemistry (2003)
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