D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 57 Citations 12,129 115 World Ranking 9340 National Ranking 324

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Internal medicine
  • Genetics

Her primary areas of investigation include Elastin, Elastic fiber assembly, Pathology, Extracellular matrix and Elastic fiber. Her research on Elastin focuses in particular on Tropoelastin. The study incorporates disciplines such as Organ culture, Artery and Dermis in addition to Elastic fiber assembly.

Her Pathology research is multidisciplinary, relying on both TGF beta signaling pathway, Integrin binding, Anatomy and Fibrillin. Her work on Fibulin as part of general Extracellular matrix study is frequently connected to Macular degeneration, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them. As part of one scientific family, Elaine C. Davis deals mainly with the area of Elastic fiber, narrowing it down to issues related to the Biophysics, and often Extracellular matrix assembly and Scaffold.

Her most cited work include:

  • A syndrome of altered cardiovascular, craniofacial, neurocognitive and skeletal development caused by mutations in TGFBR1 or TGFBR2 (1278 citations)
  • Defective angiogenesis in mice lacking endoglin. (746 citations)
  • Elastin is an essential determinant of arterial morphogenesis (627 citations)

What are the main themes of her work throughout her whole career to date?

Her primary areas of study are Elastin, Cell biology, Extracellular matrix, Pathology and Tropoelastin. The Elastin study combines topics in areas such as Biophysics, Elastic fiber, Anatomy and Fibulin. Her Cell biology research includes elements of Endothelial stem cell and Secretory protein.

Her Extracellular matrix research integrates issues from Ehlers–Danlos syndrome, Cell signaling and Biomedical engineering. Her work on Connective Tissue Disorder as part of her general Pathology study is frequently connected to Supravalvular aortic stenosis, thereby bridging the divide between different branches of science. Her research in Tropoelastin intersects with topics in Golgi apparatus, Brefeldin A, Molecular biology and Fibrillin.

She most often published in these fields:

  • Elastin (40.50%)
  • Cell biology (30.58%)
  • Extracellular matrix (27.27%)

What were the highlights of her more recent work (between 2014-2021)?

  • Cell biology (30.58%)
  • Extracellular matrix (27.27%)
  • Pathology (26.45%)

In recent papers she was focusing on the following fields of study:

Elaine C. Davis mainly investigates Cell biology, Extracellular matrix, Pathology, Fibulin and Elastin. Her work deals with themes such as Endothelial stem cell, Calcification and Peptide, which intersect with Cell biology. The concepts of her Extracellular matrix study are interwoven with issues in Ehlers–Danlos syndrome, Cell signaling and Ultimate tensile strength.

Her Pathology study combines topics from a wide range of disciplines, such as Thoracic aortic aneurysm, Angiotensin II and Fragmentation. The various areas that she examines in her Fibulin study include Mitochondrial respiration, Mitochondrion, Mutant and Function. Her study in Elastin is interdisciplinary in nature, drawing from both Lysyl oxidase, Marfan syndrome and Biomedical engineering.

Between 2014 and 2021, her most popular works were:

  • Function of latent TGFβ binding protein 4 and fibulin 5 in elastogenesis and lung development. (32 citations)
  • Loss of fibulin-4 disrupts collagen synthesis and maturation: implications for pathology resulting from EFEMP2 mutations (31 citations)
  • Angiopoietin-1 enhances skeletal muscle regeneration in mice (30 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Genetics

Her primary scientific interests are in Extracellular matrix, Elastin, Fibulin, Pathology and Angiotensin II. Her Extracellular matrix research includes themes of Vascular smooth muscle, Biomedical engineering and Latent TGF-beta binding protein. Her studies in Elastin integrate themes in fields like Lysyl oxidase and Connective Tissue Disorder.

Her Pathology study combines topics in areas such as Thoracic aortic aneurysm, Thrombospondin 1, Mechanotransduction, Smooth muscle and Marfan syndrome. Her Angiotensin II research incorporates themes from Anatomy, Cytoskeleton and Actin. Her FBLN5 study incorporates themes from Binding protein, Elastic fiber assembly, Immunology, Molecular biology and Lung.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

A syndrome of altered cardiovascular, craniofacial, neurocognitive and skeletal development caused by mutations in TGFBR1 or TGFBR2

Bart L Loeys;Junji Chen;Enid R Neptune;Daniel P Judge.
Nature Genetics (2005)

1776 Citations

Defective angiogenesis in mice lacking endoglin.

Dean Y. Li;Lise K. Sorensen;Benjamin S. Brooke;Lisa D. Urness.
Science (1999)

1174 Citations

Elastin is an essential determinant of arterial morphogenesis

Dean Y. Li;Benjamin Brooke;Elaine C. Davis;Robert P. Mecham.
Nature (1998)

881 Citations

Fibulin-5 is an elastin-binding protein essential for elastic fibre development in vivo

Hiromi Yanagisawa;Elaine C Davis;Barry C Starcher;Takashi Ouchi.
Nature (2002)

651 Citations

Novel arterial pathology in mice and humans hemizygous for elastin

Dean Y. Li;Gilles Faury;Douglas G. Taylor;Elaine C. Davis.
Journal of Clinical Investigation (1998)

417 Citations

Mutations in the Gene Encoding the RER Protein FKBP65 Cause Autosomal-Recessive Osteogenesis Imperfecta

Yasemin Alanay;Hrispima Avaygan;Natalia Camacho;G. Eda Utine.
American Journal of Human Genetics (2010)

334 Citations

Mutations in Fibrillin-1 Cause Congenital Scleroderma: Stiff Skin Syndrome

B. L. Loeys;B. L. Loeys;E. E. Gerber;D. Riegert-Johnson;S. Iqbal.
Science Translational Medicine (2010)

274 Citations

Smooth muscle cell to elastic lamina connections in developing mouse aorta. Role in aortic medial organization.

E C Davis.
Laboratory Investigation (1993)

227 Citations

Elastic fiber formation: a dynamic view of extracellular matrix assembly using timer reporters.

Beth A. Kozel;Brenda J. Rongish;Andras Czirok;Julia Zach.
Journal of Cellular Physiology (2006)

199 Citations

Elastic Fiber Structure and Assembly

Robert P. Mecham;Elaine C. Davis.
Extracellular Matrix Assembly and Structure (1994)

191 Citations

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