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Michelle Letarte

Michelle Letarte

D-Index & Metrics

Biology and Biochemistry

D-Index
68
Citations
18731
World Ranking
7716
National Ranking
267

Overview

Michelle Letarte is affiliated with the University of Toronto in Canada. Their research primarily spans the field of Medicine, with contributions to subfields such as Genetics, Pulmonary and Respiratory Medicine, Social Psychology, General Agricultural and Biological Sciences, and Management, Monitoring, Policy and Law.

The scientist's work covers several main topics, including vascular anomalies and treatments, tracheal and airway disorders, animal and plant science education, diverse educational innovation studies, and conservation, ecology, and wildlife education.

Among the recent papers authored by Michelle Letarte are:

  • Potential Second-Hits in Hereditary Hemorrhagic Telangiectasia, 2020, Journal of Clinical Medicine
  • Conducting Authentic Moth Research with Students to Encourage Scientific Inquiry, 2024, Science Scope

Frequent coauthors collaborating with Michelle Letarte include:

  • Carmelo Bernabéu
  • Pınar Bayrak-Toydemir
  • Jamie McDonald
  • Brian Keas
  • Peter White

Michelle Letarte's publications have appeared mainly in the following scientific venues:

  • Journal of Clinical Medicine
  • Science Scope

Best Publications

  • Soluble endoglin contributes to the pathogenesis of preeclampsia.

    Shivalingappa Venkatesha;Mourad Toporsian;Chun Lam;Jun-ichi Hanai

  • Endoglin, a TGF-β binding protein of endothelial cells, is the gene for hereditary haemorrhagic telangiectasia type 1

    McAllister Ka;Grogg Km;Johnson Dw;Gallione Cj

  • Endoglin Is an Accessory Protein That Interacts with the Signaling Receptor Complex of Multiple Members of the Transforming Growth Factor-β Superfamily

    Nadia Pece Barbara;Jeffrey L. Wrana;Michelle Letarte

  • A murine model of hereditary hemorrhagic telangiectasia.

    Annie Bourdeau;Daniel J. Dumont;Michelle Letarte

  • Hereditary haemorrhagic telangiectasia: current views on genetics and mechanisms of disease

    Salma A Abdalla;Michelle Letarte

  • Up-regulation of endoglin on vascular endothelial cells in human solid tumors: implications for diagnosis and therapy.

    Francis J. Burrows;Elaine J. Derbyshire;Pier Luigi Tazzari;Peter Amlot

  • Endoglin modulates cellular responses to TGF-beta 1.

    P Lastres;A Letamendía;H Zhang;C Rius

  • A model of human acute lymphoblastic leukemia in immune-deficient SCID mice

    Suzanne Kamel-Reid;Michelle Letarte;Christian Sirard;Monica Doedens

  • A Role for Endoglin in Coupling eNOS Activity and Regulating Vascular Tone Revealed in Hereditary Hemorrhagic Telangiectasia

    Mourad Toporsian;Robert Gros;Mohammed G. Kabir;Sonia Vera

  • Identification and expression of two forms of the human transforming growth factor-β-binding protein endoglin with distinct cytoplasmic regions

    Teresa Bellón;Angel Corbi;Pedro Lastres;Carmela Calés

  • Regulated expression on human macrophages of endoglin, an Arg-Gly-Asp-containing surface antigen.

    Pedro Lastres;Teresa Bellon;Carlos Cabañas;Francisco Sanchez-Madrid

  • Identification of distinct epitopes of endoglin, an RGD-containing glycoprotein of endothelial cells, leukemic cells, and syncytiotrophoblasts.

    Anne Gougos;Sylvie St Jacques;Adonna Greaves;Philip J. O'Connell

  • Endoglin regulates trophoblast differentiation along the invasive pathway in human placental villous explants.

    I. Caniggia;C. V. Taylor;J. W. K. Ritchie;S. J. Lye

  • Molecular characterization and in situ localization of murine endoglin reveal that it is a transforming growth factor-beta binding protein of endothelial and stromal cells

    S St-Jacques;U Cymerman;N Pece;M Letarte

  • VE-cadherin is a critical endothelial regulator of TGF-β signalling

    Noemi Rudini;Angelina Felici;Costanza Giampietro;MariaGrazia Lampugnani

  • Analysis of ALK-1 and endoglin in newborns from families with hereditary hemorrhagic telangiectasia type 2

    Salma A. Abdalla;Nadia Pece-Barbara;Sonia Vera;Esther Tapia

  • Mutant Endoglin in Hereditary Hemorrhagic Telangiectasia Type 1 Is Transiently Expressed Intracellularly and Is Not a Dominant Negative

    Nadia Pece;Sonia Vera;Urszula Cymerman;Robert I. White

  • Expression Analysis of Four Endoglin Missense Mutations Suggests That Haploinsufficiency Is the Predominant Mechanism for Hereditary Hemorrhagic Telangiectasia Type 1

    Nadia Pece-Barbara;Urszula Cymerman;Sonia Vera;Douglas A. Marchuk

  • Endoglin-deficient mice, a unique model to study hereditary hemorrhagic telangiectasia.

    Annie Bourdeau;Marie E Faughnan;Michelle Letarte

  • Primary pulmonary hypertension in families with hereditary haemorrhagic telangiectasia

    S A Abdalla;C J Gallione;R J Barst;E M Horn

Frequent Co-Authors

Carmelo Bernabeu
Carmelo Bernabeu Spanish National Research Council
José M. López-Novoa
José M. López-Novoa University of Salamanca
F. Stuart Foster
F. Stuart Foster University of Toronto
Erwin W. Gelfand
Erwin W. Gelfand University of Colorado Anschutz Medical Campus
Robert S. Kerbel
Robert S. Kerbel University of Toronto
Miguel Quintanilla
Miguel Quintanilla Spanish National Research Council
Jeffrey L. Wrana
Jeffrey L. Wrana Lunenfeld-Tanenbaum Research Institute
Barry P. Rosen
Barry P. Rosen Florida International University
Elisabetta Dejana
Elisabetta Dejana Uppsala University
Marcy E. MacDonald
Marcy E. MacDonald Harvard University

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