Christian Becker mainly focuses on Genetics, Cancer research, Internal medicine, Missense mutation and Endocrinology. His Genetics study frequently draws connections to other fields, such as Molecular biology. His studies deal with areas such as Genome, Human genome, Gene expression profiling, Mutation and Immunology as well as Cancer research.
Christian Becker combines subjects such as Single-nucleotide polymorphism and Allele with his study of Internal medicine. His Missense mutation study combines topics in areas such as Disease gene identification and Nephrotic syndrome. His Endocrinology study incorporates themes from Cilium, Renal-hepatic-pancreatic dysplasia and Pathology.
Christian Becker spends much of his time researching Genetics, Cell biology, Cancer research, Single-nucleotide polymorphism and Missense mutation. His work often combines Genetics and Gene mapping studies. His research in Cell biology intersects with topics in Gene expression, Receptor, Olfactory epithelium, RNA and Chromatin.
The various areas that Christian Becker examines in his Cancer research study include Immunohistochemistry, Cancer, Internal medicine, Endocrinology and Gene expression profiling. His Single-nucleotide polymorphism research integrates issues from Odds ratio and Allele. His Disease gene identification research includes elements of Nephrotic syndrome and Autosome.
His primary scientific interests are in Cell biology, Hearing loss, Transcriptome, Gene and Interphase. The various areas that Christian Becker examines in his Cell biology study include Mutation, Mitochondrial DNA and DNA replication. His Hearing loss study incorporates themes from Molecular analysis, Etiology, Genetics and Candidate gene.
His Transcriptome research is multidisciplinary, incorporating perspectives in HMGB2, Senescence, Cell type and Genomic organization. His Gene research is multidisciplinary, relying on both Domestication and Population structure. Christian Becker combines subjects such as Chromatin and Mitosis with his study of Interphase.
Gene, Cell biology, Transcriptome, Carcinoma and Cancer research are his primary areas of study. The study incorporates disciplines such as Senescence and Cell type in addition to Gene. His work on Mitochondrion as part of general Cell biology research is often related to ENDOG, thus linking different fields of science.
His Transcriptome research is multidisciplinary, incorporating elements of Cell, In situ hybridization, Large cell, Lung and STK11. His biological study spans a wide range of topics, including Immunohistochemistry, Cancer, Breast cancer, Fusion transcript and Pancreas. The concepts of his Cancer research study are interwoven with issues in Receptor and Breast carcinoma.
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Comprehensive genomic profiles of small cell lung cancer
Julie George;Jing Shan Lim;Se Jin Jang;Yupeng Cun.
Nature (2015)
Integrative genome analyses identify key somatic driver mutations of small-cell lung cancer
Martin Peifer;Lynnette Fernández-Cuesta;Martin L. Sos;Julie George.
Nature Genetics (2012)
Lifestyle transitions in plant pathogenic Colletotrichum fungi deciphered by genome and transcriptome analyses
Richard J. O'Connell;Michael R. Thon;Stéphane Hacquard;Stefan G. Amyotte.
Nature Genetics (2012)
The centrosomal protein nephrocystin-6 is mutated in Joubert syndrome and activates transcription factor ATF4.
John A. Sayer;John A. Sayer;Edgar A. Otto;John F. O'Toole;Gudrun Nurnberg.
Nature Genetics (2006)
Positional cloning uncovers mutations in PLCE1 responsible for a nephrotic syndrome variant that may be reversible.
Bernward Hinkes;Roger C. Wiggins;Rasheed Gbadegesin;Christopher N. Vlangos.
Nature Genetics (2006)
Correlation between genetic and geographic structure in Europe.
Oscar Lao;Timothy T. Lu;Michael Nothnagel;Olaf Junge.
Current Biology (2008)
Mutations in the Tight-Junction Gene Claudin 19 (CLDN19) Are Associated with Renal Magnesium Wasting, Renal Failure, and Severe Ocular Involvement
Martin Konrad;André Schaller;Dominik Seelow;Amit V. Pandey.
American Journal of Human Genetics (2006)
Familial hemophagocytic lymphohistiocytosis type 5 (FHL-5) is caused by mutations in Munc18-2 and impaired binding to syntaxin 11.
Udo zur Stadt;Jan Rohr;Wenke Seifert;Wenke Seifert;Florian Koch.
American Journal of Human Genetics (2009)
Mutations in STRA6 cause a broad spectrum of malformations including anophthalmia, congenital heart defects, diaphragmatic hernia, alveolar capillary dysplasia, lung hypoplasia, and mental retardation
Francesca Pasutto;Heinrich Sticht;Gerhard Hammersen;Gabriele Gillessen-Kaesbach.
American Journal of Human Genetics (2007)
A genome-wide association scan identifies the hepatic cholesterol transporter ABCG8 as a susceptibility factor for human gallstone disease.
Stephan Buch;Clemens Schafmayer;Henry Völzke;Christian Becker.
Nature Genetics (2007)
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