What is he best known for?
The fields of study he is best known for:
His primary areas of investigation include Cancer research, Cancer, Angiogenesis, STAT3 and Apoptosis.
His work deals with themes such as IκB kinase and Signal transduction, which intersect with Cancer research.
His research on Cancer frequently connects to adjacent areas such as Pharmacology.
His biological study spans a wide range of topics, including RNA interference, Transcription factor and Kinase, Protein tyrosine phosphatase.
His research integrates issues of Activator, Regulation of gene expression, Metastasis and Immunology in his study of Transcription factor.
His Apoptosis study incorporates themes from Cell culture, Cell growth and In vivo.
His most cited work include:
- Targeting the STAT3 signaling pathway in cancer: role of synthetic and natural inhibitors. (398 citations)
- Thymoquinone: Potential cure for inflammatory disorders and cancer (303 citations)
- Thymoquinone: Potential cure for inflammatory disorders and cancer (303 citations)
What are the main themes of his work throughout his whole career to date?
Alan Prem Kumar focuses on Cancer research, Cancer, Apoptosis, Metastasis and Cancer cell.
The Cancer research study combines topics in areas such as STAT3, Signal transduction, Breast cancer and Cell growth.
His research investigates the connection between STAT3 and topics such as Transcription factor that intersect with problems in Regulation of gene expression.
A large part of his Cancer studies is devoted to Carcinogenesis.
His studies in Apoptosis integrate themes in fields like Reactive oxygen species, Cell biology, Downregulation and upregulation, Thymoquinone and In vivo.
Alan Prem Kumar combines subjects such as Caspase and Cell cycle with his study of Pharmacology.
He most often published in these fields:
- Cancer research (56.07%)
- Cancer (36.82%)
- Apoptosis (21.76%)
What were the highlights of his more recent work (between 2019-2021)?
- Cancer research (56.07%)
- Cancer (36.82%)
- Cancer cell (15.48%)
In recent papers he was focusing on the following fields of study:
Alan Prem Kumar mainly focuses on Cancer research, Cancer, Cancer cell, Metastasis and PI3K/AKT/mTOR pathway.
His Cancer research research is multidisciplinary, incorporating perspectives in Wnt signaling pathway, Signal transduction, Cisplatin, CXCR4 and Doxorubicin.
Alan Prem Kumar has researched Cancer in several fields, including microRNA and Radiation therapy.
The various areas that Alan Prem Kumar examines in his Cancer cell study include Intermediate filament, Cytoskeleton and Microtubule, Actin, Cell biology.
His Metastasis study combines topics in areas such as Survival rate and Cell growth.
His study looks at the relationship between Cancer stem cell and topics such as STAT3, which overlap with Pharmacology.
Between 2019 and 2021, his most popular works were:
- Targeting AKT/mTOR in Oral Cancer: Mechanisms and Advances in Clinical Trials. (20 citations)
- Cytoskeletal Proteins in Cancer and Intracellular Stress: A Therapeutic Perspective (16 citations)
- Peruvoside targets apoptosis and autophagy through MAPK Wnt/β-catenin and PI3K/AKT/mTOR signaling pathways in human cancers (16 citations)
In his most recent research, the most cited papers focused on:
His scientific interests lie mostly in Cancer research, Cancer cell, PI3K/AKT/mTOR pathway, Metastasis and Cancer.
His work carried out in the field of Cancer research brings together such families of science as Wnt signaling pathway, Signal transduction and Programmed cell death.
His studies in Signal transduction integrate themes in fields like Cell cycle checkpoint, Cell cycle, Non-coding RNA, Cell migration and Cell growth.
His Cancer cell study incorporates themes from Curcumin, Intermediate filament, Nanocarriers and Docetaxel.
Alan Prem Kumar usually deals with PI3K/AKT/mTOR pathway and limits it to topics linked to Protein kinase B and Autophagy, Neuroprotection, Flavonoid, Chrysin and Pharmacology.
His studies deal with areas such as Carcinogenesis, microRNA and Angiogenesis as well as Metastasis.
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