What is he best known for?
The fields of study he is best known for:
Apoptosis, Cell biology, Mitochondrion, Programmed cell death and DNA fragmentation are his primary areas of study.
His work on Adenine nucleotide translocator, Reactive oxygen species and Inner mitochondrial membrane as part of his general Cell biology study is frequently connected to DNA damage, thereby bridging the divide between different branches of science.
Many of his studies involve connections with topics such as Mitochondrial permeability transition pore and Mitochondrion.
His research in Mitochondrial permeability transition pore intersects with topics in Cyclosporin a, Mitochondrial apoptosis-induced channel and Membrane potential.
He has included themes like Ceramide, Fragmentation and Cytolysis in his Programmed cell death study.
Philippe Marchetti has researched DNA fragmentation in several fields, including Molecular biology, Semen and Immunology.
His most cited work include:
- Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death. (1403 citations)
- Mitochondrial control of nuclear apoptosis (1285 citations)
- Reduction in mitochondrial potential constitutes an early irreversible step of programmed lymphocyte death in vivo. (1082 citations)
What are the main themes of his work throughout his whole career to date?
Philippe Marchetti spends much of his time researching Apoptosis, Cell biology, Mitochondrion, Programmed cell death and Cancer research.
Philippe Marchetti interconnects Cell, Reactive oxygen species and Immunology in the investigation of issues within Apoptosis.
His Adenine nucleotide translocator, Signal transduction and Inner mitochondrial membrane study in the realm of Cell biology interacts with subjects such as DNA damage.
His Mitochondrion research is multidisciplinary, relying on both Lamellarin D, Oxidative phosphorylation, Pharmacology and Mitochondrial permeability transition pore.
His study looks at the relationship between Mitochondrial permeability transition pore and topics such as Mitochondrial apoptosis-induced channel, which overlap with Mitochondrial fission and DNAJA3.
The concepts of his Programmed cell death study are interwoven with issues in Molecular biology, Ceramide, Fragmentation and Cytolysis.
He most often published in these fields:
- Apoptosis (42.15%)
- Cell biology (35.54%)
- Mitochondrion (33.88%)
What were the highlights of his more recent work (between 2017-2021)?
- Oxidative phosphorylation (10.74%)
- Cell biology (35.54%)
- Cancer research (20.66%)
In recent papers he was focusing on the following fields of study:
Philippe Marchetti mostly deals with Oxidative phosphorylation, Cell biology, Cancer research, Mitochondrion and Cell.
His Oxidative phosphorylation study combines topics from a wide range of disciplines, such as Glycolysis, Oxidative stress and Intracellular.
His research integrates issues of Cancer and Metabolism in his study of Cell biology.
His studies deal with areas such as Tyrosine kinase and Programmed cell death as well as Cancer research.
His Mitochondrion study integrates concerns from other disciplines, such as Muscle tissue and Apoptosis.
His Apoptosis research focuses on Toxicity and how it relates to Reactive oxygen species and Inflammation.
Between 2017 and 2021, his most popular works were:
- Air pollution-derived PM2.5 impairs mitochondrial function in healthy and chronic obstructive pulmonary diseased human bronchial epithelial cells. (46 citations)
- Glucose metabolism and NRF2 coordinate the antioxidant response in melanoma resistant to MAPK inhibitors (35 citations)
- Melanoma metabolism contributes to the cellular responses to MAPK/ERK pathway inhibitors. (27 citations)
In his most recent research, the most cited papers focused on:
His primary areas of investigation include Mitochondrion, Cell biology, Oxidative phosphorylation, Apoptosis and Melanoma.
His study in Mitochondrion is interdisciplinary in nature, drawing from both Muscle tissue and Food science.
His work on Cancer expands to the thematically related Cell biology.
His Apoptosis research is multidisciplinary, incorporating elements of Cytokine secretion and Signal transduction.
His Melanoma research integrates issues from MEK inhibitor, Programmed cell death, Pentose phosphate pathway and Glutathione.
His Programmed cell death research includes elements of Targeted therapy, Intracellular and MAPK/ERK pathway.
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