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Neuroscience

D-Index
32
Citations
3937
World Ranking
9537
National Ranking
4028

Overview

Peter R. Patrylo is affiliated with Southern Illinois University School of Medicine in the United States. Their research primarily focuses on neuroscience, with specific interests spanning several subfields including cellular and molecular neuroscience, neurology, and cognitive neuroscience.

Their scholarly work covers core topics related to neuroscience and neuropharmacology research, neuroinflammation and neurodegeneration mechanisms, and memory and neural mechanisms. These topics reflect an engagement with both fundamental neuroscience processes and clinical or pathological contexts.

Peter R. Patrylo has contributed to peer-reviewed literature, notably publishing the paper titled "Altered hippocampal astroglial metabolism is associated with aging and preserved spatial learning and memory" in 2021, appearing in the journal Neurobiology of Aging.

  • Altered hippocampal astroglial metabolism is associated with aging and preserved spatial learning and memory, 2021, Neurobiology of Aging

Collaboration is evident in their frequent co-authors, which include Jeremy Ebersole, Gregory M. Rose, Tore Eid, and Kevin L. Behar. This indicates active involvement in research networks and interdisciplinary projects within their fields of study.

  • Jeremy Ebersole
  • Gregory M. Rose
  • Tore Eid
  • Kevin L. Behar

The primary publication venue for their work is Neurobiology of Aging, suggesting a focus on neuroscience topics related to aging processes.

  • Neurobiology of Aging

Best Publications

  • Recurrent spontaneous motor seizures after repeated low-dose systemic treatment with kainate: assessment of a rat model of temporal lobe epilepsy

    Jennifer L Hellier;Peter R Patrylo;Paul S Buckmaster;F.Edward Dudek

  • Uncoupling protein 2 prevents neuronal death including that occurring during seizures: a mechanism for preconditioning.

    Sabrina Diano;Russell T. Matthews;Peter Patrylo;Lichuan Yang

  • Physiological Unmasking of New Glutamatergic Pathways in the Dentate Gyrus of Hippocampal Slices From Kainate-Induced Epileptic Rats

    Peter R. Patrylo;F. Edward Dudek

  • β‐Secretase‐1 elevation in transgenic mouse models of Alzheimer’s disease is associated with synaptic/axonal pathology and amyloidogenesis: implications for neuritic plaque development

    Xue Mei Zhang;Yan Cai;Yan Cai;Kun Xiong;Huaibin Cai

  • Lateral hypothalamus: early developmental expression and response to hypocretin (orexin).

    Anthony N. Van Den Pol;Peter R. Patrylo;Prabhat K. Ghosh;Xiao-Bing Gao

  • Prolonged field bursts in the dentate gyrus: dependence on low calcium, high potassium, and nonsynaptic mechanisms.

    J. S. Schweitzer;P. R. Patrylo;F. E. Dudek

  • Doublecortin expression in adult cat and primate cerebral cortex relates to immature neurons that develop into GABAergic subgroups

    Yan Cai;Kun Xiong;Yaping Chu;Duan-Wu Luo

  • NPY inhibits glutamatergic excitation in the epileptic human dentate gyrus.

    Peter R. Patrylo;Anthony N. van den Pol;Dennis D. Spencer;Anne Williamson

  • Assessment of Inhibition and Epileptiform Activity in the Septal Dentate Gyrus of Freely Behaving Rats During the First Week After Kainate Treatment

    Jennifer L. Hellier;Peter R. Patrylo;Ping Dou;Michelle Nett

  • Is brain amyloid production a cause or a result of dementia of the Alzheimer's type?

    Robert G. Struble;Tom Ala;Peter R. Patrylo;Gregory J. Brewer

  • Physiology of human cortical neurons adjacent to cavernous malformations and tumors.

    Anne Williamson;Peter R. Patrylo;Sunghoon Lee;Dennis D. Spencer

  • Decrease in inhibition in dentate granule cells from patients with medial temporal lobe epilepsy

    Anne Williamson;Peter R. Patrylo;Dennis D. Spencer

  • Glutamate Inhibits GABA Excitatory Activity in Developing Neurons

    Anthony N. van den Pol;Anthony N. van den Pol;Xiao-Bing Gao;Peter R. Patrylo;Prabhat K. Ghosh

  • Doublecortin-expressing cells are present in layer II across the adult guinea pig cerebral cortex: Partial colocalization with mature interneuron markers

    Kun Xiong;Duan-Wu Luo;Peter R. Patrylo;Xue-Gang Luo

  • GABA Uptake and Heterotransport Are Impaired in the Dentate Gyrus of Epileptic Rats and Humans With Temporal Lobe Sclerosis

    Peter R. Patrylo;Dennis D. Spencer;Anne Williamson

  • Doublecortin-expressing cells persist in the associative cerebral cortex and amygdala in aged nonhuman primates

    Xue-Mei Zhang;Xue-Mei Zhang;Yan Cai;Yan Cai;Yaping Chu;Er-Yun Chen

  • Glucose tolerance and insulin sensitivity are impaired in APP/PS1 transgenic mice prior to amyloid plaque pathogenesis and cognitive decline

    Lauren Macklin;Chelsea M. Griffith;Yan Cai;Gregory M. Rose

  • β-Secretase-1 elevation in aged monkey and Alzheimer's disease human cerebral cortex occurs around the vasculature in partnership with multisystem axon terminal pathogenesis and β-amyloid accumulation.

    Yan Cai;Kun Xiong;Xue Mei Zhang;Xue Mei Zhang;Huaibin Cai

  • Lipolysaccharide-Induced Neuroinflammation Is Associated with Alzheimer-Like Amyloidogenic Axonal Pathology and Dendritic Degeneration in Rats

    Xiaohua Deng;Meili Li;Weiming Ai;Lixin He

  • Astrocytes and Glutamine Synthetase in Epileptogenesis.

    Tore Eid;Tore Eid;Tih‐Shih W. Lee;Peter Patrylo;Hitten P. Zaveri

  • Basic research in epilepsy and aging.

    Ilo E. Leppik;Kevin M. Kelly;Leyla deToledo-Morrell;Peter R. Patrylo

Frequent Co-Authors

Xiao-Xin Yan
Xiao-Xin Yan Central South University
Huaibin Cai
Huaibin Cai National Institutes of Health
Anne Williamson
Anne Williamson Yale University
Robert G. Struble
Robert G. Struble Southern Illinois University School of Medicine
Dennis D. Spencer
Dennis D. Spencer Yale University
F. Edward Dudek
F. Edward Dudek University of Utah
Tore Eid
Tore Eid Yale University
Yaping Chu
Yaping Chu Rush University Medical Center
Hitten P. Zaveri
Hitten P. Zaveri Yale University
Anthony N. van den Pol
Anthony N. van den Pol Yale University

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Related Online Degrees & Career Pathways

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Each of these options broadens your potential career pathways—whether it’s clinical practice, research, or community support—and complements a background in neuroscience.

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