Kevin J. Staley focuses on Neuroscience, GABAA receptor, Epilepsy, Inhibitory postsynaptic potential and Biophysics. His studies deal with areas such as Extracellular and Neurotransmission as well as Neuroscience. His biological study spans a wide range of topics, including Depolarization and Excitatory postsynaptic potential.
As a member of one scientific family, Kevin J. Staley mostly works in the field of Excitatory postsynaptic potential, focusing on Bumetanide and, on occasion, GABAergic. His Epilepsy research includes elements of Neural Inhibition and Electroencephalography. Kevin J. Staley has researched Inhibitory postsynaptic potential in several fields, including Anticonvulsant and Pharmacology.
Neuroscience, Epilepsy, Epileptogenesis, Hippocampal formation and GABAA receptor are his primary areas of study. His study brings together the fields of Neurotransmission and Neuroscience. His Epilepsy research incorporates elements of Anesthesia, Pharmacology and Electroencephalography.
His Epileptogenesis research integrates issues from Chronic epilepsy, Kainate receptor and Time course. His research integrates issues of Reversal potential, Biophysics, Depolarization, Inhibitory postsynaptic potential and Anticonvulsant in his study of GABAA receptor. His work investigates the relationship between Inhibitory postsynaptic potential and topics such as Bumetanide that intersect with problems in Endocrinology, Internal medicine and Furosemide.
Kevin J. Staley mainly investigates Neuroscience, Epilepsy, GABAA receptor, Hippocampal formation and Electroencephalography. His work carried out in the field of Neuroscience brings together such families of science as Glutamatergic, Pathophysiology and Cognitive impairment. His study in Epilepsy is interdisciplinary in nature, drawing from both Biomarker, Kainate receptor, Odds ratio and Optogenetics.
The various areas that Kevin J. Staley examines in his GABAA receptor study include Cotransporter, Reversal potential, Biophysics and GABAergic, Inhibitory postsynaptic potential. His work deals with themes such as Ictal and Hippocampus, which intersect with Hippocampal formation. The concepts of his Anticonvulsant study are interwoven with issues in Bumetanide and Phenobarbital.
His scientific interests lie mostly in Epilepsy, Electroencephalography, Neuroscience, Hippocampal formation and Pharmacology. His Epilepsy study incorporates themes from Hematoma, Pediatrics and Risk factor. His Electroencephalography research is multidisciplinary, incorporating perspectives in Biomarker, Confidence interval, Audiology and Scalp.
His study in the field of GABAergic also crosses realms of Gene dosage. His work on Epileptogenesis as part of general Hippocampal formation study is frequently connected to Neurotoxicity, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them. His studies in Pharmacology integrate themes in fields like Cotransporter, Transporter, Edema and GABAA receptor.
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NKCC1 transporter facilitates seizures in the developing brain
Volodymyr I Dzhala;Delia M Talos;Dan A Sdrulla;Audrey C Brumback.
Nature Medicine (2005)
Ionic mechanisms of neuronal excitation by inhibitory GABAA receptors
Kevin J. Staley;Brandi L. Soldo;William R. Proctor.
Science (1995)
Roles of the cation–chloride cotransporters in neurological disease
Kristopher T Kahle;Kevin J Staley;Brian V Nahed;Gerardo Gamba.
Nature Reviews Neurology (2008)
Shunting of excitatory input to dentate gyrus granule cells by a depolarizing GABAA receptor-mediated postsynaptic conductance.
K. J. Staley;I. Mody.
Journal of Neurophysiology (1992)
Membrane properties of dentate gyrus granule cells: comparison of sharp microelectrode and whole-cell recordings
K. J. Staley;T. S. Otis;I. Mody.
Journal of Neurophysiology (1992)
Development of Spontaneous Recurrent Seizures after Kainate-Induced Status Epilepticus
P. A. Williams;A. M. White;S. Clark;D. J. Ferraro.
The Journal of Neuroscience (2009)
Modulation of mammalian dendritic GABAA receptor function by the kinetics of Cl− and HCO3− transport
Kevin J. Staley;William R. Proctor;William R. Proctor.
The Journal of Physiology (1999)
Bumetanide enhances phenobarbital efficacy in a neonatal seizure model.
Volodymyr I. Dzhala;Audrey C. Brumback;Kevin J. Staley;Kevin J. Staley.
Annals of Neurology (2008)
Molecular mechanisms of ischemic cerebral edema: role of electroneutral ion transport.
Kristopher T. Kahle;J. Marc Simard;Kevin J. Staley;Brian V. Nahed.
Physiology (2009)
Alteration of GABAA Receptor Function Following Gene Transfer of the CLC-2 Chloride Channel
Kevin Staley;Roderic Smith;Jerome Schaack;Christine Wilcox.
Neuron (1996)
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