World's Best Scientists 2026 revealed!
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Biology and Biochemistry
UAE
2026

D-Index & Metrics

Biology and Biochemistry

D-Index
55
Citations
18332
World Ranking
14783
National Ranking
3

Research.com Recognitions

  • 2026 - Research.com Biology and Biochemistry in United Arab Emirates Leader Award
  • 2025 - Research.com Biology and Biochemistry in United Arab Emirates Leader Award

Overview

Mohamed Rahmani is affiliated with Khalifa University in the United Arab Emirates. Their research spans multiple areas within medicine and biochemistry, genetics, and molecular biology, with a notable focus on molecular biology and oncology. They have contributed extensively to fields such as infectious diseases, cancer research, and hematology.

Their work covers several main topics, including:

  • Protein Degradation and Inhibitors
  • COVID-19 Clinical Research Studies
  • Cancer, Hypoxia, and Metabolism
  • Cancer Cells and Metastasis
  • HER2/EGFR in Cancer Research
  • Cell death mechanisms and regulation
  • RNA Interference and Gene Delivery

Rahmani's research output includes publications in the following venues:

  • Frontiers in Immunology
  • Cancers
  • Clinical Cancer Research
  • Scientific Reports
  • Journal of Molecular Medicine

Frequent co-authors collaborating with Rahmani include:

  • Rifat Hamoudi
  • Sarah Hammoudeh
  • Thenmozhi Venkatachalam
  • Arabella Musa Hammoudeh
  • Bassam Mahboub

Notable recent papers authored by Rahmani present a range of research foci and were published between 2021 and 2024:

  • "Targeting BCL-2 in Cancer: Advances, Challenges, and Perspectives," 2021, Cancers
  • "Systems Immunology Analysis Reveals the Contribution of Pulmonary and Extrapulmonary Tissues to the Immunopathogenesis of Severe COVID-19 Patients," 2021, Frontiers in Immunology
  • "Insight into molecular mechanisms underlying hepatic dysfunction in severe COVID-19 patients using systems biology," 2021, World Journal of Gastroenterology
  • "Dual mTORC1/2 Inhibition Synergistically Enhances AML Cell Death in Combination with the BCL2 Antagonist Venetoclax," 2023, Clinical Cancer Research
  • "1H-NMR metabolomics analysis identifies hypoxanthine as a novel metastasis-associated metabolite in breast cancer," 2024, Scientific Reports

Rahmani's scientific contributions cover critical areas including cell death mechanisms and regulation, as well as cancer metabolism and the immunopathogenesis of infectious diseases such as COVID-19. Their interdisciplinary approach integrates molecular biology techniques with clinical research studies, reflecting a broad engagement with both fundamental and translational research questions.

Best Publications

  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

    Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin

  • Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

    Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin

  • Apoptosis Induced by the Kinase Inhibitor BAY 43-9006 in Human Leukemia Cells Involves Down-regulation of Mcl-1 through Inhibition of Translation

    Mohamed Rahmani;Eric Maynard Davis;Cheryl Bauer;Paul Dent

  • The proteasome inhibitor bortezomib interacts synergistically with histone deacetylase inhibitors to induce apoptosis in Bcr/Abl+ cells sensitive and resistant to STI571.

    Chunrong Yu;Mohamed Rahmani;Daniel Conrad;Mark Subler

  • Blockade of Histone Deacetylase Inhibitor-Induced RelA/p65 Acetylation and NF-κB Activation Potentiates Apoptosis in Leukemia Cells through a Process Mediated by Oxidative Damage, XIAP Downregulation, and c-Jun N-Terminal Kinase 1 Activation

    Yun Dai;Mohamed Rahmani;Paul Dent;Steven Grant

  • A selective sphingosine kinase 1 inhibitor integrates multiple molecular therapeutic targets in human leukemia

    Steven W. Paugh;Barbara S. Paugh;Mohamed Rahmani;Dmitri Kapitonov

  • A Bcr/Abl-independent, Lyn-dependent Form of Imatinib Mesylate (STI-571) Resistance Is Associated with Altered Expression of Bcl-2

    Yun Dai;Mohamed Rahmani;Seth J. Corey;Paul Dent

  • Coadministration of histone deacetylase inhibitors and perifosine synergistically induces apoptosis in human leukemia cells through Akt and ERK1/2 inactivation and the generation of ceramide and reactive oxygen species.

    Mohamed Rahmani;Erin Reese;Yun Dai;Cheryl Bauer

  • Pharmacologic mitogen-activated protein/extracellular signal-regulated kinase kinase/mitogen-activated protein kinase inhibitors interact synergistically with STI571 to induce apoptosis in Bcr/Abl-expressing human leukemia cells.

    Chunrong Yu;Geoffrey Krystal;Lyuba Varticovksi;Robert McKinstry

  • The kinase inhibitor sorafenib induces cell death through a process involving induction of endoplasmic reticulum stress.

    Mohamed Rahmani;Eric Maynard Davis;Timothy Ryan Crabtree;Joseph Reza Habibi

  • Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

    Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin

  • Targeting Mcl-1 for the therapy of cancer

    Bridget A Quinn;Rupesh Dash;Belal Azab;Siddik Sarkar

  • Coadministration of the heat shock protein 90 antagonist 17-allylamino- 17-demethoxygeldanamycin with suberoylanilide hydroxamic acid or sodium butyrate synergistically induces apoptosis in human leukemia cells.

    Mohamed Rahmani;Chunrong Yu;Yun Dai;Erin Reese

  • Histone Deacetylase Inhibitors Promote STI571-mediated Apoptosis in STI571-sensitive and -resistant Bcr/Abl+ Human Myeloid Leukemia Cells

    Chunrong Yu;Mohamed Rahmani;Jorge Almenara;Mark Subler

  • Vorinostat and sorafenib increase ER stress, autophagy and apoptosis via ceramide-dependent CD95 and PERK activation

    Margaret A. Park;Guo Zhang;Aditi Pandya Martin;Hossein Hamed

  • The hierarchical relationship between MAPK signaling and ROS generation in human leukemia cells undergoing apoptosis in response to the proteasome inhibitor Bortezomib

    Chunrong Yu;Mohamed Rahmani;Paul Dent;Steven Grant

  • Proteasome inhibitors potentiate leukemic cell apoptosis induced by the cyclin-dependent kinase inhibitor flavopiridol through a SAPK/JNK- and NF-κB-dependent process

    Yun Dai;Mohamed Rahmani;Steven Grant

  • The Histone Deacetylase Inhibitor MS-275 Interacts Synergistically with Fludarabine to Induce Apoptosis in Human Leukemia Cells

    Sonia C. Maggio;Roberto R. Rosato;Lora B. Kramer;Yun Dai

  • Bortezomib and flavopiridol interact synergistically to induce apoptosis in chronic myeloid leukemia cells resistant to imatinib mesylate through both Bcr/Abl-dependent and -independent mechanisms

    Yun Dai;Mohamed Rahmani;Xin-Yan Pei;Paul Dent

  • Dual Inhibition of Bcl-2 and Bcl-xL Strikingly Enhances PI3K Inhibition-Induced Apoptosis in Human Myeloid Leukemia Cells through a GSK3- and Bim-Dependent Mechanism

    Mohamed Rahmani;Mandy Mayo Aust;Elisa Attkisson;David C. Williams

Frequent Co-Authors

Steven Grant
Steven Grant Virginia Commonwealth University
Paul Dent
Paul Dent Virginia Commonwealth University
Paul B. Fisher
Paul B. Fisher Virginia Commonwealth University
Rifat Hamoudi
Rifat Hamoudi University of Sharjah
Qutayba Hamid
Qutayba Hamid University of Sharjah
David T. Curiel
David T. Curiel Washington University in St. Louis
Evelina Gatti
Evelina Gatti Aix-Marseille University
Devanand Sarkar
Devanand Sarkar Virginia Commonwealth University
Sujit K. Bhutia
Sujit K. Bhutia National Institute of Technology Rourkela
Beth Levine
Beth Levine The University of Texas Southwestern Medical Center

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