What is he best known for?
The fields of study he is best known for:
His scientific interests lie mostly in Cell biology, Biochemistry, Phosphatidylinositol, Endocytic cycle and Lipidomics.
His Cell biology study integrates concerns from other disciplines, such as Autophagy, Synaptic vesicle, Phosphatidic acid and Clathrin.
His studies in Cell membrane, Mutant, Lipid metabolism, Fatty acid and Enzyme are all subfields of Biochemistry research.
The concepts of his Phosphatidylinositol study are interwoven with issues in Cell cortex and Endosome.
His biological study spans a wide range of topics, including GTPase, Phosphatidylinositol 3-Kinases, Amphiphysin, Small GTPase and Membrane tubulation.
His studies deal with areas such as Targeted metabolomics, Computational biology and Chromatography as well as Lipidomics.
His most cited work include:
- The emerging field of lipidomics (913 citations)
- Dual Role of 3-Methyladenine in Modulation of Autophagy via Different Temporal Patterns of Inhibition on Class I and III Phosphoinositide 3-Kinase (716 citations)
- Essential Role of Phosphoinositide Metabolism in Synaptic Vesicle Recycling (693 citations)
What are the main themes of his work throughout his whole career to date?
Markus R. Wenk mainly investigates Biochemistry, Cell biology, Lipidomics, Lipid metabolism and Internal medicine.
His study in Phosphatidic acid, Lipidome, Endoplasmic reticulum, Enzyme and Cell membrane are all subfields of Biochemistry.
Markus R. Wenk interconnects Endocytic cycle and Endocytosis in the investigation of issues within Cell biology.
His Lipidomics research includes themes of Sphingolipid, Computational biology and Chromatography, Mass spectrometry.
The Sphingolipid study combines topics in areas such as Ceramide and Sphingomyelin.
His Endocrinology research extends to Internal medicine, which is thematically connected.
He most often published in these fields:
- Biochemistry (37.39%)
- Cell biology (30.09%)
- Lipidomics (22.19%)
What were the highlights of his more recent work (between 2018-2021)?
- Lipidomics (22.19%)
- Internal medicine (15.50%)
- Cell biology (30.09%)
In recent papers he was focusing on the following fields of study:
Markus R. Wenk mostly deals with Lipidomics, Internal medicine, Cell biology, Sphingolipid and Lipid metabolism.
His research in Lipidomics intersects with topics in Ceramide, Computational biology and Chromatography, Mass spectrometry.
His studies in Internal medicine integrate themes in fields like Explant culture, Docosahexaenoic acid, Endocrinology and Fetus.
His Cell biology research is multidisciplinary, relying on both Amyloid beta, Caenorhabditis elegans, Transgene, Beta and Innate immune system.
The study incorporates disciplines such as Biophysics, Membrane, Sphingomyelin and Cytoskeleton in addition to Sphingolipid.
His Lipid metabolism research incorporates themes from Phagolysosome, Pathogen, Microbiology and Hepatocyte.
Between 2018 and 2021, his most popular works were:
- Lipidomics needs more standardization (40 citations)
- Movement of accessible plasma membrane cholesterol by the GRAMD1 lipid transfer protein complex (31 citations)
- Large-scale lipidomics identifies associations between plasma sphingolipids and T2DM incidence. (30 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Internal medicine
Lipidomics, Internal medicine, Ceramide, Endocrinology and Cell biology are his primary areas of study.
His Lipidomics research integrates issues from In silico, Computational biology and Data science.
Markus R. Wenk has included themes like Diabetes mellitus, Oxidative phosphorylation and Lipoxygenase in his Internal medicine study.
Markus R. Wenk studied Ceramide and Sphingolipid that intersect with Sphingomyelin, Cell, Bacterial outer membrane and Cytoskeleton.
His study in Endocrinology is interdisciplinary in nature, drawing from both Lipid signaling, Meibomian gland, Eyelid, Tears and Meibomian gland dysfunction.
His Cell biology study incorporates themes from Blood–brain barrier and Single-cell analysis.
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