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Biology and Biochemistry

D-Index
69
Citations
38806
World Ranking
7210
National Ranking
183

Overview

Kate Schroder is affiliated with the University of Queensland in Australia. Their research spans multiple disciplines, focusing primarily on Biochemistry, Genetics and Molecular Biology, Medicine, and Immunology and Microbiology. A significant portion of their work delves into Molecular Biology, Immunology, Infectious Diseases, Neurology, and Physiology.

The main topics covered by Kate Schroder's research include:

  • Inflammasome and immune disorders
  • Immune Response and Inflammation
  • Heme Oxygenase-1 and Carbon Monoxide
  • Immune cells in cancer
  • Interferon and immune responses
  • COVID-19 Clinical Research Studies
  • SARS-CoV-2 and COVID-19 Research

Kate Schroder has contributed regularly to various academic venues. Their frequent publication outlets include:

  • bioRxiv (Cold Spring Harbor Laboratory)
  • Nature reviews. Immunology
  • Immunology and Cell Biology
  • Clinical & Translational Immunology
  • Cell Reports

Recent notable papers authored or co-authored by Kate Schroder comprise:

  • "NLRP3 and pyroptosis blockers for treating inflammatory diseases," 2022, Trends in Pharmacological Sciences
  • "Neutrophil-Derived S100A8/A9 Amplify Granulopoiesis After Myocardial Infarction," 2020, Circulation
  • "The NLRP3 inflammasome triggers sterile neuroinflammation and Alzheimer's disease," 2020, Current Opinion in Immunology
  • "Endothelial cells are not productively infected by SARS-CoV-2," 2021, Clinical & Translational Immunology
  • "Inflammatory Caspases: Toward a Unified Model for Caspase Activation by Inflammasomes," 2022, Annual Review of Immunology

Kate Schroder frequently collaborates with other researchers. Their prominent co-authors include:

  • Sabrina Sofia Burgener
  • Caroline L. Holley
  • Grace Lawrence
  • Stefan Emming
  • Rebecca C. Coll

Best Publications

  • Interferon-γ: an overview of signals, mechanisms and functions

    Kate Schroder;Paul John Hertzog;Timothy Ravasi;David A Hume

  • A small-molecule inhibitor of the NLRP3 inflammasome for the treatment of inflammatory diseases

    Rebecca C Coll;Avril A B Robertson;Jae Jin Chae;Sarah C Higgins

  • NLRP3 inflammasome activation: the convergence of multiple signalling pathways on ROS production?

    Jurg Tschopp;Kate Schroder;Kate Schroder

  • The NLRP3 Inflammasome: A Sensor for Metabolic Danger?

    Kate Schroder;Rongbin Zhou;Jurg Tschopp

  • NLRP3 inflammasome blockade reduces liver inflammation and fibrosis in experimental NASH in mice.

    Auvro R. Mridha;Alexander Wree;Alexander Wree;Avril A.B. Robertson;Matthew M. Yeh

  • MCC950 directly targets the NLRP3 ATP-hydrolysis motif for inflammasome inhibition

    Rebecca C. Coll;James R. Hill;Christopher J. Day;Alina Zamoshnikova

  • The regulated retrotransposon transcriptome of mammalian cells.

    Geoffrey J Faulkner;Yasumasa Kimura;Carsten O Daub;Shivangi Wani

  • Inflammasome inhibition prevents α-synuclein pathology and dopaminergic neurodegeneration in mice

    Richard Gordon;Eduardo A. Albornoz;Daniel C. Christie;Monica R. Langley

  • Osteal tissue macrophages are intercalated throughout human and mouse bone lining tissues and regulate osteoblast function in vitro and in vivo.

    Ming K. Chang;Liza-Jane Raggatt;Kylie A. Alexander;Julia S. Kuliwaba

  • Noncanonical inflammasome signaling elicits gasdermin D–dependent neutrophil extracellular traps

    Kaiwen W. Chen;Kaiwen W. Chen;Mercedes Monteleone;Dave Boucher;Gabriel Sollberger

  • AIM2 and NLRP3 inflammasomes activate both apoptotic and pyroptotic death pathways via ASC

    V Sagulenko;S J Thygesen;D P Sester;A Idris

  • The macrophage-inducible C-type lectin, Mincle, is an essential component of the innate immune response to Candida albicans

    Christine A Wells;Judith A Salvage-Jones;Xin Li;Kelly Hitchens

  • Expression analysis of G Protein-Coupled Receptors in mouse macrophages.

    Jane E Lattin;Kate Schroder;Andrew I Su;John R Walker

  • The transcriptional network that controls growth arrest and differentiation in a human myeloid leukemia cell line

    Harukazu Suzuki;Alistair R.R. Forrest;Erik Van Nimwegen;Carsten O. Daub

  • Caspase-1 self-cleavage is an intrinsic mechanism to terminate inflammasome activity

    Dave Boucher;Mercedes Monteleone;Rebecca C. Coll;Kaiwen W. Chen

  • Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic manner.

    Stephanie A. Conos;Stephanie A. Conos;Kaiwen W Chen;Dominic De Nardo;Dominic De Nardo;Hideki Hara

  • K+ Efflux-Independent NLRP3 Inflammasome Activation by Small Molecules Targeting Mitochondria

    Christina J. Groß;Ritu Mishra;Katharina S. Schneider;Guillaume Médard

  • The Neutrophil NLRC4 Inflammasome Selectively Promotes IL-1β Maturation without Pyroptosis during Acute Salmonella Challenge

    Kaiwen W. Chen;Christina J. Groß;Flor Vásquez Sotomayor;Katryn J. Stacey

  • Tiny RNAs associated with transcription start sites in animals.

    Ryan J Taft;Evgeny A Glazov;Nicole Cloonan;Cas Simons

  • Differential Expression of NLRP3 among Hematopoietic Cells

    Greta Guarda;Manuel Zenger;Manuel Zenger;Amir S. Yazdi;Kate Schroder;Kate Schroder

Frequent Co-Authors

Matthew J. Sweet
Matthew J. Sweet University of Queensland
David A. Hume
David A. Hume University of Queensland
Mark E. Cooper
Mark E. Cooper Monash University
Katryn J. Stacey
Katryn J. Stacey University of Queensland
Seth L. Masters
Seth L. Masters Hudson Institute of Medical Research
Luke A. J. O'Neill
Luke A. J. O'Neill Trinity College Dublin
Jennifer L. Stow
Jennifer L. Stow University of Queensland

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