Internal medicine, Endocrinology, Potassium channel, Biochemistry and KvLQT1 are his primary areas of study. His studies deal with areas such as Medical literature and Cardiology as well as Internal medicine. Endocrinology and Familial atrial fibrillation are frequently intertwined in his study.
His Potassium channel study combines topics from a wide range of disciplines, such as Voltage-gated ion channel and KCNE3. His KCNE3 study combines topics in areas such as Molecular biology, Secretion and KCNE2. Within one scientific family, Jacques Barhanin focuses on topics pertaining to Romano–Ward syndrome under KvLQT1, and may sometimes address concerns connected to Jervell and Lange-Nielsen syndrome, KCNQ4 and Hearing loss.
Jacques Barhanin mainly focuses on Internal medicine, Endocrinology, Potassium channel, Biophysics and Cell biology. His Internal medicine study integrates concerns from other disciplines, such as Mutation and Genetics. His research combines Long QT syndrome and Endocrinology.
His Potassium channel research is multidisciplinary, relying on both Secretion, Molecular biology, Knockout mouse, KCNE3 and Patch clamp. His studies in Biophysics integrate themes in fields like Receptor, Membrane, Biochemistry and Tityus serrulatus. Skeletal muscle is closely connected to Protein subunit in his research, which is encompassed under the umbrella topic of Cell biology.
Jacques Barhanin mainly focuses on Endocrinology, Internal medicine, Potassium channel, Cell biology and Aldosterone. Jacques Barhanin studied Endocrinology and Phenotype that intersect with KvLQT1 and Ventricular tachycardia. His Internal medicine study incorporates themes from Protein subunit and Pathology.
His Potassium channel research incorporates elements of Extracellular, Secretion, Intracellular, Depolarization and Patch clamp. The various areas that Jacques Barhanin examines in his Cell biology study include Gating, Membrane potential, Ion channel and Endolymph. His Aldosterone study incorporates themes from Renin–angiotensin system, Aldosterone synthase and Pharmacology.
His main research concerns Potassium channel, Internal medicine, Endocrinology, Secretion and Biophysics. His Potassium channel research is multidisciplinary, incorporating perspectives in Gating, Intracellular, Endosome, Cell biology and Patch clamp. His study in Cell biology is interdisciplinary in nature, drawing from both KCNE3 and Skeletal muscle.
His research on Endocrinology frequently connects to adjacent areas such as Anatomy. His research investigates the connection between Secretion and topics such as Protein subunit that intersect with problems in Halothane, Neuropeptide, Orexin and Ruthenium red. The study incorporates disciplines such as Extracellular, Biochemistry, Respiratory system and Brainstem in addition to Biophysics.
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K v LQT1 and IsK (minK) proteins associate to form the I KS cardiac potassium current
Jacques Barhanin;Florian Lesage;Eric Guillemare;Michel Fink.
KCNQ1 gain-of-function mutation in familial atrial fibrillation
Yi-Han Chen;Shi-Jie Xu;Saı̈d Bendahhou;Xiao-Liang Wang.
A novel mutation in the potassium channel gene KVLQT1 causes the Jervell and Lange-Nielsen cardioauditory syndrome
Nathalie Neyroud;Frédérique Tesson;Isabelle Denjoy;Michel Leibovici.
Nature Genetics (1997)
TWIK-1, a ubiquitous human weakly inward rectifying K+ channel with a novel structure.
F. Lesage;E. Guillemare;M. Fink;F. Duprat.
The EMBO Journal (1996)
Identification of a KCNE2 Gain-of-Function Mutation in Patients with Familial Atrial Fibrillation
Yiqing Yang;Min Xia;Qingfeng Jin;Saïd Bendahhou.
American Journal of Human Genetics (2004)
Inner Ear Defects Induced by Null Mutation of the isk Gene
Douglas E Vetter;Jeffrey R Mann;Philine Wangemann;Jianzhong Liu.
Genetic and Molecular Basis of Cardiac Arrhythmias: Impact on Clinical Management Parts I and II
Silvia G. Priori;Jacques Barhanin;Richard N. W. Hauer;Wilhelm Haverkamp.
Molecular mechanism of action of the vasoconstrictor peptide endothelin.
Catherine Van Renterghem;Paul Vigne;Jacques Barhanin;Annie Schmid-Alliana.
Biochemical and Biophysical Research Communications (1988)
A Kir2.1 gain-of-function mutation underlies familial atrial fibrillation.
Min Xia;Qingfeng Jin;Saïd Bendahhou;Yusong He.
Biochemical and Biophysical Research Communications (2005)
Cloning and expression of a membrane receptor for secretory phospholipases A2.
G Lambeau;P Ancian;J Barhanin;M Lazdunski.
Journal of Biological Chemistry (1994)
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