Jacek Hawiger focuses on Molecular biology, Transcription factor, Biochemistry, Platelet and Nuclear localization sequence. His studies in Transcription factor integrate themes in fields like Chemokine, Jurkat cells and Cell biology. The concepts of his Biochemistry study are interwoven with issues in Biophysics and Thrombin.
His study on Platelet is mostly dedicated to connecting different topics, such as Prostacyclin. Jacek Hawiger has included themes like Nuclear transport, Intracellular, Peptide and Signal peptide in his Nuclear localization sequence study. As a part of the same scientific family, Jacek Hawiger mostly works in the field of Intracellular, focusing on Signal transduction and, on occasion, Immunology.
His main research concerns Biochemistry, Platelet, Fibrinogen, Cell biology and Immunology. His work carried out in the field of Biochemistry brings together such families of science as Biophysics, Thrombin and Fibrin. His Platelet study combines topics from a wide range of disciplines, such as Blood proteins, Receptor and Glycoprotein.
Jacek Hawiger has researched Fibrinogen in several fields, including Platelet activation, Endocrinology, Plasmin and Von Willebrand factor. His Cell biology research is multidisciplinary, incorporating perspectives in Proinflammatory cytokine, Transcription factor and Receptor complex. Jacek Hawiger combines subjects such as Molecular biology, Nuclear localization sequence, Nuclear transport, Importin and Signal peptide with his study of Transcription factor.
Proinflammatory cytokine, Cell biology, Inflammation, Immunology and Chemokine are his primary areas of study. His Proinflammatory cytokine study incorporates themes from Cytokine, Transcription factor, Molecular biology, GRB2 and Nuclear transport. His biological study spans a wide range of topics, including Karyopherin, Karyopherins, Endocrinology, Nuclear localization sequence and Importin.
His CARD Signaling Adaptor Proteins research extends to Cell biology, which is thematically connected. His Chemokine research includes themes of Endothelium, p38 mitogen-activated protein kinases, Intracellular and Monocyte. His Intracellular study integrates concerns from other disciplines, such as Phosphorylation and STAT1.
Jacek Hawiger spends much of his time researching Immunology, Inflammation, Chemokine, Acquired immune system and Proinflammatory cytokine. His Inflammation research includes elements of Acute kidney injury, Microcirculation and Septic shock. His study ties his expertise on Cell biology together with the subject of Chemokine.
His research integrates issues of Receptor and Endothelium in his study of Cell biology. His Proinflammatory cytokine research incorporates elements of Cytokine, Signal transduction, STAT1 and Intracellular. His Intracellular research incorporates themes from Superantigen, Pneumonia, ARDS, Lung and Phosphorylation.
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Inhibition of Nuclear Translocation of Transcription Factor NF-κB by a Synthetic Peptide Containing a Cell Membrane-permeable Motif and Nuclear Localization Sequence
Yao-Zhong Lin;SongYi Yao;Ruth Ann Veach;Troy R. Torgerson.
Journal of Biological Chemistry (1995)
Endogenous biosynthesis of prostacyclin and thromboxane and platelet function during chronic administration of aspirin in man.
G A FitzGerald;J A Oates;J Hawiger;R L Maas.
Journal of Clinical Investigation (1983)
Platelet receptor recognition site on human fibrinogen. Synthesis and structure-function relationship of peptides corresponding to the carboxy-terminal segment of the gamma chain.
Marek Kloczewiak;Sheila Timmons;Thomas J. Lukas;Jacek Hawiger.
Measurement of fibrinogen and fibrin degradation products in serum by staphylococcal clumping test
J Hawiger;S Niewiarowski;V Gurewich;D P Thomas.
Journal of Laboratory and Clinical Medicine (1970)
Intracellular protein therapy with SOCS3 inhibits inflammation and apoptosis.
Daewoong Jo;Danya Liu;Shan Yao;Robert D Collins.
Nature Medicine (2005)
Fibrinogen-independent platelet adhesion and thrombus formation on subendothelium mediated by glycoprotein IIb-IIIa complex at high shear rate.
H J Weiss;J Hawiger;Z M Ruggeri;V T Turitto.
Journal of Clinical Investigation (1989)
Factor IX is activated in vivo by the tissue factor mechanism.
KA Bauer;BL Kass;H ten Cate;JJ Hawiger.
Innate immunity and inflammation: a transcriptional paradigm.
Immunologic Research (2001)
Lipopolysaccharide induces phosphorylation of MAD3 and activation of c-Rel and related NF-kappa B proteins in human monocytic THP-1 cells.
S R Cordle;R Donald;M A Read;J Hawiger.
Journal of Biological Chemistry (1993)
Thrombin-induced exposure and prostacyclin inhibition of the receptor for factor VIII/von Willebrand factor on human platelets.
T Fujimoto;S Ohara;J Hawiger.
Journal of Clinical Investigation (1982)
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