1993 - Fellow of the American Association for the Advancement of Science (AAAS)
His primary areas of investigation include Biochemistry, Cancer research, Cell growth, Molecular biology and Carcinogenesis. I. Bernard Weinstein combines subjects such as Cancer, Internal medicine, Endocrinology and Cyclin D with his study of Cancer research. His biological study spans a wide range of topics, including Apoptosis, Receptor tyrosine kinase, Cell culture and Cell biology.
His Molecular biology research includes themes of Embryo, Promoter, Gene, Long terminal repeat and In vivo. His work deals with themes such as Epidermal growth factor, Protein kinase C and Carcinogen, which intersect with Carcinogenesis. The study incorporates disciplines such as Cancer cell, Cell cycle checkpoint and Cyclin D1 in addition to Growth inhibition.
I. Bernard Weinstein mainly focuses on Biochemistry, Molecular biology, Cancer research, Cell culture and Cell biology. His studies deal with areas such as In vitro, Gene, Transfection and Phorbol, Protein kinase C as well as Molecular biology. I. Bernard Weinstein interconnects Endocrinology, Cell growth, Apoptosis, Internal medicine and Cyclin D1 in the investigation of issues within Cancer research.
Phosphorylation is closely connected to Kinase in his research, which is encompassed under the umbrella topic of Apoptosis. His Cell culture research includes elements of Cell and Transformation. His Cell biology study combines topics from a wide range of disciplines, such as Carcinogenesis and Epidermal growth factor.
His scientific interests lie mostly in Cancer research, Cyclin D1, Cell biology, Apoptosis and Cancer cell. His Cancer research research is multidisciplinary, incorporating perspectives in Endocrinology, Growth inhibition, Cell growth, Internal medicine and Cell cycle. His Cyclin D1 study combines topics in areas such as Cyclin-dependent kinase and Colorectal cancer.
In his work, Activator is strongly intertwined with Molecular biology, which is a subfield of Cell biology. His studies in Apoptosis integrate themes in fields like Carcinogenesis and Cell culture. His Signal transduction study results in a more complete grasp of Biochemistry.
Cell growth, Cancer research, Growth inhibition, Biochemistry and Cyclin D1 are his primary areas of study. His research investigates the connection with Cell growth and areas like Receptor tyrosine kinase which intersect with concerns in Cell culture and Biological activity. The Cancer research study combines topics in areas such as Endocrinology, Receptor, Internal medicine, Prostate cancer and MAPK/ERK pathway.
Biochemistry and Myricitrin are two areas of study in which I. Bernard Weinstein engages in interdisciplinary research. His research in Cyclin D1 intersects with topics in Cancer cell and Cyclin-dependent kinase. I. Bernard Weinstein works mostly in the field of Cyclin-dependent kinase, limiting it down to topics relating to Cyclin E and, in certain cases, Molecular biology, as a part of the same area of interest.
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Addiction to Oncogenes—the Achilles Heal of Cancer
I. Bernard Weinstein.
Mechanisms of disease: Oncogene addiction--a rationale for molecular targeting in cancer therapy
I Bernard Weinstein;Andrew K Joe.
Nature Reviews Clinical Oncology (2006)
Overproduction of protein kinase C causesdisordered growth control in rat fibroblasts
Gerard M. Housey;Mark D. Johnson;W.L. Wendy Hsiao;Catherine A. O'Brian.
Resveratrol induces growth inhibition, S-phase arrest, apoptosis, and changes in biomarker expression in several human cancer cell lines.
Andrew K Joe;Hui Liu;Masumi Suzui;Muhammet E Vural.
Clinical Cancer Research (2002)
Amplification and expression of the human cyclin D gene in esophageal cancer.
Wei Jiang;Scott M. Kahn;Naohiro Tomita;Yu-Jing Zhang.
Cancer Research (1992)
Constitutive activation of signal transducers and activators of transcription 3 correlates with cyclin D1 overexpression and may provide a novel prognostic marker in head and neck squamous cell carcinoma.
Muneyuki Masuda;Masumi Suzui;Ryuji Yasumatu;Torahiko Nakashima.
Cancer Research (2002)
(−)-Epigallocatechin Gallate and Polyphenon E Inhibit Growth and Activation of the Epidermal Growth Factor Receptor and Human Epidermal Growth Factor Receptor-2 Signaling Pathways in Human Colon Cancer Cells
Masahito Shimizu;Atsuko Deguchi;Jin T.E. Lim;Hisataka Moriwaki.
Clinical Cancer Research (2005)
Molecular epidemiology and carcinogen-DNA adduct detection: New approaches to studies of human cancer causation
Frederica P. Perera;I.Bernard Weinstein.
Journal of Chronic Diseases (1982)
Multiple functions of p27Kip1 and its alterations in tumor cells: a review
Alessandro Sgambato;Achille Cittadini;Beatrice Faraglia;I. Bernard Weinstein.
Journal of Cellular Physiology (2000)
Tumour promotor induces plasminogen activator
Michael Wigler;I. Bernard Weinstein.
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