His primary scientific interests are in Biochemistry, Molecular biology, Okadaic acid, Ornithine decarboxylase and Carcinogenesis. As part of his studies on Biochemistry, he frequently links adjacent subjects like Pharmacology. Hirota Fujiki has included themes like Heterogeneous nuclear ribonucleoprotein, Protein kinase C, MAP2K7 and Virology in his Molecular biology study.
His study in Okadaic acid is interdisciplinary in nature, drawing from both Toxin and DMBA. His Ornithine decarboxylase research is multidisciplinary, incorporating elements of In vitro, Structure–activity relationship, Enzyme inducer, Carcinogen and Marine toxin. His biological study focuses on Tumor promotion.
Hirota Fujiki focuses on Biochemistry, Molecular biology, Okadaic acid, Carcinogenesis and Internal medicine. His study in Protein kinase C, Receptor, Ornithine decarboxylase, In vitro and Aplysiatoxin is done as part of Biochemistry. His Molecular biology research incorporates themes from Cell, Messenger RNA, Gene expression, Gene and Tissue culture.
As a part of the same scientific family, Hirota Fujiki mostly works in the field of Carcinogenesis, focusing on Pharmacology and, on occasion, Apoptosis. As part of the same scientific family, Hirota Fujiki usually focuses on Internal medicine, concentrating on Endocrinology and intersecting with Thapsigargin and 12-O-Tetradecanoylphorbol-13-acetate. As a member of one scientific family, he mostly works in the field of Tumor promotion, focusing on Cancer research and, on occasion, Epigallocatechin gallate, Immunology and Tumor necrosis factor alpha.
His main research concerns Cancer, Internal medicine, Tumor necrosis factor alpha, Carcinogenesis and Cancer research. The various areas that Hirota Fujiki examines in his Cancer study include Lung cancer and Pharmacology. His research investigates the connection between Internal medicine and topics such as Endocrinology that intersect with problems in Oncogene.
His work deals with themes such as Tumor promotion and Biochemistry, which intersect with Tumor necrosis factor alpha. His Tumor promotion study combines topics from a wide range of disciplines, such as Proinflammatory cytokine, Okadaic acid and Traditional medicine. His studies deal with areas such as Signal transduction and DMBA as well as Okadaic acid.
Hirota Fujiki mainly focuses on Cancer, Internal medicine, Anticarcinogen, Tumor promotion and Cancer prevention. His work on Breast cancer as part of general Cancer study is frequently connected to Green tea extract, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them. As a part of the same scientific study, Hirota Fujiki usually deals with the Internal medicine, concentrating on Endocrinology and frequently concerns with Cytokine.
His work investigates the relationship between Anticarcinogen and topics such as Apoptosis that intersect with problems in Catechin. His Tumor promotion study integrates concerns from other disciplines, such as Tumor progression, Okadaic acid, Biotechnology and Caffeine. His study explores the link between Tumor necrosis factor alpha and topics such as Biochemistry that cross with problems in Tamoxifen.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Okadaic acid: an additional non-phorbol-12-tetradecanoate-13-acetate-type tumor promoter
Masami Suganuma;Hirota Fujiki;Hiroko Suguri;Shigeru Yoshizawa.
Proceedings of the National Academy of Sciences of the United States of America (1988)
Inhibition of protein phosphatases by microcystins and nodularin associated with hepatotoxicity.
Shigeru Yoshizawa;Rie Matsushima;Mariyo F. Watanabe;Ken-ichi Harada.
Journal of Cancer Research and Clinical Oncology (1990)
Wide Distribution of [3H](-)-epigallocatechin Gallate, a Cancer Preventive Tea Polyphenol, in Mouse Tissue
Masami Suganuma;Sachiko Okabe;Masumi Oniyama;Yukiko Tada.
Essential Role of Tumor Necrosis Factor α (TNF-α) in Tumor Promotion as Revealed by TNF-α-deficient Mice
Masami Suganuma;Sachiko Okabe;Michael W. Marino;Ayako Sakai.
Cancer Research (1999)
Antitumor promoting activity of (−)-epigallocatechin gallate, the main constituent of “Tannin” in green tea
Shigeru Yoshizawa;Takahiko Horiuchi;Hirota Fujiki;Takashi Yoshida.
Phytotherapy Research (1987)
Synergistic effects of (--)-epigallocatechin gallate with (--)-epicatechin, sulindac, or tamoxifen on cancer-preventive activity in the human lung cancer cell line PC-9.
Masami Suganuma;Sachiko Okabe;Yasuko Kai;Naoko Sueoka.
Cancer Research (1999)
Indole alkaloids: dihydroteleocidin B, teleocidin, and lyngbyatoxin A as members of a new class of tumor promoters
Hirota Fujiki;Masami Mori;Michie Nakayasu;Masaaki Terada.
Proceedings of the National Academy of Sciences of the United States of America (1981)
Green tea and cancer chemoprevention.
Masami Suganuma;Sachiko Okabe;Naoko Sueoka;Eisaburo Sueoka.
Mutation Research (1999)
Cancer inhibition by green tea
Hirota Fujiki;Masami Suganuma;Sachiko Okabe;Naoko Sueoka.
Mutation Research (1998)
Effect of (-)-epigallocatechin gallate, the main constituent of green tea, on lung metastasis with mouse B16 melanoma cell lines.
Shun'ichiro Taniguchi;Hirota Fujiki;Hiroaki Kobayashi;Hiroki Go.
Cancer Letters (1992)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: