David Dinsdale mainly focuses on Cell biology, Apoptosis, Programmed cell death, Molecular biology and Neurodegeneration. His Cell biology research includes elements of Genetics, Transcription factor and Gene isoform. As part of his studies on Apoptosis, he often connects relevant subjects like DNA.
His Programmed cell death research is multidisciplinary, incorporating elements of Cancer research and In vitro. His Molecular biology research is multidisciplinary, incorporating perspectives in Cytoplasm, Cytoplasmic inclusion, Obatoclax and Caspase 8. The various areas that he examines in his Neurodegeneration study include Chronic lymphocytic leukemia, Small molecule, Neuroprotection and Gene knockdown.
His primary areas of investigation include Cell biology, Apoptosis, Molecular biology, Programmed cell death and Cancer research. His Cell biology research includes themes of Genetics and Neurodegeneration. His research investigates the connection with Apoptosis and areas like Proteolysis which intersect with concerns in Protease.
His work is dedicated to discovering how Molecular biology, Type-II Pneumocytes are connected with Pathology and other disciplines. His research on Programmed cell death also deals with topics like
David Dinsdale spends much of his time researching Cell biology, Cancer research, Mitochondrion, Programmed cell death and Apoptosis. His Cell biology research integrates issues from Autophagy, Cell migration and Neurodegeneration. David Dinsdale has researched Cancer research in several fields, including Cell culture, Mesothelioma, Cell growth, Platelet and CDKN2A.
In his research, Calcium in biology and Endocrinology is intimately related to Internal medicine, which falls under the overarching field of Programmed cell death. In Apoptosis, he works on issues like Leukemia, which are connected to Proteasome inhibitor, Hematology and MCL1. His study looks at the relationship between Lung and fields such as Toxicity, as well as how they intersect with chemical problems.
His primary areas of study are Cell biology, Mitochondrion, Neurodegeneration, Programmed cell death and Apoptosis. His Cell biology research incorporates elements of Profilin, Oxidative stress and Gene knockdown. His Neurodegeneration research is multidisciplinary, relying on both Genetics, Neuroprotection, Unfolded protein response, Salubrinal and Mitochondrial biogenesis.
His Programmed cell death study combines topics from a wide range of disciplines, such as HaCaT and Calcium signaling. The Apoptosis study combines topics in areas such as Cell culture, Cancer research and Leukemia. The study incorporates disciplines such as Biochemistry and Cell growth in addition to Cancer research.
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Passage of inhaled particles into the blood circulation in humans.
Abderrahim Nemmar;Peter Hoet;B Vanquickenborne;D Dinsdale.
Circulation (2002)
Key morphological features of apoptosis may occur in the absence of internucleosomal DNA fragmentation.
G M Cohen;X M Sun;R T Snowden;D Dinsdale.
Biochemical Journal (1992)
Targeting autophagy potentiates tyrosine kinase inhibitor-induced cell death in Philadelphia chromosome-positive cells, including primary CML stem cells.
Cristian Bellodi;Maria Rosa Lidonnici;Ashley Hamilton;G. Vignir Helgason.
Journal of Clinical Investigation (2009)
Sustained translational repression by eIF2α-P mediates prion neurodegeneration
Julie A. Moreno;Helois Radford;Diego Peretti;Joern R. Steinert.
Nature (2012)
Bcl-2 inhibitors: small molecules with a big impact on cancer therapy
Meike Vogler;David Dinsdale;Martin J. S. Dyer;Gerald M. Cohen.
Cell Death & Differentiation (2009)
Ultrafine particles affect experimental thrombosis in an in vivo hamster model.
Abderrahim Nemmar;Marc F. Hoylaerts;Peter H. M. Hoet;David Dinsdale.
American Journal of Respiratory and Critical Care Medicine (2002)
Diesel Exhaust Particles in Lung Acutely Enhance Experimental Peripheral Thrombosis
Abderrahim Nemmar;Peter H.M. Hoet;David Dinsdale;Jozef Vermylen.
Circulation (2003)
Differential roles of p63 isoforms in epidermal development: selective genetic complementation in p63 null mice
E Candi;A Rufini;A Terrinoni;D Dinsdale.
Cell Death & Differentiation (2006)
Different forms of cell death induced by putative BCL2 inhibitors.
M Vogler;K Weber;D Dinsdale;I Schmitz.
Cell Death & Differentiation (2009)
Formation of large molecular weight fragments of DNA is a key committed step of apoptosis in thymocytes.
G. M. Cohen;Xiao-Ming Sun;H. Fearnhead;M. Macfarlane.
Journal of Immunology (1994)
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