D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 57 Citations 12,949 123 World Ranking 9297 National Ranking 701

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Apoptosis

David Dinsdale mainly focuses on Cell biology, Apoptosis, Programmed cell death, Molecular biology and Neurodegeneration. His Cell biology research includes elements of Genetics, Transcription factor and Gene isoform. As part of his studies on Apoptosis, he often connects relevant subjects like DNA.

His Programmed cell death research is multidisciplinary, incorporating elements of Cancer research and In vitro. His Molecular biology research is multidisciplinary, incorporating perspectives in Cytoplasm, Cytoplasmic inclusion, Obatoclax and Caspase 8. The various areas that he examines in his Neurodegeneration study include Chronic lymphocytic leukemia, Small molecule, Neuroprotection and Gene knockdown.

His most cited work include:

  • Passage of inhaled particles into the blood circulation in humans. (1218 citations)
  • Key morphological features of apoptosis may occur in the absence of internucleosomal DNA fragmentation. (618 citations)
  • Targeting autophagy potentiates tyrosine kinase inhibitor-induced cell death in Philadelphia chromosome-positive cells, including primary CML stem cells. (459 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Cell biology, Apoptosis, Molecular biology, Programmed cell death and Cancer research. His Cell biology research includes themes of Genetics and Neurodegeneration. His research investigates the connection with Apoptosis and areas like Proteolysis which intersect with concerns in Protease.

His work is dedicated to discovering how Molecular biology, Type-II Pneumocytes are connected with Pathology and other disciplines. His research on Programmed cell death also deals with topics like

  • Internal medicine which is related to area like Immunology,
  • Autophagy which intersects with area such as Tyrosine-kinase inhibitor. The concepts of his Cancer research study are interwoven with issues in Cell culture and Stem cell.

He most often published in these fields:

  • Cell biology (35.16%)
  • Apoptosis (21.88%)
  • Molecular biology (21.09%)

What were the highlights of his more recent work (between 2009-2019)?

  • Cell biology (35.16%)
  • Cancer research (13.28%)
  • Mitochondrion (9.38%)

In recent papers he was focusing on the following fields of study:

David Dinsdale spends much of his time researching Cell biology, Cancer research, Mitochondrion, Programmed cell death and Apoptosis. His Cell biology research integrates issues from Autophagy, Cell migration and Neurodegeneration. David Dinsdale has researched Cancer research in several fields, including Cell culture, Mesothelioma, Cell growth, Platelet and CDKN2A.

In his research, Calcium in biology and Endocrinology is intimately related to Internal medicine, which falls under the overarching field of Programmed cell death. In Apoptosis, he works on issues like Leukemia, which are connected to Proteasome inhibitor, Hematology and MCL1. His study looks at the relationship between Lung and fields such as Toxicity, as well as how they intersect with chemical problems.

Between 2009 and 2019, his most popular works were:

  • Sustained translational repression by eIF2α-P mediates prion neurodegeneration (409 citations)
  • The autophagy-associated factors DRAM1 and p62 regulate cell migration and invasion in glioblastoma stem cells (171 citations)
  • HVCN1 modulates BCR signal strength via regulation of BCR-dependent generation of reactive oxygen species (157 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Apoptosis

His primary areas of study are Cell biology, Mitochondrion, Neurodegeneration, Programmed cell death and Apoptosis. His Cell biology research incorporates elements of Profilin, Oxidative stress and Gene knockdown. His Neurodegeneration research is multidisciplinary, relying on both Genetics, Neuroprotection, Unfolded protein response, Salubrinal and Mitochondrial biogenesis.

His Programmed cell death study combines topics from a wide range of disciplines, such as HaCaT and Calcium signaling. The Apoptosis study combines topics in areas such as Cell culture, Cancer research and Leukemia. The study incorporates disciplines such as Biochemistry and Cell growth in addition to Cancer research.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Passage of inhaled particles into the blood circulation in humans.

Abderrahim Nemmar;Peter Hoet;B Vanquickenborne;D Dinsdale.
Circulation (2002)

1861 Citations

Key morphological features of apoptosis may occur in the absence of internucleosomal DNA fragmentation.

G M Cohen;X M Sun;R T Snowden;D Dinsdale.
Biochemical Journal (1992)

969 Citations

Targeting autophagy potentiates tyrosine kinase inhibitor-induced cell death in Philadelphia chromosome-positive cells, including primary CML stem cells.

Cristian Bellodi;Maria Rosa Lidonnici;Ashley Hamilton;G. Vignir Helgason.
Journal of Clinical Investigation (2009)

651 Citations

Sustained translational repression by eIF2α-P mediates prion neurodegeneration

Julie A. Moreno;Helois Radford;Diego Peretti;Joern R. Steinert.
Nature (2012)

589 Citations

Bcl-2 inhibitors: small molecules with a big impact on cancer therapy

Meike Vogler;David Dinsdale;Martin J. S. Dyer;Gerald M. Cohen.
Cell Death & Differentiation (2009)

489 Citations

Ultrafine particles affect experimental thrombosis in an in vivo hamster model.

Abderrahim Nemmar;Marc F. Hoylaerts;Peter H. M. Hoet;David Dinsdale.
American Journal of Respiratory and Critical Care Medicine (2002)

392 Citations

Diesel Exhaust Particles in Lung Acutely Enhance Experimental Peripheral Thrombosis

Abderrahim Nemmar;Peter H.M. Hoet;David Dinsdale;Jozef Vermylen.
Circulation (2003)

337 Citations

Differential roles of p63 isoforms in epidermal development: selective genetic complementation in p63 null mice

E Candi;A Rufini;A Terrinoni;D Dinsdale.
Cell Death & Differentiation (2006)

291 Citations

Different forms of cell death induced by putative BCL2 inhibitors.

M Vogler;K Weber;D Dinsdale;I Schmitz.
Cell Death & Differentiation (2009)

280 Citations

Formation of large molecular weight fragments of DNA is a key committed step of apoptosis in thymocytes.

G. M. Cohen;Xiao-Ming Sun;H. Fearnhead;M. Macfarlane.
Journal of Immunology (1994)

279 Citations

If you think any of the details on this page are incorrect, let us know.

Contact us

Best Scientists Citing David Dinsdale

Gerry Melino

Gerry Melino

University of Rome Tor Vergata

Publications: 172

Gerald M. Cohen

Gerald M. Cohen

University of Liverpool

Publications: 60

Benoit Nemery

Benoit Nemery

KU Leuven

Publications: 56

Peter Hoet

Peter Hoet

KU Leuven

Publications: 54

Eleonora Candi

Eleonora Candi

University of Rome Tor Vergata

Publications: 37

Richard A. Knight

Richard A. Knight

University of Cambridge

Publications: 32

Ken Donaldson

Ken Donaldson

University of Edinburgh

Publications: 30

Alvin M. Malkinson

Alvin M. Malkinson

University of Colorado Denver

Publications: 29

Nicholas L. Mills

Nicholas L. Mills

University of Edinburgh

Publications: 28

David E. Newby

David E. Newby

University of Edinburgh

Publications: 28

Claudio Hetz

Claudio Hetz

University of Chile

Publications: 27

Tessa L. Holyoake

Tessa L. Holyoake

University of Glasgow

Publications: 27

Zbigniew Darzynkiewicz

Zbigniew Darzynkiewicz

New York Medical College

Publications: 24

Marc Hoylaerts

Marc Hoylaerts

KU Leuven

Publications: 23

Martin J. S. Dyer

Martin J. S. Dyer

University of Leicester

Publications: 23

Marion MacFarlane

Marion MacFarlane

University of Cambridge

Publications: 22

Trending Scientists

Zoran Obradovic

Zoran Obradovic

Temple University

Daniel M Dias

Daniel M Dias

IBM (United States)

Daniel Rittel

Daniel Rittel

Technion – Israel Institute of Technology

Jianqiang Gu

Jianqiang Gu

Tianjin University

Satish Chandra Babu Myneni

Satish Chandra Babu Myneni

Princeton University

R. Pecora

R. Pecora

Stanford University

John C. Maerz

John C. Maerz

University of Georgia

Rowland R. Kao

Rowland R. Kao

University of Edinburgh

M. J. Osborn

M. J. Osborn

University of Connecticut Health Center

Friedrich Marks

Friedrich Marks

Heidelberg University

Christopher A. Bradfield

Christopher A. Bradfield

University of Wisconsin–Madison

Steven G. Younkin

Steven G. Younkin

Mayo Clinic

Song-You Hong

Song-You Hong

Yonsei University

Francisco J. Esteva

Francisco J. Esteva

New York University

Norberto Perico

Norberto Perico

Mario Negri Institute for Pharmacological Research

Benjamin D. Levine

Benjamin D. Levine

The University of Texas Southwestern Medical Center

Something went wrong. Please try again later.