David A. Greenberg

What is he best known for?

The fields of study he is best known for:

• Gene
• Genetics
• Statistics

David A. Greenberg mostly deals with Genetics, Locus, Genetic linkage, Internal medicine and Penetrance. His Genetics study combines topics in areas such as Juvenile myoclonic epilepsy and Epilepsy. His research integrates issues of Microsatellite, Allele, Graves' disease and Thyroglobulin in his study of Locus.

His Genetic linkage research is multidisciplinary, relying on both Epistasis and Linkage disequilibrium. His studies deal with areas such as Endocrinology and Immunology as well as Internal medicine. His Penetrance study combines topics from a wide range of disciplines, such as Statistics and Linkage.

His most cited work include:

• Evidence for a Susceptibility Gene for Autism on Chromosome 2 and for Genetic Heterogeneity (295 citations)
• Association between a GABRB3 polymorphism and autism. (283 citations)
• Juvenile myoclonic epilepsy (JME) may be linked to the BF and HLA loci on human chromosome 6. (256 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Genetics, Locus, Genetic linkage, Internal medicine and Statistics. His research brings together the fields of Juvenile myoclonic epilepsy and Genetics. His research on Locus also deals with topics like

• Epilepsy and related Proband,
• Candidate gene and related Genetic determinism.

His Genetic linkage research includes themes of Pedigree chart, Genetic association, Genotype, Gene mapping and Genetic heterogeneity. The concepts of his Internal medicine study are interwoven with issues in Endocrinology, Immunology and Oncology. His Statistics research is multidisciplinary, incorporating elements of Nuclear family and Genetic model.

He most often published in these fields:

• Genetics (66.81%)
• Locus (31.44%)
• Genetic linkage (25.76%)

What were the highlights of his more recent work (between 2008-2021)?

• Genetics (66.81%)
• Internal medicine (21.83%)
• Epilepsy (17.47%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Genetics, Internal medicine, Epilepsy, Locus and Genome-wide association study. His Allele, Genetic linkage, Single-nucleotide polymorphism, Genetic association and Genetic heterogeneity study are his primary interests in Genetics. His Internal medicine research integrates issues from Endocrinology, Immunology, Oncology and Cardiology.

His studies deal with areas such as Channelopathy and Gene as well as Epilepsy. His study focuses on the intersection of Locus and fields such as Genetic predisposition with connections in the field of Autoimmune thyroiditis. His work in Genome-wide association study addresses issues such as Computational biology, which are connected to fields such as Statistic, Locus heterogeneity and Epistasis.

Between 2008 and 2021, his most popular works were:

• NOS1AP Is a Genetic Modifier of the Long-QT Syndrome (212 citations)
• Centrotemporal sharp wave EEG trait in rolandic epilepsy maps to Elongator Protein Complex 4 (ELP4) (156 citations)
• HLA associations reveal genetic heterogeneity in psoriatic arthritis and in the psoriasis phenotype (145 citations)

In his most recent research, the most cited papers focused on:

• Gene
• Genetics
• Mutation

His scientific interests lie mostly in Genetics, Internal medicine, Genome-wide association study, Epilepsy and Immunology. His is involved in several facets of Genetics study, as is seen by his studies on Locus, Genetic predisposition, Candidate gene, Genetic association and Single-nucleotide polymorphism. His research investigates the link between Locus and topics such as Genotyping that cross with problems in Breast cancer.

His biological study spans a wide range of topics, including CYP2D6 Gene, Genetic heterogeneity, Endocrinology and Oncology. His work on Genetic linkage expands to the thematically related Epilepsy. His Idiopathic generalized epilepsy study integrates concerns from other disciplines, such as Juvenile myoclonic epilepsy and Myoclonic epilepsy.